-
1
-
-
0027478391
-
Receptor editing in selfreactive bone marrow B cells
-
Tiegs, S. L., D. M. Russell, and D. Nemazee. 1993. Receptor editing in selfreactive bone marrow B cells. J. Exp. Med. 177: 1009-1020.
-
(1993)
J. Exp. Med.
, vol.177
, pp. 1009-1020
-
-
Tiegs, S.L.1
Russell, D.M.2
Nemazee, D.3
-
2
-
-
84903712667
-
The role of CD22 and Siglec-G in B-cell tolerance and autoimmune disease
-
Müller, J., and L. Nitschke. 2014. The role of CD22 and Siglec-G in B-cell tolerance and autoimmune disease. Nat Rev Rheumatol 10: 422-428.
-
(2014)
Nat Rev Rheumatol
, vol.10
, pp. 422-428
-
-
Müller, J.1
Nitschke, L.2
-
3
-
-
84861161715
-
Role of inhibitory BCR co-receptors in immunity
-
Tsubata, T. 2012. Role of inhibitory BCR co-receptors in immunity. Infect. Disord. Drug Targets 12: 181-190.
-
(2012)
Infect. Disord. Drug Targets
, vol.12
, pp. 181-190
-
-
Tsubata, T.1
-
4
-
-
0033695963
-
Spontaneous autoimmune disease in FcgRIIB-deficient mice results from strain-specific epistasis
-
Bolland, S., and J. V. Ravetch. 2000. Spontaneous autoimmune disease in FcgRIIB-deficient mice results from strain-specific epistasis. Immunity 13: 277-285.
-
(2000)
Immunity
, vol.13
, pp. 277-285
-
-
Bolland, S.1
Ravetch, J.V.2
-
5
-
-
69549129469
-
Augmented TLR9-induced Btk activation in PIR-B-deficient B-1 cells provokes excessive autoantibody production and autoimmunity
-
Kubo, T., Y. Uchida, Y. Watanabe, M. Abe, A. Nakamura, M. Ono, S. Akira, and T. Takai. 2009. Augmented TLR9-induced Btk activation in PIR-B-deficient B-1 cells provokes excessive autoantibody production and autoimmunity. J. Exp. Med. 206: 1971-1982.
-
(2009)
J. Exp. Med.
, vol.206
, pp. 1971-1982
-
-
Kubo, T.1
Uchida, Y.2
Watanabe, Y.3
Abe, M.4
Nakamura, A.5
Ono, M.6
Akira, S.7
Takai, T.8
-
6
-
-
54949131682
-
Modulation of peripheral B cell tolerance by CD72 in a murine model
-
Li, D. H., M. M. Winslow, T. M. Cao, A. H. Chen, C. R. Davis, E. D. Mellins, P. J. Utz, G. R. Crabtree, and J. R. Parnes. 2008. Modulation of peripheral B cell tolerance by CD72 in a murine model. Arthritis Rheum. 58: 3192-3204.
-
(2008)
Arthritis Rheum.
, vol.58
, pp. 3192-3204
-
-
Li, D.H.1
Winslow, M.M.2
Cao, T.M.3
Chen, A.H.4
Davis, C.R.5
Mellins, E.D.6
Utz, P.J.7
Crabtree, G.R.8
Parnes, J.R.9
-
7
-
-
0028784215
-
Multiple defects in the immune system of Lyn-deficient mice, culminating in autoimmune disease
-
Hibbs, M. L., D. M. Tarlinton, J. Armes, D. Grail, G. Hodgson, R. Maglitto, S. A. Stacker, and A. R. Dunn. 1995. Multiple defects in the immune system of Lyn-deficient mice, culminating in autoimmune disease. Cell 83: 301-311.
-
(1995)
Cell
, vol.83
, pp. 301-311
-
-
Hibbs, M.L.1
Tarlinton, D.M.2
Armes, J.3
Grail, D.4
Hodgson, G.5
Maglitto, R.6
Stacker, S.A.7
Dunn, A.R.8
-
8
-
-
34447624878
-
B cell-specific deletion of protein-tyrosine phosphatase Shp1 promotes B-1a cell development and causes systemic autoimmunity
-
Pao, L. I., K. P. Lam, J. M. Henderson, J. L. Kutok, M. Alimzhanov, L. Nitschke, M. L. Thomas, B. G. Neel, and K. Rajewsky. 2007. B cell-specific deletion of protein-tyrosine phosphatase Shp1 promotes B-1a cell development and causes systemic autoimmunity. Immunity 27: 35-48.
