-
1
-
-
0042709605
-
Molecular mechanisms of amyloidosis
-
DOI 10.1056/NEJMra023144
-
Merlini G, Bellotti V. Molecular mechanisms of amyloidosis. N Engl J Med. 2003;349(6):583-596. (Pubitemid 36951371)
-
(2003)
New England Journal of Medicine
, vol.349
, Issue.6
, pp. 583-596
-
-
Merlini, G.1
Bellotti, V.2
-
2
-
-
48149110688
-
Amyloidosis: Is a cure possible?
-
Merlini G, Palladini G. Amyloidosis: is a cure possible? Ann Oncol. 2008;19(suppl 4):iv63-iv66.
-
(2008)
Ann Oncol.
, vol.19
, Issue.SUPPL. 4
-
-
Merlini, G.1
Palladini, G.2
-
3
-
-
79955842001
-
Amyloidosis: Pathogenesis and new therapeutic options
-
Merlini G, Seldin DC, Gertz MA. Amyloidosis: pathogenesis and new therapeutic options. J Clin Oncol. 2011;29(14):1924-1933.
-
(2011)
J Clin Oncol.
, vol.29
, Issue.14
, pp. 1924-1933
-
-
Merlini, G.1
Seldin, D.C.2
Gertz, M.A.3
-
4
-
-
84871772553
-
New criteria for response to treatment in immunoglobulin light chain amyloidosis based on free light chain measurement and cardiac biomarkers: Impact on survival outcomes
-
Palladini G, Dispenzieri A, Gertz MA, et al. New criteria for response to treatment in immunoglobulin light chain amyloidosis based on free light chain measurement and cardiac biomarkers: impact on survival outcomes. J Clin Oncol. 2012;30(36):4541-4549.
-
(2012)
J Clin Oncol.
, vol.30
, Issue.36
, pp. 4541-4549
-
-
Palladini, G.1
Dispenzieri, A.2
Gertz, M.A.3
-
5
-
-
33646597285
-
Circulating amyloidogenic free light chains and serum N-terminal natriuretic peptide type B decrease simultaneously in association with improvement of survival in AL
-
DOI 10.1182/blood-2005-11-4385
-
Palladini G, Lavatelli F, Russo P, et al. Circulatingamyloidogenic free light chains and serum N-terminal natriuretic peptide type B decreasesimultaneously in association with improvement of survival in AL. Blood. 2006;107(10):3854-3858. (Pubitemid 43726787)
-
(2006)
Blood
, vol.107
, Issue.10
, pp. 3854-3858
-
-
Palladini, G.1
Lavatelli, F.2
Russo, P.3
Perlini, S.4
Perfetti, V.5
Bosoni, T.6
Obici, L.7
Bradwell, A.R.8
D'Eril, G.M.9
Fogari, R.10
Moratti, R.11
Merlini, G.12
-
6
-
-
0035797855
-
Infusion of light chains from patients with cardiac amyloidosis causes diastolic dysfunction in isolated mouse hearts
-
Liao R, Jain M, Teller P, et al. Infusion of light chains from patients with cardiac amyloidosis causes diastolic dysfunction in isolated mousehearts. Circulation. 2001;104(14):1594-1597. (Pubitemid 32947519)
-
(2001)
Circulation
, vol.104
, Issue.14
, pp. 1594-1597
-
-
Liao, R.1
Jain, M.2
Teller, P.3
Connors, L.H.4
Ngoy, S.5
Skinner, M.6
Falk, R.H.7
Apstein, C.S.8
-
7
-
-
2342525940
-
Human Amyloidogenic Light Chains Directly Impair Cardiomyocyte Function Through an Increase in Cellular Oxidant Stress
-
DOI 10.1161/01.RES.0000126569.75419.74
-
Brenner DA, Jain M, Pimentel DR, et al. Human amyloidogenic light chains directly impair cardiomyocyte function through an increase in cellular oxidant stress. Circ Res. 2004;94(8): 1008-1010. (Pubitemid 38579694)
-
(2004)
Circulation Research
, vol.94
, Issue.8
, pp. 1008-1010
-
-
Brenner, D.A.1
Jain, M.2
Pimentel, D.R.3
Wang, B.4
Connors, L.H.5
Skinner, M.6
Apstein, C.S.7
Liao, R.8
-
8
-
-
77749251993
-
Amyloidogenic light chains induce cardiomyocyte contractile dysfunction and apoptosis via a non-canonical p38alpha MAPK pathway
-
Shi J, Guan J, Jiang B, et al. Amyloidogenic light chains induce cardiomyocyte contractile dysfunction and apoptosis via a non-canonical p38alpha MAPK pathway. Proc Natl Acad Sci USA. 2010;107(9):4188-4193.
