Interaction between chromosome 2 and 3 regulates pulse pressure in the stroke-prone spontaneously hypertensive rat

Hypertension

Volumn 62, Issue 1, 2013, Pages 33-40

  Koh Tan, H H Caline ^a   McBride, Martin W ^a   McClure, John D ^a   Beattie, Elisabeth ^a   Young, Barbara ^b   Dominiczak, Anna F ^a   Graham, Delyth ^a  

^a UNIVERSITY OF GLASGOW   (United Kingdom)

^b JOHN HUNTER HOSPITAL   (Australia)

Author keywords

bicongenic strains; congenic strains; epistasis; pulse pressure; renal pathology; stroke prone spontaneously hypertensive; Wistar Kyoto

Indexed keywords

SODIUM CHLORIDE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTERY THROMBOSIS; ARTICLE; BIOAVAILABILITY; BLEEDING; BLOOD PRESSURE VARIABILITY; BLOOD VESSEL FUNCTION; CHROMOSOME 2; CHROMOSOME 3; CONGENIC STRAIN; CONTROLLED STUDY; DIASTOLIC BLOOD PRESSURE; EPISTASIS; GENE INTERACTION; GENETIC LINKAGE; GENOTYPE ENVIRONMENT INTERACTION; HEMODYNAMICS; HISTOLOGY; HISTOPATHOLOGY; HOMOZYGOSITY; HYALINE DEGENERATION; HYPERTENSION; KIDNEY DISEASE; MALIGNANT HYPERTENSION; MICROSATELLITE MARKER; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN URINE LEVEL; PROTEINURIA; PULSE PRESSURE; QUANTITATIVE TRAIT LOCUS; RAT; STROKE PRONE SPONTANEOUSLY HYPERTENSIVE RAT; SYSTOLIC BLOOD PRESSURE; URINALYSIS; WISTAR KYOTO RAT;

BICONGENIC STRAINS; CONGENIC STRAINS; EPISTASIS; PULSE PRESSURE; RENAL PATHOLOGY; STROKE-PRONE SPONTANEOUSLY HYPERTENSIVE; WISTAR KYOTO;

ANIMALS; AORTA, THORACIC; BLOOD PRESSURE; CHROMOSOME MAPPING; CHROMOSOMES, MAMMALIAN; DISEASE MODELS, ANIMAL; GENE-ENVIRONMENT INTERACTION; HYPERTENSION; KIDNEY; MALE; RATS; RATS, INBRED SHR; RATS, INBRED WKY; STROKE; VASOCONSTRICTION; VENTRICULAR FUNCTION, LEFT;

EID: 84880754065     PISSN: 0194911X     EISSN: 15244563     Source Type: Journal    
DOI: 10.1161/HYPERTENSIONAHA.111.00814     Document Type: Article

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