-
(2007)
Immunity
, vol.27
, pp. 35-48
-
-
Pao, L.I.1
Lam, K.P.2
Henderson, J.M.3
Kutok, J.L.4
Alimzhanov, M.5
Nitschke, L.6
Thomas, M.L.7
Neel, B.G.8
Rajewsky, K.9
-
9
-
-
34250753787
-
Siglec-G is a B1 cell-inhibitory receptor that controls expansion and calcium signaling of the B1 cell population
-
Hoffmann, A., S. Kerr, J. Jellusova, J. Zhang, F. Weisel, U. Wellmann, T. H. Winkler, B. Kneitz, P. R. Crocker, and L. Nitschke. 2007. Siglec-G is a B1 cell-inhibitory receptor that controls expansion and calcium signaling of the B1 cell population. Nat. Immunol. 8: 695-704.
-
(2007)
Nat. Immunol.
, vol.8
, pp. 695-704
-
-
Hoffmann, A.1
Kerr, S.2
Jellusova, J.3
Zhang, J.4
Weisel, F.5
Wellmann, U.6
Winkler, T.H.7
Kneitz, B.8
Crocker, P.R.9
Nitschke, L.10
-
10
-
-
0031080328
-
CD22 is a negative regulator of B-cell receptor signalling
-
Nitschke, L., R. Carsetti, B. Ocker, G. Köhler, and M. C. Lamers. 1997. CD22 is a negative regulator of B-cell receptor signalling. Curr. Biol. 7: 133-143.
-
(1997)
Curr. Biol.
, vol.7
, pp. 133-143
-
-
Nitschke, L.1
Carsetti, R.2
Ocker, B.3
Köhler, G.4
Lamers, M.C.5
-
11
-
-
0030467610
-
CD22 regulates thymus-independent responses and the lifespan of B cells
-
Otipoby, K. L., K. B. Andersson, K. E. Draves, S. J. Klaus, A. G. Farr, J. D. Kerner, R. M. Perlmutter, C. L. Law, and E. A. Clark. 1996. CD22 regulates thymus-independent responses and the lifespan of B cells. Nature 384: 634-637.
-
(1996)
Nature
, vol.384
, pp. 634-637
-
-
Otipoby, K.L.1
Andersson, K.B.2
Draves, K.E.3
Klaus, S.J.4
Farr, A.G.5
Kerner, J.D.6
Perlmutter, R.M.7
Law, C.L.8
Clark, E.A.9
-
12
-
-
0030499431
-
CD22 is both a positive and negative regulator of B lymphocyte antigen receptor signal transduction: Altered signaling in CD22-deficient mice
-
Sato, S., A. S. Miller, M. Inaoki, C. B. Bock, P. J. Jansen, M. L. Tang, and T. F. Tedder. 1996. CD22 is both a positive and negative regulator of B lymphocyte antigen receptor signal transduction: altered signaling in CD22-deficient mice. Immunity 5: 551-562.
-
(1996)
Immunity
, vol.5
, pp. 551-562
-
-
Sato, S.1
Miller, A.S.2
Inaoki, M.3
Bock, C.B.4
Jansen, P.J.5
Tang, M.L.6
Tedder, T.F.7
-
13
-
-
77951627428
-
CD22 3 Siglec-G double-deficient mice have massively increased B1 cell numbers and develop systemic autoimmunity
-
Jellusova, J., U. Wellmann, K. Amann, T. H. Winkler, and L. Nitschke. 2010. CD22 3 Siglec-G double-deficient mice have massively increased B1 cell numbers and develop systemic autoimmunity. J. Immunol. 184: 3618-3627.
-
(2010)
J. Immunol.
, vol.184
, pp. 3618-3627
-
-
Jellusova, J.1
Wellmann, U.2
Amann, K.3
Winkler, T.H.4
Nitschke, L.5
-
14
-
-
84897567205
-
Siglec-G deficiency leads to more severe collagen-induced arthritis and earlier onset of lupus-like symptoms in MRL/lpr mice
-
Bökers, S., A. Urbat, C. Daniel, K. Amann, K. G. Smith, M. Espéli, and L. Nitschke. 2014. Siglec-G deficiency leads to more severe collagen-induced arthritis and earlier onset of lupus-like symptoms in MRL/lpr mice. J. Immunol. 192: 2994-3002.
