Oxidative stress caused by ozone exposure induces β-amyloid 1-42 overproduction and mitochondrial accumulation by activating the amyloidogenic pathway

Neuroscience

Volumn 304, Issue , 2015, Pages 340-348

  Hernandez Zimbron L F ^a   Rivas Arancibia, S ^a  

^a UNIVERSIDAD NACIONAL AUTÓNOMA DE MÉXICO   (Mexico)

Author keywords

Alzheimer's disease; Neurodegeneration; Oxidative stress; Ozone

Indexed keywords

ADAM10 ENDOPEPTIDASE; ALPHA SECRETASE; AMYLOID BETA PROTEIN[1-40]; AMYLOID BETA PROTEIN[1-42]; CYCLOOXYGENASE 1; GAMMA SECRETASE; MARKER; OPA1 PROTEIN; OZONE; PRESENILIN 1; PRESENILIN 2; UNCLASSIFIED DRUG; ADAM PROTEIN; ADAM10 PROTEIN, RAT; AIR POLLUTANT; AMYLOID BETA PROTEIN; AMYLOID BETA-PROTEIN (1-42); PEPTIDE FRAGMENT; PSEN1 PROTEIN, RAT; PSEN2 PROTEIN, RAT;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BRAIN MITOCHONDRION; CONTROLLED STUDY; HIPPOCAMPUS; LONG TERM EXPOSURE; MALE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; AIR POLLUTANT; ANIMAL; CHRONIC DISEASE; DISEASE MODEL; METABOLISM; MITOCHONDRION; PATHOLOGY; PHYSIOLOGY; RANDOMIZATION; SIGNAL TRANSDUCTION; WISTAR RAT;

ADAM PROTEINS; AIR POLLUTANTS; AMYLOID BETA-PEPTIDES; ANIMALS; CHRONIC DISEASE; DISEASE MODELS, ANIMAL; HIPPOCAMPUS; MALE; MITOCHONDRIA; OXIDATIVE STRESS; OZONE; PEPTIDE FRAGMENTS; PRESENILIN-1; PRESENILIN-2; RANDOM ALLOCATION; RATS, WISTAR; SIGNAL TRANSDUCTION;

EID: 84938902297     PISSN: 03064522     EISSN: 18737544     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2015.07.011     Document Type: Article

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