Transcriptional repression of Sin3B by Bmi-1 prevents cellular senescence and is relieved by oncogene activation

Oncogene

Volumn 34, Issue 30, 2015, Pages 4011-4017

  DiMauro, T ^a   Cantor, D J ^a   Bainor, A J ^a   David, G ^a  

^a NEW YORK UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BMI1 PROTEIN; PROTEIN; REACTIVE OXYGEN METABOLITE; SHORT HAIRPIN RNA; SIN3B PROTEIN; TAMOXIFEN; UNCLASSIFIED DRUG; BMI1 PROTEIN, MOUSE; ONCOPROTEIN; REPRESSOR PROTEIN; SIN3B PROTEIN, MOUSE;

ARTICLE; CELL AGING; CELL CYCLE; CELL PROLIFERATION; CELL STRESS; CELL SURVIVAL; CHROMATIN; CONTROLLED STUDY; DISSOCIATION; GENE EXPRESSION; GENE INACTIVATION; GENE LOCUS; GENE REPRESSION; GENETIC TRANSCRIPTION; ONCOGENE; PHENOTYPIC VARIATION; PRIORITY JOURNAL; PROTEIN TARGETING; SENESCENCE; TRANSCRIPTION INITIATION; TRANSCRIPTION INITIATION SITE; ANIMAL; CELL CULTURE; GENE EXPRESSION REGULATION; GENETICS; METABOLISM; MOUSE; PHYSIOLOGY; UPREGULATION;

MUS;

ANIMALS; CELL AGING; CELL PROLIFERATION; CELLS, CULTURED; GENE EXPRESSION REGULATION, NEOPLASTIC; MICE; ONCOGENES; POLYCOMB REPRESSIVE COMPLEX 1; PROTO-ONCOGENE PROTEINS; REPRESSOR PROTEINS; TRANSCRIPTION, GENETIC; UP-REGULATION;

EID: 84937901506     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/onc.2014.322     Document Type: Article

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