-
(2014)
J. Immunol.
, vol.192
, pp. 2994-3002
-
-
Bökers, S.1
Urbat, A.2
Daniel, C.3
Amann, K.4
Smith, K.G.5
Espéli, M.6
Nitschke, L.7
-
15
-
-
0034665505
-
Dysregulated expression of the Cd22 gene as a result of a short interspersed nucleotide element insertion in Cd22a lupus-prone mice
-
Mary, C., C. Laporte, D. Parzy, M. L. Santiago, F. Stefani, F. Lajaunias, R. M. Parkhouse, T. L. O'Keefe, M. S. Neuberger, S. Izui, and L. Reininger. 2000. Dysregulated expression of the Cd22 gene as a result of a short interspersed nucleotide element insertion in Cd22a lupus-prone mice. J. Immunol. 165: 2987-2996.
-
(2000)
J. Immunol.
, vol.165
, pp. 2987-2996
-
-
Mary, C.1
Laporte, C.2
Parzy, D.3
Santiago, M.L.4
Stefani, F.5
Lajaunias, F.6
Parkhouse, R.M.7
O'Keefe, T.L.8
Neuberger, M.S.9
Izui, S.10
Reininger, L.11
-
16
-
-
60549105605
-
B cell antigen receptor signal strength and peripheral B cell development are regulated by a 9-O-acetyl sialic acid esterase
-
Cariappa, A., H. Takematsu, H. Liu, S. Diaz, K. Haider, C. Boboila, G. Kalloo, M. Connole, H. N. Shi, N. Varki, et al. 2009. B cell antigen receptor signal strength and peripheral B cell development are regulated by a 9-O-acetyl sialic acid esterase. J. Exp. Med. 206: 125-138.
-
(2009)
J. Exp. Med.
, vol.206
, pp. 125-138
-
-
Cariappa, A.1
Takematsu, H.2
Liu, H.3
Diaz, S.4
Haider, K.5
Boboila, C.6
Kalloo, G.7
Connole, M.8
Shi, H.N.9
Varki, N.10
-
17
-
-
77954543178
-
Functionally defective germline variants of sialic acid acetylesterase in autoimmunity
-
Surolia, I., S. P. Pirnie, V. Chellappa, K. N. Taylor, A. Cariappa, J. Moya, H. Liu, D. W. Bell, D. R. Driscoll, S. Diederichs, et al. 2010. Functionally defective germline variants of sialic acid acetylesterase in autoimmunity. Nature 466: 243-247.
-
(2010)
Nature
, vol.466
, pp. 243-247
-
-
Surolia, I.1
Pirnie, S.P.2
Chellappa, V.3
Taylor, K.N.4
Cariappa, A.5
Moya, J.6
Liu, H.7
Bell, D.W.8
Driscoll, D.R.9
Diederichs, S.10
-
18
-
-
76149134682
-
Decoration of Tindependent antigen with ligands for CD22 and Siglec-G can suppress immunity and induce B cell tolerance in vivo
-
Duong, B. H., H. Tian, T. Ota, G. Completo, S. Han, J. L. Vela, M. Ota, M. Kubitz, N. Bovin, J. C. Paulson, and D. Nemazee. 2010. Decoration of Tindependent antigen with ligands for CD22 and Siglec-G can suppress immunity and induce B cell tolerance in vivo. J. Exp. Med. 207: 173-187.
-
(2010)
J. Exp. Med.
, vol.207
, pp. 173-187
-
-
Duong, B.H.1
Tian, H.2
Ota, T.3
Completo, G.4
Han, S.5
Vela, J.L.6
Ota, M.7
Kubitz, M.8
Bovin, N.9
Paulson, J.C.10
Nemazee, D.11
-
19
-
-
84881451966
-
Copresentation of antigen and ligands of Siglec-G induces B cell tolerance independent of CD22
-
Pfrengle, F., M. S. Macauley, N. Kawasaki, and J. C. Paulson. 2013. Copresentation of antigen and ligands of Siglec-G induces B cell tolerance independent of CD22. J. Immunol. 191: 1724-1731.
-
(2013)
J. Immunol.
, vol.191
, pp. 1724-1731
-
-
Pfrengle, F.1
Macauley, M.S.2
Kawasaki, N.3
Paulson, J.C.4
-
20
-
-
0035214030
-
A new siglec family member, siglec-10, is expressed in cells of the immune system and has signaling properties similar to CD33
-
Whitney, G., S. Wang, H. Chang, K. Y. Cheng, P. Lu, X. D. Zhou, W. P. Yang, M. McKinnon, and M. Longphre. 2001. A new siglec family member, siglec-10, is expressed in cells of the immune system and has signaling properties similar to CD33. Eur. J. Biochem. 268: 6083-6096.
-
(2001)
Eur. J. Biochem.
