-
1
-
-
0842320913
-
Postnatal development of inflammation in a murine model of Niemann-Pick type C disease: Immunohistochemical observations of microglia and astroglia
-
doi: 10.1016/s0014-4886(03)00345-5
-
Baudry, M., Yao, Y., Simmons, D., Liu, J., and Bi, X. (2003). Postnatal development of inflammation in a murine model of Niemann-Pick type C disease: immunohistochemical observations of microglia and astroglia. Exp. Neurol. 184, 887-903. doi: 10.1016/s0014-4886(03)00345-5
-
(2003)
Exp. Neurol.
, vol.184
, pp. 887-903
-
-
Baudry, M.1
Yao, Y.2
Simmons, D.3
Liu, J.4
Bi, X.5
-
2
-
-
84869500645
-
Connexin and pannexin hemichannels in inflammatory responses of glia and neurons
-
doi: 10.1016/j.brainres.2012.08.042
-
Bennett, M. V., Garre, J. M., Orellana, J. A., Bukauskas, F. F., Nedergaard, M., and Saez, J. C. (2012). Connexin and pannexin hemichannels in inflammatory responses of glia and neurons. Brain Res. 1487, 3-15. doi: 10.1016/j.brainres.2012.08.042
-
(2012)
Brain Res.
, vol.1487
, pp. 3-15
-
-
Bennett, M.V.1
Garre, J.M.2
Orellana, J.A.3
Bukauskas, F.F.4
Nedergaard, M.5
Saez, J.C.6
-
3
-
-
84899702659
-
Evidence for Aberrant astrocyte hemichannel activity in Juvenile Neuronal Ceroid Lipofuscinosis (JNCL)
-
doi: 10.1371/journal.pone.0095023
-
Burkovetskaya, M., Karpuk, N., Xiong, J., Bosch, M., Boska, M. D., Takeuchi, H., et al. (2014). Evidence for Aberrant astrocyte hemichannel activity in Juvenile Neuronal Ceroid Lipofuscinosis (JNCL). PLoS One 9: e95023. doi: 10.1371/journal.pone.0095023
-
(2014)
PLoS One
, vol.9
-
-
Burkovetskaya, M.1
Karpuk, N.2
Xiong, J.3
Bosch, M.4
Boska, M.D.5
Takeuchi, H.6
-
4
-
-
0141705404
-
Gating and regulation of connexin 43 (Cx43) hemichannels
-
doi: 10.1073/pnas.1434298100
-
Contreras, J. E., Saez, J. C., Bukauskas, F. F., and Bennett, M. V. (2003). Gating and regulation of connexin 43 (Cx43) hemichannels. Proc. Natl. Acad. Sci. U S A 100, 11388-11393. doi: 10.1073/pnas.1434298100
-
(2003)
Proc. Natl. Acad. Sci. U S A
, vol.100
, pp. 11388-11393
-
-
Contreras, J.E.1
Saez, J.C.2
Bukauskas, F.F.3
Bennett, M.V.4
-
5
-
-
0037107348
-
Cln3(Deltaex7/8) knock-in mice with the common JNCL mutation exhibit progressive neurologic disease that begins before birth
-
doi: 10.1093/hmg/11.22.2709
-
Cotman, S. L., Vrbanac, V., Lebel, L. A., Lee, R. L., Johnson, K. A., Donahue, L. R., et al. (2002). Cln3(Deltaex7/8) knock-in mice with the common JNCL mutation exhibit progressive neurologic disease that begins before birth. Hum. Mol. Genet. 11, 2709-2721. doi: 10.1093/hmg/11.22.2709
-
(2002)
Hum. Mol. Genet.
