Loss of the eukaryotic initiation factor 2α kinase general control nonderepressible 2 protects mice from pressure overload-induced congestive heart failure without affecting ventricular hypertrophy

Hypertension

Volumn 63, Issue 1, 2014, Pages 128-135

  Lu, Zhongbing ^a,b   Xu, Xin ^b   Fassett, John ^b   Kwak, Dongmin ^b   Liu, Xiaoyu ^b   Hu, Xinli ^b,d   Wang, Huan ^b   Guo, Haipeng ^b   Xu, Dachun ^b   Yan, Shuo ^b   Mcfalls, Edward O ^c   Lu, Fei ^b   Bache, Robert J ^b   Chen, Yingjie ^b  

^a UNIVERSITY OF CHINESE ACADEMY OF SCIENCES   (China)

^b UNIVERSITY OF MINNESOTA MEDICAL SCHOOL   (United States)

^c UNIVERSITY OF MINNESOTA   (United States)

^d PEKING UNIVERSITY   (China)

Author keywords

Amino acids; GCN2 protein, mouse; Heart failure; Oxidative stress

Indexed keywords

ADENOSINE TRIPHOSPHATASE (CALCIUM); PROTEIN BCL 2; PROTEIN KINASE R;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA CONSTRICTION; APOPTOSIS; ARTICLE; CONGESTIVE HEART FAILURE; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; HEART LEFT VENTRICLE HYPERTROPHY; HEART LEFT VENTRICLE OVERLOAD; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HEART STRESS; HEART VENTRICLE HYPERTROPHY; LUNG WEIGHT; MALE; MOUSE; NONHUMAN; PNEUMONIA; PRIORITY JOURNAL; PROTEIN EXPRESSION; WILD TYPE;

AMINO ACIDS; GCN2 PROTEIN, MOUSE; HEART FAILURE; OXIDATIVE STRESS;

ANIMALS; APOPTOSIS; CELLS, CULTURED; HEART FAILURE; HYPERTROPHY, LEFT VENTRICULAR; LUNG DISEASES; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCYTES, CARDIAC; OXIDATIVE STRESS; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES;

EID: 84891634186     PISSN: 0194911X     EISSN: 15244563     Source Type: Journal    
DOI: 10.1161/HYPERTENSIONAHA.113.02313     Document Type: Article

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