, vol.268
, pp. 6083-6096
-
-
Whitney, G.1
Wang, S.2
Chang, H.3
Cheng, K.Y.4
Lu, P.5
Zhou, X.D.6
Yang, W.P.7
McKinnon, M.8
Longphre, M.9
-
21
-
-
84901255884
-
The ligand-binding domain of Siglec-G is crucial for its selective inhibitory function on B1 cells
-
Hutzler, S., L. Özgör, Y. Naito-Matsui, K. Kläsener, T. H. Winkler, M. Reth, and L. Nitschke. 2014. The ligand-binding domain of Siglec-G is crucial for its selective inhibitory function on B1 cells. J. Immunol. 192: 5406-5414.
-
(2014)
J. Immunol.
, vol.192
, pp. 5406-5414
-
-
Hutzler, S.1
Özgör, L.2
Naito-Matsui, Y.3
Kläsener, K.4
Winkler, T.H.5
Reth, M.6
Nitschke, L.7
-
22
-
-
0028347321
-
A 13-amino-acid motif in the cytoplasmic domain of FcgRIIB modulates B-cell receptor signalling
-
Muta, T., T. Kurosaki, Z. Misulovin, M. Sanchez, M. C. Nussenzweig, and J. V. Ravetch. 1994. A 13-amino-acid motif in the cytoplasmic domain of FcgRIIB modulates B-cell receptor signalling. Nature 368: 70-73.
-
(1994)
Nature
, vol.368
, pp. 70-73
-
-
Muta, T.1
Kurosaki, T.2
Misulovin, Z.3
Sanchez, M.4
Nussenzweig, M.C.5
Ravetch, J.V.6
-
23
-
-
0029835940
-
Role of the inositol phosphatase SHIP in negative regulation of the immune system by the receptor FcgRIIB
-
Ono, M., S. Bolland, P. Tempst, and J. V. Ravetch. 1996. Role of the inositol phosphatase SHIP in negative regulation of the immune system by the receptor FcgRIIB. Nature 383: 263-266.
-
(1996)
Nature
, vol.383
, pp. 263-266
-
-
Ono, M.1
Bolland, S.2
Tempst, P.3
Ravetch, J.V.4
-
24
-
-
0343307005
-
Deletion of SHIP or SHP-1 reveals two distinct pathways for inhibitory signaling
-
Ono, M., H. Okada, S. Bolland, S. Yanagi, T. Kurosaki, and J. V. Ravetch. 1997. Deletion of SHIP or SHP-1 reveals two distinct pathways for inhibitory signaling. Cell 90: 293-301.
-
(1997)
Cell
, vol.90
, pp. 293-301
-
-
Ono, M.1
Okada, H.2
Bolland, S.3
Yanagi, S.4
Kurosaki, T.5
Ravetch, J.V.6
-
25
-
-
0033151596
-
SHIP recruitment attenuates FcgRIIB-induced B cell apoptosis
-
Pearse, R. N., T. Kawabe, S. Bolland, R. Guinamard, T. Kurosaki, and J. V. Ravetch. 1999. SHIP recruitment attenuates FcgRIIB-induced B cell apoptosis. Immunity 10: 753-760.
-
(1999)
Immunity
, vol.10
, pp. 753-760
-
-
Pearse, R.N.1
Kawabe, T.2
Bolland, S.3
Guinamard, R.4
Kurosaki, T.5
Ravetch, J.V.6
-
26
-
-
33947575824
-
FcgRIIb controls bone marrow plasma cell persistence and apoptosis
-
Xiang, Z., A. J. Cutler, R. J. Brownlie, K. Fairfax, K. E. Lawlor, E. Severinson, E.U.Walker, R. A. Manz,D.M. Tarlinton, and K. G. Smith. 2007. FcgRIIb controls bone marrow plasma cell persistence and apoptosis. Nat. Immunol. 8: 419-429.
-
(2007)
Nat. Immunol.
, vol.8
, pp. 419-429
-
-
Xiang, Z.1
Cutler, A.J.2
Brownlie, R.J.3
Fairfax, K.4
Lawlor, K.E.5
Severinson, E.6
Walker, E.U.7
Manz, R.A.8
Tarlinton, D.M.9
Smith, K.G.10
-
27
-
-
84897491044
-
Inhibitory Fcg receptor is required for the maintenance of tolerance through distinct mechanisms
-
Li, F., P. Smith, and J. V. Ravetch. 2014. Inhibitory Fcg receptor is required for the maintenance of tolerance through distinct mechanisms. J. Immunol. 192: 3021-3028.
