Fetal hypoxia increases vulnerability of hypoxic-ischemic brain injury in neonatal rats: Role of glucocorticoid receptors

Neurobiology of Disease

Volumn 65, Issue , 2014, Pages 172-179

  Gonzalez Rodriguez, Pablo J ^a   Xiong, Fuxia ^a   Li, Yong ^a   Zhou, Jianjun ^a   Zhang, Lubo ^a  

^a LOMA LINDA UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Glucocorticoids; Hypoxia; Hypoxic ischemic brain injury; Neonate

Indexed keywords

GLUCOCORTICOID RECEPTOR;

ANIMAL TISSUE; ARTICLE; BRAIN DEVELOPMENT; CONTROLLED STUDY; DNA METHYLATION; DOWN REGULATION; EPIGENETICS; EXON; FETUS; FETUS HYPOXIA; GENE EXPRESSION REGULATION; GENE REPRESSION; GESTATIONAL AGE; GLUCOCORTICOID RECEPTOR GENE; HIPPOCAMPUS; HYPOXIC ISCHEMIC ENCEPHALOPATHY; INTRAUTERINE GROWTH RETARDATION; NEWBORN; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; RAT;

GLUCOCORTICOIDS; HYPOXIA; HYPOXIC-ISCHEMIC BRAIN INJURY; NEONATE;

AGE FACTORS; ANIMALS; ANIMALS, NEWBORN; BODY WEIGHT; BRAIN; BRAIN INFARCTION; DEXAMETHASONE; DISEASE MODELS, ANIMAL; DNA METHYLATION; DOSE-RESPONSE RELATIONSHIP, DRUG; FEMALE; FETAL HYPOXIA; GENE EXPRESSION REGULATION, DEVELOPMENTAL; GLUCOCORTICOIDS; HYPOXIA-ISCHEMIA, BRAIN; MALE; PREGNANCY; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, GLUCOCORTICOID; SEX FACTORS;

EID: 84896694892     PISSN: 09699961     EISSN: 1095953X     Source Type: Journal    
DOI: 10.1016/j.nbd.2014.01.020     Document Type: Article

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