-
(2010)
Proc Natl Acad Sci USA
, vol.107
, Issue.9
, pp. 4188-4193
-
-
Shi, J.1
Guan, J.2
Jiang, B.3
-
9
-
-
84882528609
-
Stanniocalcin1 is a key mediator of amyloidogenic light chain induced cardiotoxicity
-
Guan J, Mishra S, Shi J, et al. Stanniocalcin1 is a key mediator of amyloidogenic light chain induced cardiotoxicity. Basic Res Cardiol. 2013; 108(5):378.
-
(2013)
Basic Res Cardiol.
, vol.108
, Issue.5
, pp. 378
-
-
Guan, J.1
Mishra, S.2
Shi, J.3
-
10
-
-
0036682487
-
Analysis of V-J expression in plasma cells from primary (AL) amyloidosis and normal bone marrow identifies 3r (III) as a new amyloid-associated germline gene segment
-
DOI 10.1182/blood-2002-01-0114
-
Perfetti V, Casarini S, Palladini G, et al. Analysis of V(lambda)-J(lambda) expression in plasma cells from primary (AL) amyloidosis and normal bone marrow identifies 3r (lambdaIII) as a new amyloid-associated germline gene segment. Blood. 2002;100(3):948-953. (Pubitemid 34832622)
-
(2002)
Blood
, vol.100
, Issue.3
, pp. 948-953
-
-
Perfetti, V.1
Casarini, S.2
Palladini, G.3
Vignarelli, M.C.4
Klersy, C.5
Diegoli, M.6
Ascari, E.7
Merlini, G.8
-
11
-
-
84855614136
-
The repertoire of l light chains causing predominant amyloid heart involvement and identification of a preferentially involved germline gene IGLV1-44
-
Perfetti V, Palladini G, Casarini S, et al. The repertoire of l light chains causing predominant amyloid heart involvement and identification of a preferentially involved germline gene, IGLV1-44. Blood. 2012;119(1):144-150.
-
(2012)
Blood
, vol.119
, Issue.1
, pp. 144-150
-
-
Perfetti, V.1
Palladini, G.2
Casarini, S.3
-
12
-
-
38449086995
-
The C. Elegans pharynx: A model for organogenesis
-
Mango SE. The C. elegans pharynx: a model for organogenesis. WormBook. 2007;1-26.
-
(2007)
WormBook.
