Oncogenic Nras has bimodal effects on stem cells that sustainably increase competitiveness

Nature

Volumn 504, Issue 7478, 2013, Pages 143-147

  Li, Qing ^a   Bohin, Natacha ^a   Wen, Tiffany ^a   Ng, Victor ^a   Magee, Jeffrey ^b   Chen, Shann Ching ^c   Shannon, Kevin ^d   Morrison, Sean J ^b  

^a UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

^c ST JUDE CHILDREN S RESEARCH HOSPITAL   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

N RAS PROTEIN; RAS PROTEIN; STAT5 PROTEIN; UNCLASSIFIED DRUG;

CYTOLOGY; MUTATION; RODENT; SIGNAL;

ANIMAL CELL; ARTICLE; CELL CYCLE PHASE; CELL DIVISION; CELL KINETICS; CELL PROLIFERATION; CELL RENEWAL; COMPETITIVE ABILITY; CONTROLLED STUDY; GENE EXPRESSION; GENETIC TRANSCRIPTION; HEMATOPOIETIC STEM CELL; INTRACELLULAR SIGNALING; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; ONCOGENE N RAS; PRIORITY JOURNAL; STEM CELL EXPANSION; WILD TYPE;

ANIMALS; BONE MARROW TRANSPLANTATION; CELL PROLIFERATION; GENE EXPRESSION PROFILING; GENES, RAS; HEMATOPOIETIC STEM CELLS; MICE; MICE, INBRED C57BL; TIME FACTORS;

MUS;

EID: 84889598519     PISSN: 00280836     EISSN: 14764687     Source Type: Journal    
DOI: 10.1038/nature12830     Document Type: Article

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