, vol.11
, pp. 2709-2721
-
-
Cotman, S.L.1
Vrbanac, V.2
Lebel, L.A.3
Lee, R.L.4
Johnson, K.A.5
Donahue, L.R.6
-
6
-
-
84883692214
-
A novel c. 1135_1138delCTGT mutation in CLN3 leads to juvenile neuronal ceroid lipofuscinosis
-
doi: 10.1177/0883073813494812
-
Drack, A. V., Miller, J. N., and Pearce, D. A. (2013). A novel c. 1135_1138delCTGT mutation in CLN3 leads to juvenile neuronal ceroid lipofuscinosis. J. Child Neurol. 28, 1112-1116. doi: 10.1177/0883073813494812
-
(2013)
J. Child Neurol.
, vol.28
, pp. 1112-1116
-
-
Drack, A.V.1
Miller, J.N.2
Pearce, D.A.3
-
7
-
-
84866064720
-
The role of gap junction channels during physiologic and pathologic conditions of the human central nervous system
-
doi: 10.1007/s11481-012-9352-5
-
Eugenin, E. A., Basilio, D., Saez, J. C., Orellana, J. A., Raine, C. S., Bukauskas, F., et al. (2012). The role of gap junction channels during physiologic and pathologic conditions of the human central nervous system. J. Neuroimmune Pharmacol. 7, 499-518. doi: 10.1007/s11481-012-9352-5
-
(2012)
J. Neuroimmune Pharmacol.
, vol.7
, pp. 499-518
-
-
Eugenin, E.A.1
Basilio, D.2
Saez, J.C.3
Orellana, J.A.4
Raine, C.S.5
Bukauskas, F.6
-
8
-
-
79953689889
-
Altered sensitivity of cerebellar granule cells to glutamate receptor overactivation in the Cln3(Deltaex7/8)-knock-in mouse model of juvenile neuronal ceroid lipofuscinosis
-
doi: 10.1016/j.neuint.2011.02.003
-
Finn, R., Kovacs, A. D., and Pearce, D. A. (2011). Altered sensitivity of cerebellar granule cells to glutamate receptor overactivation in the Cln3(Deltaex7/8)-knock-in mouse model of juvenile neuronal ceroid lipofuscinosis. Neurochem. Int. 58, 648-655. doi: 10.1016/j.neuint.2011.02.003
-
(2011)
Neurochem. Int.
, vol.58
, pp. 648-655
-
-
Finn, R.1
Kovacs, A.D.2
Pearce, D.A.3
-
9
-
-
84895418697
-
Functional analysis and regulation of purified connexin hemichannels
-
doi: 10.3389/fphys.2014.00071
-
Fiori, M. C., Reuss, L., Cuello, L. G., and Altenberg, G. A. (2014). Functional analysis and regulation of purified connexin hemichannels. Front. Physiol. 5: 71. doi: 10.3389/fphys.2014.00071
-
(2014)
Front. Physiol.
, vol.5
, pp. 71
-
-
Fiori, M.C.1
Reuss, L.2
Cuello, L.G.3
Altenberg, G.A.4
-
10
-
-
84881572026
-
Connexin and pannexin hemichannels in brain glial cells: Properties, pharmacology and roles
-
doi: 10.3389/fphar.2013.00088
-
Giaume, C., Leybaert, L., Naus, C. C., and Saez, J. C. (2013). Connexin and pannexin hemichannels in brain glial cells: properties, pharmacology and roles. Front. Pharmacol. 4: 88. doi: 10.3389/fphar.2013.00088
-
(2013)
Front. Pharmacol.
, vol.4
, pp. 88
-
-
Giaume, C.1
Leybaert, L.2
Naus, C.C.3
Saez, J.C.4
-
11
-
-
0030577376
-
A model for Batten disease protein CLN3: Functional implications from homology and mutations
-
doi: 10.1016/s0014-5793(96)01290-2
-
Janes, R. W., Munroe, P. B., Mitchison, H. M., Gardiner, R. M., Mole, S. E., and Wallace, B. A. (1996). A model for Batten disease protein CLN3: functional implications from homology and mutations. FEBS Lett. 399, 75-77. doi: 10.1016/s0014-5793(96)01290-2
-
(1996)
FEBS Lett.
