PIAS3 promotes homology-directed repair and distal non-homologous end joining

Oncology Letters

Volumn 6, Issue 4, 2013, Pages 1045-1048

  Liu, Shicui ^a,b   Fan, Zhongyi ^b   Geng, Zhengying ^b   Zhang, Hao ^c   Ye, Qinong ^c   Jiao, Shunchang ^b   Xu, Xiaojie ^c  

^a NANKAI UNIVERSITY   (China)

^b CHINESE PLA GENERAL HOSPITAL   (China)

^c BEIJING INSTITUTE OF BIOTECHNOLOGY   (China)

Author keywords

Double strand break repair; Homologous recombination; Non homologous end joining; PIAS

Indexed keywords

ATM PROTEIN; BRCA1 PROTEIN; DNA DEPENDENT PROTEIN KINASE; GREEN FLUORESCENT PROTEIN; PROTEIN INHIBITOR OF ACTIVATED STAT; PROTEIN INHIBITOR OF ACTIVATED STAT 3; PYROXYLIN; SUMO PROTEIN; UNCLASSIFIED DRUG;

ARTICLE; CELL SURVIVAL; CONTROLLED STUDY; DNA END JOINING REPAIR; DNA STRAND BREAKAGE; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; IONIZING RADIATION; PLASMID; POLYACRYLAMIDE GEL ELECTROPHORESIS; PROTEIN EXPRESSION; RADIOSENSITIVITY; TUMOR SUPPRESSOR GENE; WESTERN BLOTTING;

EID: 84881595511     PISSN: 17921074     EISSN: 17921082     Source Type: Journal    
DOI: 10.3892/ol.2013.1472     Document Type: Article

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