Short-form RON overexpression augments benzyl isothiocyanate-induced apoptosis in human breast cancer cells

Molecular Carcinogenesis

Volumn 55, Issue 5, 2016, Pages 473-485

  Sehrawat, Anuradha ^a   Singh, Shivendra V ^b  

^a UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF PITTSBURGH CANCER INSTITUTE   (United States)

Author keywords

Apoptosis; Benzyl isothiocyanate; Chemoprevention; SfRON

Indexed keywords

BENZYL ISOTHIOCYANATE; MITOGEN ACTIVATED PROTEIN KINASE P38; PEPTIDES AND PROTEINS; PROTEIN BAK; PROTEIN BAX; REACTIVE OXYGEN METABOLITE; SHORT FORM RECEPTEUR D ORIGINE NANTAIS; STRESS ACTIVATED PROTEIN KINASE; UNCLASSIFIED DRUG; ISOTHIOCYANIC ACID DERIVATIVE; PROTEIN TYROSINE KINASE; RON PROTEIN;

APOPTOSIS; ARTICLE; BREAST CANCER CELL LINE; CONTROLLED STUDY; G2 PHASE CELL CYCLE CHECKPOINT; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; BREAST NEOPLASMS; CELL PROLIFERATION; DRUG EFFECTS; DRUG POTENTIATION; FEMALE; GENE EXPRESSION; GENETICS; HUMAN; MCF-7 CELL LINE; METABOLISM; TUMOR CELL LINE;

APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; BREAST NEOPLASMS; CELL LINE, TUMOR; CELL PROLIFERATION; DRUG SYNERGISM; FEMALE; GENE EXPRESSION; HUMANS; ISOTHIOCYANATES; MCF-7 CELLS; RECEPTOR PROTEIN-TYROSINE KINASES;

EID: 84927139102     PISSN: 08991987     EISSN: 10982744     Source Type: Journal    
DOI: 10.1002/mc.22295     Document Type: Article

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