, vol.399
, pp. 75-77
-
-
Janes, R.W.1
Munroe, P.B.2
Mitchison, H.M.3
Gardiner, R.M.4
Mole, S.E.5
Wallace, B.A.6
-
12
-
-
44649092806
-
Connexin 43 hemichannels are permeable to ATP
-
doi: 10.1523/JNEUROSCI.5048-07.2008
-
Kang, J., Kang, N., Lovatt, D., Torres, A., Zhao, Z., Lin, J., et al. (2008). Connexin 43 hemichannels are permeable to ATP. J. Neurosci. 28, 4702-4711. doi: 10.1523/JNEUROSCI.5048-07.2008
-
(2008)
J. Neurosci.
, vol.28
, pp. 4702-4711
-
-
Kang, J.1
Kang, N.2
Lovatt, D.3
Torres, A.4
Zhao, Z.5
Lin, J.6
-
13
-
-
79955605102
-
Physiology of microglia
-
doi: 10.1152/physrev.00011.2010
-
Kettenmann, H., Hanisch, U. K., Noda, M., and Verkhratsky, A. (2011). Physiology of microglia. Physiol. Rev. 91, 461-553. doi: 10.1152/physrev.00011.2010
-
(2011)
Physiol. Rev.
, vol.91
, pp. 461-553
-
-
Kettenmann, H.1
Hanisch, U.K.2
Noda, M.3
Verkhratsky, A.4
-
14
-
-
48249146963
-
Glial connexins and gap junctions in CNS inflammation and disease
-
doi: 10.1111/j.1471-4159.2008.05405.x
-
Kielian, T. (2008). Glial connexins and gap junctions in CNS inflammation and disease. J. Neurochem. 106, 1000-1016. doi: 10.1111/j.1471-4159.2008.05405.x
-
(2008)
J. Neurochem.
, vol.106
, pp. 1000-1016
-
-
Kielian, T.1
-
15
-
-
84896738387
-
Neuroinflammation: The role and consequences
-
doi: 10.1016/j.neures.2013.10.004
-
Lyman, M., Lloyd, D. G., Ji, X., Vizcaychipi, M. P., and Ma, D. (2014). Neuroinflammation: the role and consequences. Neurosci. Res. 79, 1-12. doi: 10.1016/j.neures.2013.10.004
-
(2014)
Neurosci. Res.
, vol.79
, pp. 1-12
-
-
Lyman, M.1
Lloyd, D.G.2
Ji, X.3
Vizcaychipi, M.P.4
Ma, D.5
-
16
-
-
84877120288
-
Connexin47 protein phosphorylation and stability in oligodendrocytes depend on expression of Connexin43 protein in astrocytes
-
doi: 10.1523/JNEUROSCI.5874-12.2013
-
May, D., Tress, O., Seifert, G., and Willecke, K. (2013). Connexin47 protein phosphorylation and stability in oligodendrocytes depend on expression of Connexin43 protein in astrocytes. J. Neurosci. 33, 7985-7996. doi: 10.1523/JNEUROSCI.5874-12.2013
-
(2013)
J. Neurosci.
, vol.33
, pp. 7985-7996
-
-
May, D.1
Tress, O.2
Seifert, G.3
Willecke, K.4
-
17
-
-
77957877855
-
Astroglial connexin immunoreactivity is specifically altered at beta-amyloid plaques in β-amyloid precursor protein/presenilin1 mice
-
doi: 10.1016/j.neuroscience.2010.08.001
-
Mei, X., Ezan, P., Giaume, C., and Koulakoff, A. (2010). Astroglial connexin immunoreactivity is specifically altered at beta-amyloid plaques in β-amyloid precursor protein/presenilin1 mice. Neuroscience 171, 92-105. doi: 10.1016/j.neuroscience.2010.08.001
-
(2010)
Neuroscience
, vol.171
, pp. 92-105
-
-
Mei, X.1
Ezan, P.2
Giaume, C.3
Koulakoff, A.4
-
18
-
-
0033585476
-
Prevalence of lysosomal storage disorders
-
doi: 10.1001/jama.281.3.249
-
Meikle, P. J., Hopwood, J. J., Clague, A. E., and Carey, W. F. (1999). Prevalence of lysosomal storage disorders. JAMA 281, 249-254. doi: 10.1001/jama.281.3.249
-
(1999)
JAMA
, vol.281
, pp. 249-254
-
-
Meikle, P.J.1
Hopwood, J.J.2
Clague, A.E.3
Carey, W.F.4
-
19
-
-
84872284081
-
Ceramide function in the brain: When a slight tilt is enough
-
doi: 10.1007/s00018-012-1038-x
-
Mencarelli, C., and Martinez-Martinez, P. (2013). Ceramide function in the brain: when a slight tilt is enough. Cell. Mol. Life Sci. 70, 181-203. doi: 10.1007/s00018-012-1038-x
-
(2013)
Cell. Mol. Life Sci.