, pp. 1-26
-
-
Mango, S.E.1
-
13
-
-
0043270437
-
Food transport in the C. Elegans pharynx
-
DOI 10.1242/jeb.00433
-
Avery L, Shtonda BB. Food transport in the C. elegans pharynx. J Exp Biol. 2003;206(Pt 14): 2441-2457. (Pubitemid 36914464)
-
(2003)
Journal of Experimental Biology
, vol.206
, Issue.14
, pp. 2441-2457
-
-
Avery, L.1
Shtonda, B.B.2
-
14
-
-
0033168208
-
Feeding is inhibited by sublethal concentrations of toxicants and by heat stress in the nematode Caenorhabditis elegans: Relationship to the cellular stress response
-
DOI 10.1002/(SICI)1097-010X(19990701)284:2<147::AID-JEZ4>3.0.CO;2-Z
-
Jones D, Candido EP. Feeding is inhibited by sublethal concentrations of toxicants and by heat stress in the nematode Caenorhabditis elegans: relationship to the cellular stress response. J Exp Zool. 1999;284(2):147-157. (Pubitemid 29311513)
-
(1999)
Journal of Experimental Zoology
, vol.284
, Issue.2
, pp. 147-157
-
-
Jones, D.1
Candido, E.P.M.2
-
15
-
-
23244448608
-
th International Symposium on Amyloid and Amyloidosis
-
DOI 10.1002/ajh.20381
-
Gertz MA, Comenzo R, Falk RH, et al. Definition of organ involvement and treatment response in immunoglobulin light chain amyloidosis (AL): a consensus opinion from the 10th International Symposium on Amyloid and Amyloidosis, Tours, France, 18-22 April 2004. Am J Hematol. 2005; 79(4):319-328. (Pubitemid 41098849)
-
(2005)
American Journal of Hematology
, vol.79
, Issue.4
, pp. 319-328
-
-
Gertz, M.A.1
Comenzo, R.2
Falk, R.H.3
Fermand, J.P.4
Hazenberg, B.P.5
Hawkins, P.N.6
Merlini, G.7
Moreau, P.8
Ronco, P.9
Sanchorawala, V.10
Sezer, O.11
Solomon, A.12
Grateau, G.13
-
16
-
-
79952007082
-
Definition of organ involvement and response to treatment in AL amyloidosis: An updated consensus opinion [abstract]
-
Gertz MA, Merlini G. Definition of organ involvement and response to treatment in AL amyloidosis: an updated consensus opinion [abstract]. Amyloid. 2010;17(s):48.
-
(2010)
Amyloid
, vol.17
, Issue.S
, pp. 48
-
-
Gertz, M.A.1
Merlini, G.2
-
17
-
-
0038518441
-
Serum N-terminal pro-brain natriuretic peptide is a sensitive marker of myocardial dysfunction in AL amyloidosis
-
DOI 10.1161/01.CIR.0000068314.02595.B2
-
Palladini G, Campana C, Klersy C, et al. Serum N-terminal pro-brain natriuretic peptide is a sensitive marker of myocardial dysfunction in ALamyloidosis. Circulation. 2003;107(19): 2440-2445. (Pubitemid 36605226)
-
(2003)
Circulation
, vol.107
, Issue.19
, pp. 2440-2445
-
-
Palladini, G.1
Campana, C.2
Klersy, C.3
Balduini, A.4
Vadacca, G.5
Perfetti, V.6
Perlini, S.7
Obici, L.8
Ascari, E.9
D'Eril, G.M.10
Moratti, R.11
Merlini, G.12
-
18
-
-
84884695112
-
A strategy for synthesis of pathogenic human immunoglobulin free light chains in E. Coli
-
Rognoni P, Lavatelli F, Casarini S, et al. A strategy for synthesis of pathogenic human immunoglobulin free light chains in E. coli. PLoS ONE. 2013;8(9):e76022.
-
(2013)
PLoS One
, vol.8
, Issue.9
-
-
Rognoni, P.1
Lavatelli, F.2
Casarini, S.3
-
19
-
-
27744561904
-
Clinical aspects of systemic amyloid diseases
-
DOI 10.1016/j.bbapap.2005.08.014, PII S1570963905002815, Dialysis-related Amyloidosis: from Molecular Mechanisms to Therapies
-
Obici L, Perfetti V, Palladini G, Moratti R, MerliniG. Clinical aspects of systemic amyloid diseases. Biochim Biophys Acta. 2005;1753(1):11-22. (Pubitemid 41597427)
-
(2005)
Biochimica et Biophysica Acta - Proteins and Proteomics
, vol.1753
, Issue.1
, pp. 11-22
-
-
Obici, L.1
Perfetti, V.2
Palladini, G.3
Moratti, R.4
Merlini, G.5
-
20
-
-
0016063911
-
The genetics of Caenorhabditis elegans
-
Brenner S. The genetics of Caenorhabditis elegans. Genetics. 1974;77(1):71-94.
