Early and persistent abnormal decoding by glial cells at the neuromuscular junction in an ALS model

Journal of Neuroscience

Volumn 35, Issue 2, 2015, Pages 688-706

  Arbour, Danielle ^a   Tremblay, Elsa ^a   Martineau, Éric ^a   Julien, Jean Pierre ^b   Robitaille, Richard ^a  

^a UNIVERSITÉ DE MONTRÉAL   (Canada)

^b UNIVERSITÉ LAVAL   (Canada)

Author keywords

Motor unit properties; Muscarinic receptors; Perisynaptic Schwann cells; SOD1; Synaptic transmission

Indexed keywords

AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN ELECTROPHYSIOLOGY; CONTROLLED STUDY; DENERVATION; ENDPLATE POTENTIAL; GLIA CELL; IMMUNOHISTOCHEMISTRY; MALE; MOTOR PERFORMANCE; MOUSE; NERVE ENDING; NEUROMUSCULAR SYNAPSE; NONHUMAN; PRIORITY JOURNAL; SCHWANN CELL; SYNAPTIC POTENTIAL; SYNAPTIC TRANSMISSION; ANIMAL; GENETICS; METABOLISM; PATHOPHYSIOLOGY; PHYSIOLOGY;

CALCIUM; COPPER ZINC SUPEROXIDE DISMUTASE; MUSCARINIC RECEPTOR; SUPEROXIDE DISMUTASE;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; CALCIUM; MICE; NEUROMUSCULAR JUNCTION; RECEPTORS, MUSCARINIC; SCHWANN CELLS; SUPEROXIDE DISMUTASE; SYNAPTIC POTENTIALS;

EID: 84920996952     PISSN: 02706474     EISSN: 15292401     Source Type: Journal    
DOI: 10.1523/JNEUROSCI.1379-14.2015     Document Type: Article

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