Preventing p38 MAPK-mediated MafA degradation ameliorates β-Cell dysfunction under oxidative stress

Molecular Endocrinology

Volumn 27, Issue 7, 2013, Pages 1078-1090

  El Khattabi, Ilham ^a   Sharma, Arun ^a  

^a HARVARD MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GLYCOGEN SYNTHASE KINASE 3; INSULIN; MITOGEN ACTIVATED PROTEIN KINASE P38; PROTEIN KINASE D; THREONINE; TRANSCRIPTION FACTOR MAFA;

ANIMAL TISSUE; ARTICLE; ENZYME ACTIVITY; ENZYME DEGRADATION; ENZYME PHOSPHORYLATION; IMMUNOBLOTTING; INSULIN RELEASE; NONHUMAN; OXIDATIVE STRESS; PANCREAS INSUFFICIENCY; PANCREAS ISLET BETA CELL; PANCREAS ISLET BETA CELL DYSFUNCTION; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PROTEIN INTERACTION; RAT;

AMINO ACID SUBSTITUTION; ANIMALS; AUTOANTIGENS; ENZYME ACTIVATION; HUMANS; INSULIN; INSULIN-SECRETING CELLS; MAF TRANSCRIPTION FACTORS, LARGE; MICE; MODELS, BIOLOGICAL; OXIDATIVE STRESS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PHOSPHOTHREONINE; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEIN BINDING; PROTEIN KINASE C; PROTEOLYSIS; RATS; RATS, SPRAGUE-DAWLEY; SIGNAL TRANSDUCTION; UBIQUITIN;

EID: 84879466892     PISSN: 08888809     EISSN: None     Source Type: Journal    
DOI: 10.1210/me.2012-1346     Document Type: Article

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