-
(1974)
Genetics
, vol.77
, Issue.1
, pp. 71-94
-
-
Brenner, S.1
-
21
-
-
84865040298
-
Specific recognition of biologically active amyloid-b oligomers by a new surface plasmon resonancebased immunoassay and an in vivo assay in Caenorhabditis elegans
-
Stravalaci M, Bastone A, Beeg M, et al. Specific recognition of biologically active amyloid-b oligomers by a new surface plasmon resonancebased immunoassay and an in vivo assay in Caenorhabditis elegans. J Biol Chem. 2012; 287(33):27796-27805.
-
(2012)
J Biol Chem.
, vol.287
, Issue.33
, pp. 27796-27805
-
-
Stravalaci, M.1
Bastone, A.2
Beeg, M.3
-
22
-
-
84871356165
-
C. Elegans expressing human b2-microglobulin: A novel model for studying the relationship between the molecular assembly and the toxic phenotype
-
Diomede L, Soria C, Romeo M, et al. C. elegans expressing human b2-microglobulin: a novel model for studying the relationship between the molecular assembly and the toxic phenotype. PLoS ONE. 2012;7(12):e52314.
-
(2012)
PLoS One
, vol.7
, Issue.12
-
-
Diomede, L.1
Soria, C.2
Romeo, M.3
-
23
-
-
44949142269
-
The selective detection of mitochondrial superoxide by live cell imaging
-
DOI 10.1038/nprot.2008.56, PII NPROT.2008.56
-
Robinson KM, Janes MS, Beckman JS. The selective detection of mitochondrial superoxide by live cell imaging. Nat Protoc. 2008;3(6):941-947. (Pubitemid 351818682)
-
(2008)
Nature Protocols
, vol.3
, Issue.6
, pp. 941-947
-
-
Robinson, K.M.1
Janes, M.S.2
Beckman, J.S.3
-
24
-
-
38149142769
-
Detection of 2-hydroxyethidium in cellular systems: A unique marker product of superoxide and hydroethidine
-
Zielonka J, Vasquez-Vivar J, Kalyanaraman B. Detection of 2-hydroxyethidium in cellular systems: a unique marker product of superoxide and hydroethidine. Nat Protoc. 2008;3(1):8-21.
-
(2008)
Nat Protoc.
, vol.3
, Issue.1
, pp. 8-21
-
-
Zielonka, J.1
Vasquez-Vivar, J.2
Kalyanaraman, B.3
-
25
-
-
78751642693
-
A novel approach for the purification and proteomic analysis of pathogenic immunoglobulin free light chains from serum
-
Lavatelli F, Brambilla F, Valentini V, et al. A novel approach for the purification and proteomic analysis of pathogenic immunoglobulin free light chains from serum. Biochim Biophys Acta. 2011; 1814(3):409-419.
-
(2011)
Biochim Biophys Acta.
, vol.1814
, Issue.3
, pp. 409-419
-
-
Lavatelli, F.1
Brambilla, F.2
Valentini, V.3
-
26
-
-
84879576449
-
Human amyloidogenic light chain proteins result in cardiac dysfunction, cell death, and early mortality in zebrafish
-
Mishra S, Guan J, Plovie E, et al. Human amyloidogenic light chain proteins result in cardiac dysfunction, cell death, and early mortality in zebrafish. Am J Physiol Heart Circ Physiol. 2013;305(1):H95-H103.
-
(2013)
Am J Physiol Heart Circ Physiol.
, vol.305
, Issue.1
-
-
Mishra, S.1
Guan, J.2
Plovie, E.3
-
27
-
-
84868358491
-
Overexpression of human amyloidogenic light chains causes heart failure in embryonic zebrafish: A preliminary report
-
Shin JT, Ward JE, Collins PA, et al. Overexpression of human amyloidogenic light chains causes heart failure in embryonic zebrafish: a preliminary report. Amyloid. 2012; 19(4):191-196.
