Delayed, long-term administration of the caspase inhibitor Q-VD-OPH reduced brain injury induced by neonatal hypoxia-ischemia

Developmental Neuroscience

Volumn 36, Issue 1, 2014, Pages 64-72

  Han, Wei ^a,d   Sun, Yanyan ^b,d   Wang, Xiaoyang ^b,d   Zhu, Changlian ^b,d   Blomgren, Klas ^a,b,c  

^a KAROLINSKA UNIVERSITY HOSPITAL   (Sweden)

^b UNIVERSITY OF GOTHENBURG   (Sweden)

^c QUEEN SILVIA CHILDREN'S HOSPITAL   (Sweden)

^d ZHENGZHOU UNIVERSITY   (China)

Author keywords

Brain tissue loss; CCL2; CCL3; Cylinder Rearing Test; IL 10; IL 4; MCP 1; MIP 1 alpha; Open Field

Indexed keywords

CASPASE 3; CASPASE INHIBITOR; INTERLEUKIN 10; INTERLEUKIN 4; MACROPHAGE INFLAMMATORY PROTEIN 1ALPHA; MONOCYTE CHEMOTACTIC PROTEIN 1; Q VD OPH; UNCLASSIFIED DRUG; MICROTUBULE ASSOCIATED PROTEIN 2; NEUROPROTECTIVE AGENT;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN DEVELOPMENT; BRAIN INJURY; BRAIN ISCHEMIA; CONTROLLED STUDY; ENZYME ACTIVITY; HYPERACTIVITY; MALE; MOTOR PERFORMANCE; MOUSE; NEWBORN HYPOXIA; NONHUMAN; OPEN FIELD TEST; PRIORITY JOURNAL; REARING; SENSORIMOTOR FUNCTION; BRAIN TISSUE; CELL DENSITY; IMMUNOHISTOCHEMISTRY; MICROGLIA; MOTOR ACTIVITY; NEUROPROTECTION; NEWBORN; PROTEIN EXPRESSION; WEIGHT GAIN;

AMINO ACID CHLOROMETHYL KETONES; ANIMALS; BEHAVIOR, ANIMAL; BRAIN; CASPASE 3; CASPASE INHIBITORS; CELL COUNT; CHEMOKINE CCL2; CHEMOKINE CCL3; HYPOXIA-ISCHEMIA, BRAIN; MALE; MICE; MICROGLIA; MOTOR ACTIVITY; QUINOLINES; TREATMENT OUTCOME;

EID: 84897114067     PISSN: 03785866     EISSN: 14219859     Source Type: Journal    
DOI: 10.1159/000357939     Document Type: Article

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