-
(2014)
J. Immunol.
, vol.192
, pp. 3021-3028
-
-
Li, F.1
Smith, P.2
Ravetch, J.V.3
-
28
-
-
80051627971
-
The inhibiting Fc receptor for IgG, FcgRIIB, is a modifier of autoimmune susceptibility
-
Boross, P., V. L. Arandhara, J. Martin-Ramirez, M. L. Santiago-Raber, F. Carlucci, R. Flierman, J. van der Kaa, C. Breukel, J. W. Claassens, M. Camps, et al. 2011. The inhibiting Fc receptor for IgG, FcgRIIB, is a modifier of autoimmune susceptibility. J. Immunol. 187: 1304-1313.
-
(2011)
J. Immunol.
, vol.187
, pp. 1304-1313
-
-
Boross, P.1
Arandhara, V.L.2
Martin-Ramirez, J.3
Santiago-Raber, M.L.4
Carlucci, F.5
Flierman, R.6
Kaa Der J.Van7
Breukel, C.8
Claassens, J.W.9
Camps, M.10
-
29
-
-
10344252778
-
Association of extensive polymorphisms in the SLAM/ CD2 gene cluster with murine lupus
-
Wandstrat, A. E., C. Nguyen, N. Limaye, A. Y. Chan, S. Subramanian, X. H. Tian, Y. S. Yim, A. Pertsemlidis, H. R. Garner, Jr., L. Morel, and E. K. Wakeland. 2004. Association of extensive polymorphisms in the SLAM/ CD2 gene cluster with murine lupus. Immunity 21: 769-780.
-
(2004)
Immunity
, vol.21
, pp. 769-780
-
-
Wandstrat, A.E.1
Nguyen, C.2
Limaye, N.3
Chan, A.Y.4
Subramanian, S.5
Tian, X.H.6
Yim, Y.S.7
Pertsemlidis, A.8
Garner, H.R.9
Morel, L.10
Wakeland, E.K.11
-
30
-
-
27144549658
-
Loss of function of a lupus-associated FcgRIIb polymorphism through exclusion from lipid rafts
-
Floto, R. A., M. R. Clatworthy, K. R. Heilbronn, D. R. Rosner, P. A. MacAry, A. Rankin, P. J. Lehner, W. H. Ouwehand, J. M. Allen, N. A. Watkins, and K. G. Smith. 2005. Loss of function of a lupus-associated FcgRIIb polymorphism through exclusion from lipid rafts. Nat. Med. 11: 1056-1058.
-
(2005)
Nat. Med.
, vol.11
, pp. 1056-1058
-
-
Floto, R.A.1
Clatworthy, M.R.2
Heilbronn, K.R.3
Rosner, D.R.4
Macary, P.A.5
Rankin, A.6
Lehner, P.J.7
Ouwehand, W.H.8
Allen, J.M.9
Watkins, N.A.10
Smith, K.G.11
-
31
-
-
26444593959
-
FcgRIIB Ile232Thr transmembrane polymorphism associated with human systemic lupus erythematosus decreases affinity to lipid rafts and attenuates inhibitory effects on B cell receptor signaling
-
Kono, H., C. Kyogoku, T. Suzuki, N. Tsuchiya, H. Honda, K. Yamamoto, K. Tokunaga, and Z. Honda. 2005. FcgRIIB Ile232Thr transmembrane polymorphism associated with human systemic lupus erythematosus decreases affinity to lipid rafts and attenuates inhibitory effects on B cell receptor signaling. Hum. Mol. Genet. 14: 2881-2892.
-
(2005)
Hum. Mol. Genet.
, vol.14
, pp. 2881-2892
-
-
Kono, H.1
Kyogoku, C.2
Suzuki, T.3
Tsuchiya, N.4
Honda, H.5
Yamamoto, K.6
Tokunaga, K.7
Honda, Z.8
-
32
-
-
77951572775
-
A defunctioning polymorphism in FCGR2B is associated with protection against malaria but susceptibility to systemic lupus erythematosus
-
Willcocks, L. C., E. J. Carr, H. A. Niederer, T. F. Rayner, T. N. Williams, W. Yang, J. A. Scott, B. C. Urban, N. Peshu, T. J. Vyse, et al. 2010. A defunctioning polymorphism in FCGR2B is associated with protection against malaria but susceptibility to systemic lupus erythematosus. Proc. Natl. Acad. Sci. USA 107: 7881-7885.
-
(2010)
Proc. Natl. Acad. Sci. USA
, vol.107
, pp. 7881-7885
-
-
Willcocks, L.C.1
Carr, E.J.2
Niederer, H.A.3
Rayner, T.F.4
Williams, T.N.5
Yang, W.6
Scott, J.A.7
Urban, B.C.8
Peshu, N.9
Vyse, T.J.10
-
33
-
-
77953760056
-
Antibody-mediated modulation of immune responses
-
Nimmerjahn, F., and J. V. Ravetch. 2010. Antibody-mediated modulation of immune responses. Immunol. Rev. 236: 265-275.
