Chronic oxidative stress as a central mechanism for glucose toxicity in pancreatic islet beta cells in diabetes
Robertson RP (2004) Chronic oxidative stress as a central mechanism for glucose toxicity in pancreatic islet beta cells in diabetes. J Biol Chem 279:42351-42354
Oxidative stressmediated, post-translational loss of MafA protein as a contributing mechanism to loss of insulin gene expression in glucotoxic beta cells
Harmon JS, Stein R, Robertson RP (2005) Oxidative stressmediated, post-translational loss of MafA protein as a contributing mechanism to loss of insulin gene expression in glucotoxic beta cells. J Biol Chem 280:11107-11113
Beta-cell-specific overexpression of glutathione peroxidase preserves intranuclear MafA and reverses diabetes in db/db mice
Harmon JS, Bogdani M, Parazzoli SD et al (2009) Beta-cell-specific overexpression of glutathione peroxidase preserves intranuclear MafA and reverses diabetes in db/db mice. Endocrinology 150:4855-4862
A switch from MafB to MafA expression accompanies differentiation to pancreatic betacells
NishimuraW, Kondo T, Salameh T et al (2006) A switch from MafB to MafA expression accompanies differentiation to pancreatic betacells. Dev Biol 293:526-539
Inconsistent formation and nonfunction of insulin-positive cells from pancreatic endoderm derived from human embryonic stem cells in athymic nude rats
Matveyenko AV, Georgia S, Bhushan A, Butler PC (2010) Inconsistent formation and nonfunction of insulin-positive cells from pancreatic endoderm derived from human embryonic stem cells in athymic nude rats. Am J Physiol Endocrinol Metab 299:E713-E720
Islet-enriched gene expression and glucose-induced insulin secretion in human and mouse islets
Dai C, Brissova M, Hang Yet al (2012) Islet-enriched gene expression and glucose-induced insulin secretion in human and mouse islets. Diabetologia 55:707-718