Reduction of infarct size by the therapeutic protein TAT-Ndi1 in vivo

Journal of Cardiovascular Pharmacology and Therapeutics

Volumn 19, Issue 3, 2014, Pages 315-320

  Mentzer, Robert M ^a,b   Wider, Joseph ^a   Perry, Cynthia N ^c   Gottlieb, Roberta A ^d  

^a WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b SAN DIEGO STATE UNIVERSITY   (United States)

^c USA   (United States)

^d CEDARS SINAI MEDICAL CENTER   (United States)

Author keywords

acute myocardial infarction; cardioprotection; complex I; heart disease; ischemia reperfusion injury; mitochondria; myocardial protection

Indexed keywords

CARDIOVASCULAR AGENT; HYBRID PROTEIN; NICOTINAMIDE ADENINE DINUCLEOTIDE; PLACEBO; PROTECTIVE AGENT; TAT NDI1 FUSION PROTEIN; TETRAZOLIUM; TRANSACTIVATOR PROTEIN; UNCLASSIFIED DRUG; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE);

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL FREE SYSTEM; COMPARATIVE STUDY; CONTROLLED STUDY; CORONARY ARTERY OCCLUSION; EXPERIMENTAL MYOCARDIAL ISCHEMIA; FEMALE; HEART INFARCTION SIZE; HEART MUSCLE CELL; HEART PROTECTION; IN VIVO STUDY; ISOLATED HEART; NEWBORN; NONHUMAN; PRIORITY JOURNAL; RANDOMIZATION; RAT; REPERFUSION INJURY; ANIMAL; BIOSYNTHESIS; DISEASE MODEL; HEART INFARCTION; HEART MITOCHONDRION; METABOLISM; MYOCARDIAL REPERFUSION INJURY; PATHOPHYSIOLOGY; SPRAGUE DAWLEY RAT;

ANIMALS; DISEASE MODELS, ANIMAL; ELECTRON TRANSPORT COMPLEX I; FEMALE; GENE PRODUCTS, TAT; MITOCHONDRIA, HEART; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; NAD; RATS; RATS, SPRAGUE-DAWLEY;

EID: 84898941825     PISSN: 10742484     EISSN: 19404034     Source Type: Journal    
DOI: 10.1177/1074248413515750     Document Type: Article

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