-
(2010)
Immunol. Rev.
, vol.236
, pp. 265-275
-
-
Nimmerjahn, F.1
Ravetch, J.V.2
-
34
-
-
0347382447
-
B cell defects in SLP65/BLNK-deficient mice can be partially corrected by the absence of CD22, an inhibitory coreceptor for BCR signaling
-
Gerlach, J., S. Ghosh, H. Jumaa, M. Reth, J. Wienands, A. C. Chan, and L. Nitschke. 2003. B cell defects in SLP65/BLNK-deficient mice can be partially corrected by the absence of CD22, an inhibitory coreceptor for BCR signaling. Eur. J. Immunol. 33: 3418-3426.
-
(2003)
Eur. J. Immunol.
, vol.33
, pp. 3418-3426
-
-
Gerlach, J.1
Ghosh, S.2
Jumaa, H.3
Reth, M.4
Wienands, J.5
Chan, A.C.6
Nitschke, L.7
-
35
-
-
33750308551
-
Identification of chromosome intervals from 129 and C57BL/6 mouse strains linked to the development of systemic lupus erythematosus
-
Heidari, Y., A. E. Bygrave, R. J. Rigby, K. L. Rose, M. J. Walport, H. T. Cook, T. J. Vyse, and M. Botto. 2006. Identification of chromosome intervals from 129 and C57BL/6 mouse strains linked to the development of systemic lupus erythematosus. Genes Immun. 7: 592-599.
-
(2006)
Genes Immun.
, vol.7
, pp. 592-599
-
-
Heidari, Y.1
Bygrave, A.E.2
Rigby, R.J.3
Rose, K.L.4
Walport, M.J.5
Cook, H.T.6
Vyse, T.J.7
Botto, M.8
-
36
-
-
84861146585
-
FcgRIIb and BAFF differentially regulate peritoneal B1 cell survival
-
Amezcua Vesely, M. C., M. Schwartz, D. A. Bermejo, C. L. Montes, K. M. Cautivo, A. M. Kalergis, D. J. Rawlings, E. V. Acosta-Rodríguez, and A. Gruppi. 2012. FcgRIIb and BAFF differentially regulate peritoneal B1 cell survival. J. Immunol. 188: 4792-4800.
-
(2012)
J. Immunol.
, vol.188
, pp. 4792-4800
-
-
Amezcua Vesely, M.C.1
Schwartz, M.2
Bermejo, D.A.3
Montes, C.L.4
Cautivo, K.M.5
Kalergis, A.M.6
Rawlings, D.J.7
Acosta-Rodríguez, E.V.8
Gruppi, A.9
-
37
-
-
79952675131
-
Follicular helper CD4 T cells (TFH)
-
Crotty, S. 2011. Follicular helper CD4 T cells (TFH). Annu. Rev. Immunol. 29: 621-663.
-
(2011)
Annu. Rev. Immunol.
, vol.29
, pp. 621-663
-
-
Crotty, S.1
-
38
-
-
65249086734
-
Kallikrein genes are associated with lupus and glomerular basement membrane-specific antibody-induced nephritis in mice and humans
-
Profile Study GroupItalian Collaborative GroupGerman Collaborative Group-Spanish Collaborative GroupArgentinian Collaborative GroupSLEGEN Consortium
-
Profile Study GroupItalian Collaborative GroupGerman Collaborative Group-Spanish Collaborative GroupArgentinian Collaborative GroupSLEGEN Consortium. 2009. Kallikrein genes are associated with lupus and glomerular basement membrane-specific antibody-induced nephritis in mice and humans. J. Clin. Invest. 119: 911-923.
-
(2009)
J. Clin. Invest.
, vol.119
, pp. 911-923
-
-
-
39
-
-
84869745566
-
Pathogenic long-lived plasma cells and their survival niches in autoimmunity, malignancy, and allergy
-
Winter, O., C. Dame, F. Jundt, and F. Hiepe. 2012. Pathogenic long-lived plasma cells and their survival niches in autoimmunity, malignancy, and allergy. J. Immunol. 189: 5105-5111.
-
(2012)
J. Immunol.
, vol.189
, pp. 5105-5111
-
-
Winter, O.1
Dame, C.2
Jundt, F.3
Hiepe, F.4
-
40
-
-
23744432462
-
Balancing diversity and tolerance: Lessons from patients with systemic lupus erythematosus
-
Jacobi, A. M., and B. Diamond. 2005. Balancing diversity and tolerance: lessons from patients with systemic lupus erythematosus. J. Exp. Med. 202: 341-344.
