Carbon monoxide offers neuroprotection from hippocampal cell damage induced by recurrent febrile seizures through the PERK-activated ER stress pathway

Neuroscience Letters

Volumn 585, Issue , 2015, Pages 126-131

  Han, Ying ^a   Yi, Wenxia ^a   Qin, Jiong ^a   Zhao, Yang ^a   Zhang, Jing ^a   Chang, Xingzhi ^a  

^a PEKING UNIVERSITY FIRST HOSPITAL   (China)

Author keywords

Carbon monoxide; Endoplasmic reticulum stress; Febrile seizure; Hippocampus; Neuronal damage; PERK

Indexed keywords

CARBON MONOXIDE; CCAAT ENHANCER BINDING PROTEIN; ENZYME INHIBITOR; GLUCOSE REGULATED PROTEIN 78; HEMIN; INITIATION FACTOR 2; PROTEIN KINASE; PROTEIN KINASE RNA LIKE ENDOPLASMIC RETICULUM KINASE; UNCLASSIFIED DRUG; PERK KINASE; PROTEIN KINASE R;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL PROTECTION; CONTROLLED STUDY; DOWN REGULATION; ENDOPLASMIC RETICULUM STRESS; EUKARYOTE; FEBRILE CONVULSION; HIPPOCAMPUS; MALE; NERVE CELL LESION; NEUROPROTECTION; NONHUMAN; RAT; RECURRENT DISEASE; UNFOLDED PROTEIN RESPONSE; UPREGULATION; ANIMAL; BLOOD; METABOLISM; NERVE CELL; PATHOLOGY; PHOSPHORYLATION; SIGNAL TRANSDUCTION; SPRAGUE DAWLEY RAT;

ANIMALS; APOPTOSIS; CARBON MONOXIDE; EIF-2 KINASE; ENDOPLASMIC RETICULUM STRESS; HIPPOCAMPUS; MALE; NEURONS; PHOSPHORYLATION; RATS, SPRAGUE-DAWLEY; RECURRENCE; SEIZURES, FEBRILE; SIGNAL TRANSDUCTION;

EID: 84914816379     PISSN: 03043940     EISSN: 18727972     Source Type: Journal    
DOI: 10.1016/j.neulet.2014.11.040     Document Type: Article

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