-
(2012)
Amyloid
, vol.19
, Issue.4
, pp. 191-196
-
-
Shin, J.T.1
Ward, J.E.2
Collins, P.A.3
-
29
-
-
76049115685
-
Mitochondrial respiratory chain dysfunction variably increases oxidant stress in Caenorhabditis elegans
-
Dingley S, Polyak E, Lightfoot R, et al. Mitochondrial respiratory chain dysfunction variably increases oxidant stress in Caenorhabditis elegans. Mitochondrion. 2010; 10(2):125-136.
-
(2010)
Mitochondrion.
, vol.10
, Issue.2
, pp. 125-136
-
-
Dingley, S.1
Polyak, E.2
Lightfoot, R.3
-
30
-
-
77952346781
-
EGCG remodels mature alpha-synuclein and amyloidbeta fibrils and reduces cellular toxicity
-
Bieschke J, Russ J, Friedrich RP, et al. EGCG remodels mature alpha-synuclein and amyloidbeta fibrils and reduces cellular toxicity. Proc Natl Acad Sci USA. 2010;107(17):7710-7715.
-
(2010)
Proc Natl Acad Sci USA
, vol.107
, Issue.17
, pp. 7710-7715
-
-
Bieschke, J.1
Russ, J.2
Friedrich, R.P.3
-
31
-
-
84881396342
-
A new face for old antibiotics: Tetracyclines in treatment of amyloidoses
-
Stoilova T, Colombo L, Forloni G, Tagliavini F, Salmona M. A new face for old antibiotics: tetracyclines in treatment of amyloidoses. J Med Chem. 2013;56(15):5987-6006.
-
(2013)
J Med Chem.
, vol.56
, Issue.15
, pp. 5987-6006
-
-
Stoilova, T.1
Colombo, L.2
Forloni, G.3
Tagliavini, F.4
Salmona, M.5
-
32
-
-
40549144848
-
Potential anthelmintics: Polyphenols from the tea plant Camellia sinensis L. Are lethally toxic to Caenorhabditis elegans
-
Mukai D, Matsuda N, Yoshioka Y, Sato M, Yamasaki T. Potential anthelmintics: polyphenols from the tea plant Camellia sinensis L. are lethally toxic to Caenorhabditis elegans. J Nat Med. 2008; 62(2):155-159.
-
(2008)
J Nat Med.
, vol.62
, Issue.2
, pp. 155-159
-
-
Mukai, D.1
Matsuda, N.2
Yoshioka, Y.3
Sato, M.4
Yamasaki, T.5
-
33
-
-
84892163127
-
Effects of the antibiotic tetracycline on the reproduction, growth and population growth rate of the nematode Caenorhabditis elegans
-
Vangheel M, Traunspurger W, Spann N. Effects of the antibiotic tetracycline on the reproduction, growth and population growth rate of the nematode Caenorhabditis elegans. Nematology. 2014;16(1):19-29.
-
(2014)
Nematology
, vol.16
, Issue.1
, pp. 19-29
-
-
Vangheel, M.1
Traunspurger, W.2
Spann, N.3
-
34
-
-
84055223035
-
Doxycycline reduces fibril formation in a transgenic mouse model of AL amyloidosis
-
Ward JE, Ren R, Toraldo G, et al. Doxycycline reduces fibril formation in a transgenic mouse model of AL amyloidosis. Blood. 2011;118(25): 6610-6617.
-
(2011)
Blood
, vol.118
, Issue.25
, pp. 6610-6617
-
-
Ward, J.E.1
Ren, R.2
Toraldo, G.3
-
35
-
-
84878199302
-
Worms under stress: C. Elegans stress response and its relevance to complex humandisease and aging
-
Rodriguez M, Snoek LB, De Bono M, Kammenga JE. Worms under stress: C. elegans stress response and its relevance to complex humandisease and aging. Trends Genet. 2013;29(6): 367-374.
