Prenatal exposure to arsenic and cadmium impacts infectious disease-related genes within the glucocorticoid receptor signal transduction pathway

International Journal of Molecular Sciences

Volumn 15, Issue 12, 2014, Pages 22374-22391

  Rager, Julia E ^a   Yosim, Andrew ^a   Fry, Rebecca C ^a,b  

^a University of North Carolina at Chapel Hill   (United States)

^b UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

Author keywords

Arsenic; Cadmium; Environmental toxicant; Epigenome; Genome; Glucocorticoid receptor; In utero; Infectious disease; Pathway; Signal transduction

Indexed keywords

ARSENIC; CADMIUM; CASPASE 9; GAMMA INTERFERON; GAMMA INTERFERON RECEPTOR 1; GLUCOCORTICOID RECEPTOR; INTERCELLULAR ADHESION MOLECULE 1; INTERLEUKIN 1BETA; INTERLEUKIN 8; PEPTIDASE; STAT1 PROTEIN; TOLL LIKE RECEPTOR; TUMOR NECROSIS FACTOR; TUMOR NECROSIS FACTOR RECEPTOR 1;

ARTICLE; DISEASE PREDISPOSITION; DNA METHYLATION; EPIGENETICS; GENE EXPRESSION; GENOTYPE ENVIRONMENT INTERACTION; HUMAN; IMMUNOCOMPETENT CELL; INFECTION; LEUKOCYTE; MAJOR HISTOCOMPATIBILITY COMPLEX; NONHUMAN; PREGNANCY; PRENATAL EXPOSURE; PROTEIN EXPRESSION; RISK; SIGNAL TRANSDUCTION; COHORT ANALYSIS; COMMUNICABLE DISEASE; DRUG EFFECTS; FEMALE; GENE REGULATORY NETWORK; GENETICS; METABOLISM; REPRODUCIBILITY; RISK FACTOR; TOXICOGENETICS;

BACTERIA (MICROORGANISMS);

ARSENIC; CADMIUM; COHORT STUDIES; COMMUNICABLE DISEASES; FEMALE; GENE REGULATORY NETWORKS; HUMANS; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; RECEPTORS, GLUCOCORTICOID; REPRODUCIBILITY OF RESULTS; RISK FACTORS; SIGNAL TRANSDUCTION; TOXICOGENETICS;

EID: 84919362783     PISSN: 16616596     EISSN: 14220067     Source Type: Journal    
DOI: 10.3390/ijms151222374     Document Type: Article

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