Deficient PKR in RAX/PKR association ameliorates ethanol-induced neurotoxicity in the developing cerebellum

Cerebellum

Volumn 14, Issue 4, 2015, Pages 386-397

  Li, Hui ^a   Chen, Jian ^b   Qi, Yuanlin ^c   Dai, Lu ^a   Zhang, Mingfang ^c   Frank, Jacqueline A ^a   Handshoe, Jonathan W ^d   Cui, Jiajun ^a   Xu, Wenhua ^e   Chen, Gang ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

^b THE FIRST AFFILIATED HOSPITAL OF XIAMEN UNIVERSITY   (China)

^c FUJIAN MEDICAL UNIVERSITY   (China)

^d UNIVERSITY OF KENTUCKY   (United States)

^e ANHUI MEDICAL UNIVERSITY   (China)

Author keywords

Alcohol; Apoptosis; Cerebellum; Fetal alcohol spectrum disorders; Neuron; PKR; Rax

Indexed keywords

ALCOHOL; INTERLEUKIN 1BETA; PROTEIN KINASE R; CENTRAL DEPRESSANT AGENT; CHOLECYSTOKININ OCTAPEPTIDE; CYTOKINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BINDING AFFINITY; BINDING SITE; BODY MASS; BRAIN DEVELOPMENT; BRAIN SIZE; CELL LOSS; CONTROLLED STUDY; CYTOKINE PRODUCTION; CYTOKINE RELEASE; DISEASE ACTIVITY; DISEASE ASSOCIATION; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME DEFICIENCY; MOLECULAR DYNAMICS; MOLECULAR PATHOLOGY; MOUSE; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN PROTEIN INTERACTION; PROTEIN TARGETING; AGE; ANIMAL; APOPTOSIS; C57BL MOUSE; CELL CULTURE; CEREBELLUM; DEFICIENCY; DISEASE MODEL; DRUG EFFECTS; FEMALE; GENE EXPRESSION REGULATION; GENETICS; GROWTH, DEVELOPMENT AND AGING; HUMAN; MALE; METABOLISM; NERVE CELL; NEUROTOXICITY SYNDROMES; NEWBORN; ORGAN SIZE; PATHOLOGY; TIME FACTOR; TRANSGENIC ANIMAL;

AGE FACTORS; ANIMALS; ANIMALS, GENETICALLY MODIFIED; ANIMALS, NEWBORN; APOPTOSIS; CELLS, CULTURED; CENTRAL NERVOUS SYSTEM DEPRESSANTS; CEREBELLUM; CYTOKINES; DISEASE MODELS, ANIMAL; EIF-2 KINASE; ETHANOL; FEMALE; GENE EXPRESSION REGULATION, DEVELOPMENTAL; HUMANS; MALE; MICE; MICE, INBRED C57BL; NEURONS; NEUROTOXICITY SYNDROMES; ORGAN SIZE; SINCALIDE; TIME FACTORS;

EID: 84943197353     PISSN: 14734222     EISSN: 14734230     Source Type: Journal    
DOI: 10.1007/s12311-015-0644-1     Document Type: Article

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