-
(2013)
Trends Genet
, vol.29
, Issue.6
, pp. 367-374
-
-
Rodriguez, M.1
Snoek, L.B.2
De Bono, M.3
Kammenga, J.E.4
-
36
-
-
77049097621
-
Behavioral phenotyping of a transgenic Caenorhabditis elegans expressing neuronal amyloid-beta
-
Dosanjh LE, Brown MK, Rao G, Link CD, Luo Y. Behavioral phenotyping of a transgenic Caenorhabditis elegans expressing neuronal amyloid-beta. J Alzheimers Dis. 2010;19(2): 681-690.
-
(2010)
J Alzheimers Dis.
, vol.19
, Issue.2
, pp. 681-690
-
-
Dosanjh, L.E.1
Brown, M.K.2
Rao, G.3
Link, C.D.4
Luo, Y.5
-
37
-
-
0028981288
-
Expression of human beta-amyloid peptide in transgenic Caenorhabditis elegans
-
Link CD. Expression of human beta-amyloid peptide in transgenic Caenorhabditis elegans. Proc Natl Acad Sci USA. 1995;92(20):9368-9372.
-
(1995)
Proc Natl Acad Sci USA
, vol.92
, Issue.20
, pp. 9368-9372
-
-
Link, C.D.1
-
38
-
-
78650685650
-
B-Amyloid peptide increases levels of iron content and oxidative stress in human cell and Caenorhabditis elegans models of Alzheimer disease
-
Wan L, Nie G, Zhang J, et al. b-Amyloid peptide increases levels of iron content and oxidative stress in human cell and Caenorhabditis elegans models of Alzheimer disease. Free Radic Biol Med. 2011;50(1):122-129.
-
(2011)
Free Radic Biol Med.
, vol.50
, Issue.1
, pp. 122-129
-
-
Wan, L.1
Nie, G.2
Zhang, J.3
-
39
-
-
84892992289
-
Novel approaches for studying amyloidogenic peptides/proteins
-
Beeg M, Diomede L, Stravalaci M, Salmona M, Gobbi M. Novel approaches for studying amyloidogenic peptides/proteins. Curr Opin Pharmacol. 2013;13(5):797-801.
-
(2013)
Curr Opin Pharmacol.
, vol.13
, Issue.5
, pp. 797-801
-
-
Beeg, M.1
Diomede, L.2
Stravalaci, M.3
Salmona, M.4
Gobbi, M.5
-
40
-
-
84871417031
-
Role of mitochondrial homeostasis and dynamics in Alzheimer's disease
-
Selfridge JE, e L, Lu J, Swerdlow RH. Role of mitochondrial homeostasis and dynamics in Alzheimer's disease. Neurobiol Dis. 2013;51: 3-12.
-
(2013)
Neurobiol Dis.
, vol.51
, pp. 3-12
-
-
Selfridge, J.E.1
Lu, J.2
Swerdlow, R.H.3
-
41
-
-
79958721260
-
Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: Implications for neuronal damage
-
Manczak M, Calkins MJ, Reddy PH. Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage. Hum Mol Genet. 2011;20(13):2495-2509.
-
(2011)
Hum Mol Genet.
, vol.20
, Issue.13
, pp. 2495-2509
-
-
Manczak, M.1
Calkins, M.J.2
Reddy, P.H.3
-
42
-
-
0038375018
-
4-iodo-4-deoxydoxorubicin and tetracyclines disrupt transthyretin amyloid fibrils in vitro producing noncytotoxic species: Screening for TTR fibril disrupters
-
DOI 10.1096/fj.02-0764com
-
Cardoso I, Merlini G, Saraiva MJ. 49-iodo-49- deoxydoxorubicin and tetracyclines disrupt transthyretin amyloid fibrils in vitro producing noncytotoxic species: screening for TTR fibril disrupters. FASEB J. 2003;17(8):803-809. (Pubitemid 36538654)
-
(2003)
FASEB Journal
, vol.17
, Issue.8
, pp. 803-809
-
-
Cardoso, I.1
Merlini, G.2
Saraiva, M.J.3
-
43
-
-
46749109619
-
The efficacy of tetracyclines in peripheral and intracerebral prion infection
-
De Luigi A, Colombo L, Diomede L, et al. The efficacy of tetracyclines in peripheral and intracerebral prion infection. PLoS One. 2008; 3(3):e1888.
