Erratum: A network-based analysis of systemic inflammation in humans (Nature (2005) 437 (1032-1037) DOI: 10.1038/nature03985);A network-based analysis of systemic inflammation in humans

Nature

Volumn 437, Issue 7061, 2005, Pages 1032-1037

  Calvano, Steve E ^a   Xiao, Wenzhong ^b   Richards, Daniel R ^c   Felciano, Ramon M ^c   Baker, Henry V ^d   Cho, Raymond J ^c   Chen, Richard O ^c   Brownstein, Bernard H ^e   Cobb, J Perren ^e   Tschoeke, S Kevin ^d   Miller Graziano, Carol ^f   Moldawer, Lyle L ^d   Mindrinos, Michael N ^b   Davis, Ronald W ^b   Tompkins, Ronald G ^g   Lowry, Stephen F ^a  

^a ROBERT WOOD JOHNSON MEDICAL SCHOOL   (United States)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c Ingenuity Systems Inc   (United States)

^d UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

^e WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^f UNIVERSITY OF ROCHESTER   (United States)

^g MASSACHUSETTS GENERAL HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DISEASES; DNA; GENES; GENETIC ENGINEERING; OLIGOMERS; PATIENT TREATMENT; PROTEINS;

BIOLOGICAL INSIGHT; MICROARRAYS; SYSTEMATIC INFLAMMATION; TUMOURS;

HUMAN ENGINEERING;

ENDOTOXIN;

MEDICINE;

ADULT; ARTICLE; BIOENERGY; CONTROLLED STUDY; FEMALE; GENE EXPRESSION; GENE INTERACTION; GENETIC TRANSCRIPTION; HUMAN; HUMAN CELL; IMMUNE SYSTEM; IMMUNOLOGICAL TOLERANCE; INFECTION SENSITIVITY; INFLAMMATION; INNATE IMMUNITY; LEUKOCYTE; MALE; NORMAL HUMAN; PRIORITY JOURNAL; STATISTICAL SIGNIFICANCE;

BACTERIA (MICROORGANISMS);

EID: 27144509163     PISSN: 00280836     EISSN: 14764687     Source Type: Journal    
DOI: 10.1038/nature04362     Document Type: Erratum

References (24)

1. Heller, R. A. et al. Discovery and analysis of inflammatory disease-related genes using cDNA microarrays. Proc. Natl Acad. Sci. USA 94, 2150-2155 (1997).
2. Golub, T. R. et al. Molecular classification of cancer: class discovery and class prediction by gene expression monitoring. Science 286, 531-537 (1999).
3. Alizadeh, A. A. et al. Distinct types of diffuse large B-cell lymphoma identified by gene expression profiling. Nature 403, 503-511 (2000).
4. Bittner, M. et al. Molecular classification of cutaneous malignant melanoma by gene expression profiling. Nature 406, 536-540 (2000).
5. Perou, C. M. et al. Molecular portraits of human breast tumours. Nature 406, 747-752 (2000).
6. Coussens, L. M. & Werb, Z. Inflammation and cancer. Nature 420, 860-867 (2002).
7. Ridker, P. M., Hennekens, C. H., Buring, J. E. & Rifai, N. C-reactive protein and other markers of inflammation in the prediction of cardiovascular disease in women. N. Engl. J. Med. 342, 836-843 (2000).
8. Cheung, C. Y. et al. Induction of proinflammatory cytokines in human macrophages by influenza A (H5N1) viruses: a mechanism for the unusual severity of human disease? Lancet 360, 1831-1837 (2002).
9. Bone, R. C. et al. Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine. Chest 101, 1644-1655 (1992).
10. Fong, Y. M. et al. The acute splanchnic and peripheral tissue metabolic response to endotoxin in humans. S. Clin. Invest. 85, 1896-1904 (1990).
11. Dinauer, M. C., Orkin, S. H., Brown, R., Jesaitis, A. J. & Parkos, C. A. The glycoprotein encoded by the X-linked chronic granulomatous disease locus is a component of the neutrophil cytochrome b complex. Nature 327, 717-720 (1987).
12. Wolk, K., Docke, W. D., von Baehr, V., Volk, H. D. & Sabat, R. Impaired antigen presentation by human monocytes during endotoxin tolerance. Blood 96, 218-223 (2000).
13. Wolk, K., Kunz, S., Crompton, N. E., Volk, H. D. & Sabat, R. Multiple mechanisms of reduced major histocompatibility complex class II expression in endotoxin tolerance. J. Biol. Chem. 278, 18030-18036 (2003).
14. Nakagawa, T. et al. Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death. Nature 434, 652-658 (2005).
15. Baines, C. P. et al. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death. Nature 434, 658-662 (2005).
16. Buttgereit, F. & Brand, M. D. A hierarchy of ATP-consuming processes in mammalian cells. Biochem. J. 312, 163-167 (1995).
17. Crouser, E. D., Julian, M. W., Blaho, D. V. & Pfeiffer, D. R. Endotoxin-induced mitochondrial damage correlates with impaired respiratory activity. Crit. Care Med. 30, 276-284 (2002).
18. Padfield, K. E. et al. Burn injury causes mitochondrial dysfunction in skeletal muscle. Proc. Natl Acad. Sci. USA 102, 5368-5373 (2005).
19. Brealey, D. et al. Association between mitochondrial dysfunction and severity and outcome of septic shock. Lancet 360, 219-223 (2002).
20. Fink, M. P. Cytopathic hypoxia. Mitochondrial dysfunction as a mechanism contributing to organ dysfunction in sepsis. Crit. Care. Clin. 17, 219-237 (2001).
21. Kalil, A. C. et al. Effects of drotrecogin alfa (activated) in human endotoxemia. Shock 21, 222-229 (2004).
22. Li, C. & Wong, W. H. Model-based analysis of oligonucleotide arrays: expression index computation and outlier detection. Proc. Natl Acad. Sci. USA 98, 31-36 (2001).
23. Tusher, V. G., Tibshirani, R. & Chu, G. Significance analysis of microarrays applied to the ionizing radiation response. Proc. Natl Acad. Sci. USA 98, 5116-5121 (2001).
24. Eisen, M. B., Spellman, P. T., Brown, P. O. & Botstein, D. Cluster analysis and display of genome-wide expression patterns. Proc. Natl Acad. Sci. USA 95, 14863-14868 (1998).