, vol.70
, pp. 181-203
-
-
Mencarelli, C.1
Martinez-Martinez, P.2
-
20
-
-
84869500260
-
Functions of connexins and large pore channels on microglial cells: The gates to environment
-
doi: 10.1016/j.brainres.2012.07.020
-
Mika, T., and Prochnow, N. (2012). Functions of connexins and large pore channels on microglial cells: the gates to environment. Brain Res. 1487, 16-24. doi: 10.1016/j.brainres.2012.07.020
-
(2012)
Brain Res.
, vol.1487
, pp. 16-24
-
-
Mika, T.1
Prochnow, N.2
-
21
-
-
0029879282
-
Elevated connexin43 immunoreactivity at sites of amyloid plaques in Alzheimer's disease
-
doi: 10.1016/0006-8993(95)01526-4
-
Nagy, J. I., Li, W., Hertzberg, E. L., and Marotta, C. A. (1996). Elevated connexin43 immunoreactivity at sites of amyloid plaques in Alzheimer's disease. Brain Res. 717, 173-178. doi: 10.1016/0006-8993(95)01526-4
-
(1996)
Brain Res.
, vol.717
, pp. 173-178
-
-
Nagy, J.I.1
Li, W.2
Hertzberg, E.L.3
Marotta, C.A.4
-
22
-
-
80051759671
-
ATP and glutamate released via astroglial connexin 43 hemichannels mediate neuronal death through activation of pannexin 1 hemichannels
-
doi: 10.1111/j.1471-4159.2011.07210.x
-
Orellana, J. A., Froger, N., Ezan, P., Jiang, J. X., Bennett, M. V., Naus, C. C., et al. (2011a). ATP and glutamate released via astroglial connexin 43 hemichannels mediate neuronal death through activation of pannexin 1 hemichannels. J. Neurochem. 118, 826-840. doi: 10.1111/j.1471-4159.2011.07210.x
-
(2011)
J. Neurochem.
, vol.118
, pp. 826-840
-
-
Orellana, J.A.1
Froger, N.2
Ezan, P.3
Jiang, J.X.4
Bennett, M.V.5
Naus, C.C.6
-
23
-
-
58149100746
-
Modulation of brain hemichannels and gap junction channels by pro-inflammatory agents and their possible role in neurodegeneration
-
doi: 10.1089/ars.2008.2130
-
Orellana, J. A., Saez, P. J., Shoji, K. F., Schalper, K. A., Palacios-Prado, N., Velarde, V., et al. (2009). Modulation of brain hemichannels and gap junction channels by pro-inflammatory agents and their possible role in neurodegeneration. Antioxid. Redox Signal. 11, 369-399. doi: 10.1089/ars.2008.2130
-
(2009)
Antioxid. Redox Signal.
, vol.11
, pp. 369-399
-
-
Orellana, J.A.1
Saez, P.J.2
Shoji, K.F.3
Schalper, K.A.4
Palacios-Prado, N.5
Velarde, V.6
-
24
-
-
79955739803
-
Amyloid β-induced death in neurons involves glial and neuronal hemichannels
-
doi: 10.1523/JNEUROSCI.6417-10.2011
-
Orellana, J. A., Shoji, K. F., Abudara, V., Ezan, P., Amigou, E., Saez, P. J., et al. (2011b). Amyloid β-induced death in neurons involves glial and neuronal hemichannels. J. Neurosci. 31, 4962-4977. doi: 10.1523/JNEUROSCI.6417-10.2011
-
(2011)
