Prenatal stress exposure increases the excitation of dopamine neurons in the ventral tegmental area and alters their reponses to psychostimulants

Neuropsychopharmacology

Volumn 38, Issue 2, 2013, Pages 293-301

  Hausknecht, Kathryn ^a   Haj Dahmane, Samir ^a   Shen, Roh Yu ^a  

^a RESEARCH INSTITUTE ON ADDICTIONS   (United States)

Author keywords

addiction risk; amphetamine; cocaine; depolarization block; self administration

Indexed keywords

AMPHETAMINE; COCAINE;

ADDICTION; ADULTHOOD; ANIMAL CELL; ARTICLE; BRAIN ELECTROPHYSIOLOGY; CELL POPULATION; CONTROLLED STUDY; DISEASE PREDISPOSITION; DOPAMINERGIC NERVE CELL; DRUG SELF ADMINISTRATION; EXPOSURE; FEMALE; IN VIVO STUDY; MALE; MARE; MOLECULAR PATHOLOGY; NERVE CELL MEMBRANE STEADY POTENTIAL; NERVE CELL STIMULATION; NONHUMAN; PREGNANCY; PREGNANCY OUTCOME; PRENATAL STRESS; PRIORITY JOURNAL; RAT; RISK ASSESSMENT; VENTRAL TEGMENTUM;

ANIMALS; ANIMALS, NEWBORN; CENTRAL NERVOUS SYSTEM STIMULANTS; DOPAMINERGIC NEURONS; FEMALE; MALE; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; RATS; RATS, SPRAGUE-DAWLEY; SELF ADMINISTRATION; STRESS, PSYCHOLOGICAL; VENTRAL TEGMENTAL AREA;

EID: 84871421690     PISSN: 0893133X     EISSN: 1740634X     Source Type: Journal    
DOI: 10.1038/npp.2012.168     Document Type: Article

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