Cellular mechanisms of tissue fibrosis. 2. Contributory pathways leading to myocardial fibrosis: Moving beyond collagen expression

American Journal of Physiology - Cell Physiology

Volumn 304, Issue 5, 2013, Pages

  Goldsmith, Edie C ^a   Bradshaw, Amy D ^c,d   Spinale, Francis G ^a,b  

^a UNIVERSITY OF SOUTH CAROLINA SCHOOL OF MEDICINE   (United States)

^b WJB Dorn VA Medical Center   (United States)

^c MEDICAL UNIVERSITY OF SOUTH CAROLINA   (United States)

^d RALPH H JOHNSON VA MEDICAL CENTER   (United States)

Author keywords

Collagen; Extracellular matrix; Fibrosis; Myocardium

Indexed keywords

COLLAGEN; FIBRILLAR COLLAGEN; MATRILYSIN; MATRIX METALLOPROTEINASE 14; OSTEONECTIN; OSTEOPONTIN; PROCOLLAGEN C PROTEINASE; STROMELYSIN; TENASCIN; THROMBOSPONDIN 2; TRANSCRIPTION FACTOR RUNX2;

ARTICLE; CANCER GROWTH; CELL TRANSDIFFERENTIATION; COLLAGEN DEGRADATION; COLLAGEN SYNTHESIS; CONNECTIVE TISSUE CELL; EPITHELIAL MESENCHYMAL TRANSITION; EXTRACELLULAR MATRIX; GOLGI COMPLEX; HEART DEVELOPMENT; HEART INFARCTION; HEART MUSCLE; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HUMAN; MESENCHYMAL STEM CELL; MONOCYTE; MYOFIBROBLAST; NONHUMAN; PERICYTE; PHENOTYPE; POSTNATAL GROWTH; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN PROCESSING; ROUGH ENDOPLASMIC RETICULUM;

ANIMALS; EXTRACELLULAR MATRIX; FIBRILLAR COLLAGENS; FIBROBLASTS; FIBROSIS; HUMANS; MATRIX METALLOPROTEINASES; MYOCARDIUM; VENTRICULAR REMODELING;

EID: 84874626027     PISSN: 03636143     EISSN: 15221563     Source Type: Journal    
DOI: 10.1152/ajpcell.00347.2012     Document Type: Article

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