J. Neurosci.
, vol.31
, pp. 4962-4977
-
-
Orellana, J.A.1
Shoji, K.F.2
Abudara, V.3
Ezan, P.4
Amigou, E.5
Saez, P.J.6
-
25
-
-
4544285361
-
Late onset neurodegeneration in the Cln3-/-mouse model of juvenile neuronal ceroid lipofuscinosis is preceded by low level glial activation
-
doi: 10.1016/j.brainres.2004.07.030
-
Pontikis, C. C., Cella, C. V., Parihar, N., Lim, M. J., Chakrabarti, S., Mitchison, H. M., et al. (2004). Late onset neurodegeneration in the Cln3-/-mouse model of juvenile neuronal ceroid lipofuscinosis is preceded by low level glial activation. Brain Res. 1023, 231-242. doi: 10.1016/j.brainres.2004.07.030
-
(2004)
Brain Res.
, vol.1023
, pp. 231-242
-
-
Pontikis, C.C.1
Cella, C.V.2
Parihar, N.3
Lim, M.J.4
Chakrabarti, S.5
Mitchison, H.M.6
-
26
-
-
84871658247
-
Understanding risk factors for Alzheimer's disease: Interplay of neuroinflammation, connexin-based communication and oxidative stress
-
doi: 10.1016/j.arcmed.2012.10.016
-
Quintanilla, R. A., Orellana, J. A., and von Bernhardi, R. (2012). Understanding risk factors for Alzheimer's disease: interplay of neuroinflammation, connexin-based communication and oxidative stress. Arch. Med. Res. 43, 632-644. doi: 10.1016/j.arcmed.2012.10.016
-
(2012)
Arch. Med. Res.
, vol.43
, pp. 632-644
-
-
Quintanilla, R.A.1
Orellana, J.A.2
von Bernhardi, R.3
-
27
-
-
37249015389
-
Cx43 hemichannels and gap junction channels in astrocytes are regulated oppositely by proinflammatory cytokines released from activated microglia
-
doi: 10.1523/jneurosci.2042-07.2007
-
Retamal, M. A., Froger, N., Palacios-Prado, N., Ezan, P., Saez, P. J., Saez, J. C., et al. (2007). Cx43 hemichannels and gap junction channels in astrocytes are regulated oppositely by proinflammatory cytokines released from activated microglia. J. Neurosci. 27, 13781-13792. doi: 10.1523/jneurosci.2042-07.2007
-
(2007)
J. Neurosci.
, vol.27
, pp. 13781-13792
-
-
Retamal, M.A.1
Froger, N.2
Palacios-Prado, N.3
Ezan, P.4
Saez, P.J.5
Saez, J.C.6
-
28
-
-
79851476221
-
Niemann-Pick type C disease: Molecular mechanisms and potential therapeutic approaches
-
doi: 10.1111/j.1471-4159.2010.06976.x
-
Rosenbaum, A. I., and Maxfield, F. R. (2011). Niemann-Pick type C disease: molecular mechanisms and potential therapeutic approaches. J. Neurochem. 116, 789-795. doi: 10.1111/j.1471-4159.2010.06976.x
-
(2011)
J. Neurochem.
, vol.116
, pp. 789-795
-
-
Rosenbaum, A.I.1
Maxfield, F.R.2
-
29
-
-
0036872333
-
Gap junctions and connexin expression in the normal and pathological central nervous system
-
doi: 10.1016/s0248-4900(02)00016-3
-
Rouach, N., Avignone, E., Meme, W., Koulakoff, A., Venance, L., Blomstrand, F., et al. (2002). Gap junctions and connexin expression in the normal and pathological central nervous system. Biol. Cell. 94, 457-475. doi: 10.1016/s0248-4900(02)00016-3
-
(2002)
Biol. Cell.
