Erratum: CXCL1 contributes to host defense in polymicrobial sepsis via modulating T cell and neutrophil functions (Journal of Immunology 193 (3549-3558) DOI: 10.4049/jimmunol.1401138);CXCL1 contributes to host defense in polymicrobial sepsis via modulating T cell and neutrophil functions

Journal of Immunology

Volumn 193, Issue 7, 2014, Pages 3549-3558

  Jin, Liliang ^a   Batra, Sanjay ^a   Douda, David Nobuhiro ^b,c   Palaniyar, Nades ^b,c   Jeyaseelan, Samithamby ^a,d  

^a LOUISIANA STATE UNIVERSITY   (United States)

^b HOSPITAL FOR SICK CHILDREN RESEARCH INSTITUTE   (Canada)

^c UNIVERSITY OF TORONTO   (Canada)

^d LOUISIANA STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CXCL1 CHEMOKINE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERCELLULAR ADHESION MOLECULE 1; INTERLEUKIN 17; MITOGEN ACTIVATED PROTEIN KINASE; PLACEBO; REACTIVE OXYGEN METABOLITE; RECOMBINANT PROTEIN; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; CXCL1 PROTEIN, MOUSE; IL17A PROTEIN, MOUSE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIBACTERIAL ACTIVITY; ARTICLE; BACTERIAL CLEARANCE; CECUM; CECUM LIGATION; CECUM PUNCTURE; CELLULAR IMMUNITY; CONTROLLED STUDY; CXCL1 GENE; CYTOKINE PRODUCTION; CYTOKINE RELEASE; DRUG EFFICACY; DRUG TARGETING; EXPERIMENTAL LIGATION; GENE DELETION; HOST PATHOGEN INTERACTION; HOST RESISTANCE; IMMUNOMODULATION; INFLAMMATION; LYMPHOCYTE DIFFERENTIATION; MALE; MOUSE; MOUSE MODEL; NEUTROPHIL CHEMOTAXIS; NONHUMAN; POLYMICROBIAL SEPSIS; PROTEIN DETERMINATION; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; PUNCTURE; SEPSIS; SURVIVAL RATE; T LYMPHOCYTE; TH17 CELL; UPREGULATION; ANIMAL; BLOOD; CELL DIFFERENTIATION; CELL MOTION; GENETICS; IMMUNOLOGY; KNOCKOUT MOUSE; METABOLISM; MICROBIOLOGY; NEUTROPHIL; PATHOLOGY; SIGNAL TRANSDUCTION;

ANIMALS; CELL DIFFERENTIATION; CELL MOVEMENT; CHEMOKINE CXCL1; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; INTERLEUKIN-17; MAP KINASE SIGNALING SYSTEM; MICE; MICE, KNOCKOUT; NEUTROPHILS; NF-KAPPA B; SEPSIS; TH17 CELLS;

EID: 84907222909     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.2101213     Document Type: Erratum

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