-
(2005)
J. Exp. Med.
, vol.202
, pp. 341-344
-
-
Jacobi, A.M.1
Diamond, B.2
-
41
-
-
24744462348
-
The role of natural antibodies in atherogenesis
-
Binder, C. J., P. X. Shaw, M. K. Chang, A. Boullier, K. Hartvigsen, S. Hörkkö, Y. I. Miller, D. A. Woelkers, M. Corr, and J. L. Witztum. 2005. The role of natural antibodies in atherogenesis. J. Lipid Res. 46: 1353-1363.
-
(2005)
J. Lipid Res.
, vol.46
, pp. 1353-1363
-
-
Binder, C.J.1
Shaw, P.X.2
Chang, M.K.3
Boullier, A.4
Hartvigsen, K.5
Hörkkö, S.6
Miller, Y.I.7
Woelkers, D.A.8
Corr, M.9
Witztum, J.L.10
-
42
-
-
0029584458
-
B1 and B2 cells differ in their potential to switch immunoglobulin isotype
-
Tarlinton, D. M., M. McLean, and G. J. Nossal. 1995. B1 and B2 cells differ in their potential to switch immunoglobulin isotype. Eur. J. Immunol. 25: 3388-3393.
-
(1995)
Eur. J. Immunol.
, vol.25
, pp. 3388-3393
-
-
Tarlinton, D.M.1
McLean, M.2
Nossal, G.J.3
-
43
-
-
77953319727
-
Class switching and consecutive loss of dsDNA-reactive B1a B cells from the peritoneal cavity during murine lupus development
-
Enghard, P., J. Y. Humrich, V. T. Chu, E. Grussie, F. Hiepe, G. R. Burmester, A. Radbruch, C. Berek, and G. Riemekasten. 2010. Class switching and consecutive loss of dsDNA-reactive B1a B cells from the peritoneal cavity during murine lupus development. Eur. J. Immunol. 40: 1809-1818.
-
(2010)
Eur. J. Immunol.
, vol.40
, pp. 1809-1818
-
-
Enghard, P.1
Humrich, J.Y.2
Chu, V.T.3
Grussie, E.4
Hiepe, F.5
Burmester, G.R.6
Radbruch, A.7
Berek, C.8
Riemekasten, G.9
-
44
-
-
78650600361
-
The double life of a B-1 cell: Self-reactivity selects for protective effector functions
-
Baumgarth, N. 2011. The double life of a B-1 cell: self-reactivity selects for protective effector functions. Nat. Rev. Immunol. 11: 34-46.
-
(2011)
Nat. Rev. Immunol.
, vol.11
, pp. 34-46
-
-
Baumgarth, N.1
-
45
-
-
0037029604
-
Genetic modifiers of systemic lupus erythematosus in FcgRIIB2/2 mice
-
Bolland, S., Y. S. Yim, K. Tus, E. K. Wakeland, and J. V. Ravetch. 2002. Genetic modifiers of systemic lupus erythematosus in FcgRIIB2/2 mice. J. Exp. Med. 195: 1167-1174.
-
(2002)
J. Exp. Med.
, vol.195
, pp. 1167-1174
-
-
Bolland, S.1
Yim, Y.S.2
Tus, K.3
Wakeland, E.K.4
Ravetch, J.V.5
-
46
-
-
12344273445
-
The inhibitory Fcg receptor modulates autoimmunity by limiting the accumulation of immunoglobulin G+ anti-DNA plasma cells
-
Fukuyama, H., F. Nimmerjahn, and J. V. Ravetch. 2005. The inhibitory Fcg receptor modulates autoimmunity by limiting the accumulation of immunoglobulin G+ anti-DNA plasma cells. Nat. Immunol. 6: 99-106.
-
(2005)
Nat. Immunol.
, vol.6
, pp. 99-106
-
-
Fukuyama, H.1
Nimmerjahn, F.2
Ravetch, J.V.3
-
47
-
-
84858434212
-
FcgRIIB: A modulator of cell activation and humoral tolerance
-
Lehmann, B., I. Schwab, S. Böhm, A. Lux, M. Biburger, and F. Nimmerjahn. 2012. FcgRIIB: a modulator of cell activation and humoral tolerance. Expert Rev. Clin. Immunol. 8: 243-254.
-
(2012)
Expert Rev. Clin. Immunol.