, vol.94
, pp. 457-475
-
-
Rouach, N.1
Avignone, E.2
Meme, W.3
Koulakoff, A.4
Venance, L.5
Blomstrand, F.6
-
30
-
-
57349185900
-
Astroglial metabolic networks sustain hippocampal synaptic transmission
-
doi: 10.1126/science.1164022
-
Rouach, N., Koulakoff, A., Abudara, V., Willecke, K., and Giaume, C. (2008). Astroglial metabolic networks sustain hippocampal synaptic transmission. Science 322, 1551-1555. doi: 10.1126/science.1164022
-
(2008)
Science
, vol.322
, pp. 1551-1555
-
-
Rouach, N.1
Koulakoff, A.2
Abudara, V.3
Willecke, K.4
Giaume, C.5
-
31
-
-
84881601333
-
Disruption in connexin-based communication is associated with intracellular Ca(2)(+) signal alterations in astrocytes from Niemann-Pick type C mice
-
doi: 10.1371/journal.pone.0071361
-
Sáez, P. J., Orellana, J. A., Vega-Riveros, N., Figueroa, V. A., Hernandez, D. E., Castro, J. F., et al. (2013a). Disruption in connexin-based communication is associated with intracellular Ca(2)(+) signal alterations in astrocytes from Niemann-Pick type C mice. PLoS One 8: e71361. doi: 10.1371/journal.pone.0071361
-
(2013)
PLoS One
, vol.8
-
-
Sáez, P.J.1
Orellana, J.A.2
Vega-Riveros, N.3
Figueroa, V.A.4
Hernandez, D.E.5
Castro, J.F.6
-
32
-
-
84877970834
-
ATP is required and advances cytokine-induced gap junction formation in microglia in vitro
-
doi: 10.1155/2013/216402
-
Sáez, P. J., Shoji, K. F., Retamal, M. A., Harcha, P. A., Ramirez, G., Jiang, J. X., et al. (2013b). ATP is required and advances cytokine-induced gap junction formation in microglia in vitro. Mediators Inflamm. 2013: 216402. doi: 10.1155/2013/216402
-
(2013)
Mediators Inflamm.
, vol.2013
, pp. 216402
-
-
Sáez, P.J.1
Shoji, K.F.2
Retamal, M.A.3
Harcha, P.A.4
Ramirez, G.5
Jiang, J.X.6
-
33
-
-
70349253972
-
Metabolic inhibition increases activity of connexin-32 hemichannels permeable to Ca2+ in transfected HeLa cells
-
doi: 10.1152/ajpcell.00200.2009
-
Sánchez, H. A., Orellana, J. A., Verselis, V. K., and Saez, J. C. (2009). Metabolic inhibition increases activity of connexin-32 hemichannels permeable to Ca2+ in transfected HeLa cells. Am. J. Physiol. Cell Physiol. 297, C665-C678. doi: 10.1152/ajpcell.00200.2009
-
(2009)
Am. J. Physiol. Cell Physiol.
, vol.297
-
-
Sánchez, H.A.1
Orellana, J.A.2
Verselis, V.K.3
Saez, J.C.4
-
34
-
-
41049095852
-
CNS-directed gene therapy for lysosomal storage diseases
-
doi: 10.1111/j.1651-2227.2008.00660.x
-
Sands, M. S., and Haskins, M. E. (2008). CNS-directed gene therapy for lysosomal storage diseases. Acta Paediatr. Suppl. 97, 22-27. doi: 10.1111/j.1651-2227.2008.00660.x
-
(2008)
Acta Paediatr. Suppl.
, vol.97
, pp. 22-27
-
-
Sands, M.S.1
Haskins, M.E.2
-
35
-
-
78649653091
-
Connexin 43 hemichannels mediate the Ca2+ influx induced by extracellular alkalinization
-
doi: 10.1152/ajpcell.00015.2010
-
Schalper, K. A., Sanchez, H. A., Lee, S. C., Altenberg, G. A., Nathanson, M. H., and Saez, J. C. (2010). Connexin 43 hemichannels mediate the Ca2+ influx induced by extracellular alkalinization. Am. J. Physiol. Cell Physiol. 299, C1504-C1515. doi: 10.1152/ajpcell.00015.2010
-
(2010)
Am. J. Physiol. Cell Physiol.
, vol.299
-
-
Schalper, K.A.1
Sanchez, H.A.2
Lee, S.C.3
Altenberg, G.A.4
Nathanson, M.H.5
Saez, J.C.6
-
36
-
-
0035081475
-
Alzheimer's disease results from the cerebral accumulation and cytotoxicity of amyloid beta-protein
-
Selkoe, D. J. (2001). Alzheimer's disease results from the cerebral accumulation and cytotoxicity of amyloid beta-protein. J. Alzheimers Dis. 3, 75-80.
