Ccr2 deletion dissociates cavity size and tau pathology after mild traumatic brain injury

Journal of Neuroinflammation

Volumn 12, Issue 1, 2015, Pages

  Gyoneva, Stefka ^a,c   Kim, Daniel ^a,b   Katsumoto, Atsuko ^a   Kokiko Cochran, O Nicole ^a   Lamb, Bruce T ^a   Ransohoff, Richard M ^a,c  

^a LERNER RESEARCH INSTITUTE   (United States)

^b CASE WESTERN RESERVE UNIVERSITY   (United States)

^c BIOGEN   (United States)

Author keywords

CCR2; MAPT; Monocyte; Tau; Traumatic brain injury

Indexed keywords

CHEMOKINE RECEPTOR CCR2; TAU PROTEIN; CCR2 PROTEIN, MOUSE; MAPT PROTEIN, MOUSE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; CELL INFILTRATION; CONTROLLED STUDY; DISEASE ASSOCIATION; FEMALE; GENOTYPE; HIPPOCAMPUS; LESION CAVITY; MALE; MONOCYTE; MOUSE; NERVE CELL LESION; NERVE FIBER; NERVOUS SYSTEM PARAMETERS; NEUROPROTECTION; NONHUMAN; PROTEIN LOCALIZATION; PROTEIN PHOSPHORYLATION; TRAUMATIC BRAIN INJURY; WESTERN BLOTTING; ANIMAL; BRAIN INJURY; DEFICIENCY; GENE DELETION; GENETICS; KNOCKOUT MOUSE; METABOLISM; PATHOLOGY;

ANIMALS; BRAIN INJURIES; FEMALE; GENE DELETION; MALE; MICE; MICE, KNOCKOUT; MONOCYTES; RECEPTORS, CCR2; TAU PROTEINS;

EID: 84949194226     PISSN: None     EISSN: 17422094     Source Type: Journal    
DOI: 10.1186/s12974-015-0443-0     Document Type: Article

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