, vol.8
, pp. 243-254
-
-
Lehmann, B.1
Schwab, I.2
Böhm, S.3
Lux, A.4
Biburger, M.5
Nimmerjahn, F.6
-
49
-
-
84861692916
-
B cell receptor signal transduction in the GC is short-circuited by high phosphatase activity
-
Khalil, A. M., J. C. Cambier, and M. J. Shlomchik. 2012. B cell receptor signal transduction in the GC is short-circuited by high phosphatase activity. Science 336: 1178-1181.
-
(2012)
Science
, vol.336
, pp. 1178-1181
-
-
Khalil, A.M.1
Cambier, J.C.2
Shlomchik, M.J.3
-
50
-
-
84857355337
-
Possible allelic structure of IgG2a and IgG2c in mice
-
Zhang, Z., T. Goldschmidt, and H. Salter. 2012. Possible allelic structure of IgG2a and IgG2c in mice. Mol. Immunol. 50: 169-171.
-
(2012)
Mol. Immunol.
, vol.50
, pp. 169-171
-
-
Zhang, Z.1
Goldschmidt, T.2
Salter, H.3
-
51
-
-
70249097764
-
B cell intrinsic MyD88 signals drive IFN-g production from T cells and control switching to IgG2c
-
Barr, T. A., S. Brown, P. Mastroeni, and D. Gray. 2009. B cell intrinsic MyD88 signals drive IFN-g production from T cells and control switching to IgG2c. J. Immunol. 183: 1005-1012.
-
(2009)
J. Immunol.
, vol.183
, pp. 1005-1012
-
-
Barr, T.A.1
Brown, S.2
Mastroeni, P.3
Gray, D.4
-
52
-
-
77954403513
-
Type i IFN enhances follicular B cell contribution to the T cell-independent antibody response
-
Swanson, C. L., T. J. Wilson, P. Strauch, M. Colonna, R. Pelanda, and R. M. Torres. 2010. Type I IFN enhances follicular B cell contribution to the T cell-independent antibody response. J. Exp. Med. 207: 1485-1500.
-
(2010)
J. Exp. Med.
, vol.207
, pp. 1485-1500
-
-
Swanson, C.L.1
Wilson, T.J.2
Strauch, P.3
Colonna, M.4
Pelanda, R.5
Torres, R.M.6
-
53
-
-
0032402990
-
Susceptibility to lupus nephritis in the NZB/W model system
-
Morel, L., and E. K. Wakeland. 1998. Susceptibility to lupus nephritis in the NZB/W model system. Curr. Opin. Immunol. 10: 718-725.
-
(1998)
Curr. Opin. Immunol.
, vol.10
, pp. 718-725
-
-
Morel, L.1
Wakeland, E.K.2
-
54
-
-
40049093765
-
Systemic lupus erythematosus
-
Rahman, A., and D. A. Isenberg. 2008. Systemic lupus erythematosus. N. Engl. J. Med. 358: 929-939.
-
(2008)
N. Engl. J. Med.
, vol.358
, pp. 929-939
-
-
Rahman, A.1
Isenberg, D.A.2
-
55
-
-
79551677401
-
Local renal autoantibody production in lupus nephritis
-
Espeli, M., S. Bökers, G. Giannico, H. A. Dickinson, V. Bardsley, A. B. Fogo, and K. G. Smith. 2011. Local renal autoantibody production in lupus nephritis. J. Am. Soc. Nephrol. 22: 296-305.
-
(2011)
J. Am. Soc. Nephrol.
, vol.22
, pp. 296-305
-
-
Espeli, M.1
Bökers, S.2
Giannico, G.3
Dickinson, H.A.4
Bardsley, V.5
Fogo, A.B.6
Smith, K.G.7
-
56
-
-
0034452815
-
Susceptibility to Leishmania major infection in the absence of IL-4
-
Noben-Trauth, N. 2000. Susceptibility to Leishmania major infection in the absence of IL-4. Immunol. Lett. 75: 41-44.
-
(2000)
Immunol. Lett.
, vol.75
, pp. 41-44
-
-
Noben-Trauth, N.1
-
57
-
-
0029004558
-
Prevention of autoimmune symptoms in autoimmune-prone mice by elimination of B-1 cells
-
Murakami, M., H. Yoshioka, T. Shirai, T. Tsubata, and T. Honjo. 1995. Prevention of autoimmune symptoms in autoimmune-prone mice by elimination of B-1 cells. Int. Immunol. 7: 877-882.
-
(1995)
Int. Immunol.
, vol.7
, pp. 877-882
-
-
Murakami, M.1
Yoshioka, H.2
Shirai, T.3
Tsubata, T.4
Honjo, T.5
|