-
(2001)
J. Alzheimers Dis.
, vol.3
, pp. 75-80
-
-
Selkoe, D.J.1
-
37
-
-
56349119351
-
Linking Abeta and tau in late-onset Alzheimer's disease: A dual pathway hypothesis
-
doi: 10.1016/j.neuron.2008.11.007
-
Small, S. A., and Duff, K. (2008). Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis. Neuron 60, 534-542. doi: 10.1016/j.neuron.2008.11.007
-
(2008)
Neuron
, vol.60
, pp. 534-542
-
-
Small, S.A.1
Duff, K.2
-
38
-
-
79959359691
-
Blockade of gap junction hemichannel suppresses disease progression in mouse models of amyotrophic lateral sclerosis and Alzheimer's disease
-
doi: 10.1371/journal.pone.0021108
-
Takeuchi, H., Mizoguchi, H., Doi, Y., Jin, S., Noda, M., Liang, J., et al. (2011). Blockade of gap junction hemichannel suppresses disease progression in mouse models of amyotrophic lateral sclerosis and Alzheimer's disease. PLoS One 6: e21108. doi: 10.1371/journal.pone.0021108
-
(2011)
PLoS One
, vol.6
-
-
Takeuchi, H.1
Mizoguchi, H.2
Doi, Y.3
Jin, S.4
Noda, M.5
Liang, J.6
-
39
-
-
84874587315
-
The missing link in the amyloid cascade of Alzheimer's disease-metal ions
-
doi: 10.1016/j.neuint.2013.01.023
-
Tiiman, A., Palumaa, P., and Tougu, V. (2013). The missing link in the amyloid cascade of Alzheimer's disease-metal ions. Neurochem. Int. 62, 367-378. doi: 10.1016/j.neuint.2013.01.023
-
(2013)
Neurochem. Int.
, vol.62
, pp. 367-378
-
-
Tiiman, A.1
Palumaa, P.2
Tougu, V.3
-
40
-
-
84857977037
-
Alzheimer's disease: Redox dysregulation as a common denominator for diverse pathogenic mechanisms
-
doi: 10.1089/ars.2011.4082
-
von Bernhardi, R., and Eugenin, J. (2012). Alzheimer's disease: redox dysregulation as a common denominator for diverse pathogenic mechanisms. Antioxid. Redox Signal. 16, 974-1031. doi: 10.1089/ars.2011.4082
-
(2012)
Antioxid. Redox Signal.
, vol.16
, pp. 974-1031
-
-
von Bernhardi, R.1
Eugenin, J.2
-
41
-
-
67649488309
-
Abeta43 is more frequent than Abeta40 in amyloid plaque cores from Alzheimer disease brains
-
doi: 10.1111/j.1471-4159.2009.06170.x
-
Welander, H., Franberg, J., Graff, C., Sundstrom, E., Winblad, B., and Tjernberg, L. O. (2009). Abeta43 is more frequent than Abeta40 in amyloid plaque cores from Alzheimer disease brains. J. Neurochem. 110, 697-706. doi: 10.1111/j.1471-4159.2009.06170.x
-
(2009)
J. Neurochem.
, vol.110
, pp. 697-706
-
-
Welander, H.1
Franberg, J.2
Graff, C.3
Sundstrom, E.4
Winblad, B.5
Tjernberg, L.O.6
-
42
-
-
84885424533
-
Microglia in juvenile neuronal ceroid lipofuscinosis are primed toward a pro-inflammatory phenotype
-
doi: 10.1111/jnc.12385
-
Xiong, J., and Kielian, T. (2013). Microglia in juvenile neuronal ceroid lipofuscinosis are primed toward a pro-inflammatory phenotype. J. Neurochem. 127, 245-258. doi: 10.1111/jnc.12385
-
(2013)
J. Neurochem.
, vol.127
, pp. 245-258
-
-
Xiong, J.1
Kielian, T.2
|