Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7

Nature Communications

Volumn 6, Issue , 2015, Pages

  Minett, Michael S ^a   Pereira, Vanessa ^a   Sikandar, Shafaq ^a   Matsuyama, Ayako ^a   Lolignier, Stéphane ^a   Kanellopoulos, Alexandros H ^a   Mancini, Flavia ^a   Iannetti, Gian D ^a   Bogdanov, Yury D ^a   Santana Varela, Sonia ^a   Millet, Queensta ^a   Baskozos, Giorgios ^a   MacAllister, Raymond ^a   Cox, James J ^a   Zhao, Jing ^a   Wood, John N ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ANALGESIC AGENT; ENDORPHIN; MESSENGER RNA; METENKEPHALIN; NALOXONE; OPIATE; PENK MRNA; PROENKEPHALIN; SODIUM CHANNEL BLOCKING AGENT; SODIUM CHANNEL NAV1.7; UNCLASSIFIED DRUG; ENKEPHALIN; SCN9A PROTEIN, MOUSE;

GENE; GENE EXPRESSION; MUTATION; NEUROLOGY; PEPTIDE; PHENOTYPE; PROTEOMICS; RODENT;

ANALGESIA; ANALGESIA (SENSORY DYSFUNCTION); ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; FEMALE; GENE EXPRESSION; HUMAN; HUMAN CELL; MALE; MUTANT; NONHUMAN; PAIN; PHENOTYPE; SENSORY NERVE CELL; SPINAL CORD; UPREGULATION; ADULT; ANIMAL; CONGENITAL ANALGESIA; GENETICS; KNOCKOUT MOUSE; METABOLISM; MOUSE; PATHOPHYSIOLOGY; SENSATION;

MUS;

ADULT; ANIMALS; ENKEPHALINS; FEMALE; HUMANS; MALE; MICE; MICE, KNOCKOUT; NAV1.7 VOLTAGE-GATED SODIUM CHANNEL; PAIN INSENSITIVITY, CONGENITAL; SENSATION; SENSORY RECEPTOR CELLS;

EID: 84949294629     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms9967     Document Type: Article

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