-
(2008)
PLoS One
, vol.3
, Issue.3
-
-
De Luigi, A.1
Colombo, L.2
Diomede, L.3
-
44
-
-
79251631666
-
Tetracycline compounds with non-antimicrobial organ protective properties: Possible mechanisms of action
-
Griffin MO, Ceballos G, Villarreal FJ. Tetracycline compounds with non-antimicrobial organ protective properties: possible mechanisms of action. Pharmacol Res. 2011; 63(2):102-107.
-
(2011)
Pharmacol Res.
, vol.63
, Issue.2
, pp. 102-107
-
-
Griffin, M.O.1
Ceballos, G.2
Villarreal, F.J.3
-
45
-
-
78650104379
-
Tetracycline and its analogues protect Caenorhabditis elegans from b amyloid-induced toxicity by targeting oligomers
-
Diomede L, Cassata G, Fiordaliso F, et al. Tetracycline and its analogues protect Caenorhabditis elegans from b amyloid-induced toxicity by targeting oligomers. Neurobiol Dis. 2010;40(2):424-431.
-
(2010)
Neurobiol Dis.
, vol.40
, Issue.2
, pp. 424-431
-
-
Diomede, L.1
Cassata, G.2
Fiordaliso, F.3
-
46
-
-
44849087785
-
EGCG redirects amyloidogenic polypeptides into unstructured, off-pathway oligomers
-
DOI 10.1038/nsmb.1437, PII NSMB1437
-
Ehrnhoefer DE, Bieschke J, Boeddrich A, et al. EGCG redirects amyloidogenic polypeptides into unstructured, off-pathway oligomers. Nat Struct Mol Biol. 2008;15(6):558-566. (Pubitemid 351799141)
-
(2008)
Nature Structural and Molecular Biology
, vol.15
, Issue.6
, pp. 558-566
-
-
Ehrnhoefer, D.E.1
Bieschke, J.2
Boeddrich, A.3
Herbst, M.4
Masino, L.5
Lurz, R.6
Engemann, S.7
Pastore, A.8
Wanker, E.E.9
-
47
-
-
79960910835
-
Natural polyphenols inhibit different steps of the process of transthyretin (TTR) amyloid fibril formation
-
Ferreira N, Saraiva MJ, Almeida MR. Natural polyphenols inhibit different steps of the process of transthyretin (TTR) amyloid fibril formation. FEBS Lett. 2011;585(15):2424-2430.
-
(2011)
FEBS Lett.
, vol.585
, Issue.15
, pp. 2424-2430
-
-
Ferreira, N.1
Saraiva, M.J.2
Almeida, M.R.3
-
48
-
-
34548813607
-
Epigallocathechin-3-gallate in AL amyloidosis: A new therapeutic option? [2]
-
DOI 10.1182/blood-2007-05-089243
-
Hunstein W. Epigallocathechin-3-gallate in AL amyloidosis: a new therapeutic option? Blood. 2007;110(6):2216. (Pubitemid 47443945)
-
(2007)
Blood
, vol.110
, Issue.6
, pp. 2216
-
-
Hunstein, W.1
-
49
-
-
77955227051
-
Effects of the main green tea polyphenol epigallocatechin-3-gallate on cardiac involvement in patients with AL amyloidosis
-
Mereles D, Buss SJ, Hardt SE, HunsteinW, Katus HA. Effects of the main green tea polyphenol epigallocatechin-3-gallate on cardiac involvement in patients with AL amyloidosis. Clin Res Cardiol. 2010;99(8):483-490.
-
(2010)
Clin Res Cardiol.
, vol.99
, Issue.8
, pp. 483-490
-
-
Mereles, D.1
Buss, S.J.2
Hardt, S.E.3
Hunstein, W.4
Katus, H.A.5
|