H2S increases survival during sepsis: Protective effect of CHOP inhibition

Journal of Immunology

Volumn 192, Issue 4, 2014, Pages 1806-1814

  Ferlito, Marcella ^a   Wang, Qihong ^a   Fulton, William B ^a   Colombani, Paul M ^a   Marchionni, Luigi ^a   Fox Talbot, Karen ^a   Paolocci, Nazareno ^a   Steenbergen, Charles ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; CYTOKINE; DDIT3 PROTEIN, MOUSE; GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153; HYDROGEN SULFIDE; LIPOPOLYSACCHARIDE; NFE2L2 PROTEIN, MOUSE; TRANSCRIPTION FACTOR NRF2;

ANIMAL; APOPTOSIS; ARTICLE; BACTERIUM; BIOSYNTHESIS; C57BL MOUSE; CECUM; DRUG ANTAGONISM; DRUG EFFECT; ENDOPLASMIC RETICULUM STRESS; ENZYME ACTIVATION; GENETICS; IMMUNOLOGY; KNOCKOUT MOUSE; MACROPHAGE; MALE; METABOLISM; MOUSE; SEPSIS; SPLEEN; SURVIVAL;

ANIMALS; APOPTOSIS; BACTERIA; CASPASE 3; CECUM; CYTOKINES; ENDOPLASMIC RETICULUM STRESS; ENZYME ACTIVATION; HYDROGEN SULFIDE; LIPOPOLYSACCHARIDES; MACROPHAGES; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NF-E2-RELATED FACTOR 2; SEPSIS; SPLEEN; SURVIVAL; TRANSCRIPTION FACTOR CHOP;

EID: 84894265581     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1300835     Document Type: Article

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