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Volumn 297, Issue 2, 2001, Pages 746-752
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Amphetamine normalizes the electrical activity of dopamine neurons in the ventral tegmental area following prenatal ethanol exposure
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Author keywords
[No Author keywords available]
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Indexed keywords
ALCOHOL;
AMPHETAMINE;
CHLORAL HYDRATE;
ANIMAL EXPERIMENT;
ARTICLE;
ATTENTION DEFICIT DISORDER;
CELL COUNT;
CELL MEMBRANE DEPOLARIZATION;
CONTROLLED STUDY;
DOPAMINERGIC NERVE CELL;
DOPAMINERGIC TRANSMISSION;
DOSE RESPONSE;
DRUG EFFECT;
DRUG MECHANISM;
FEMALE;
FETAL ALCOHOL SYNDROME;
MALE;
NERVE CELL STIMULATION;
NEWBORN;
NONHUMAN;
PRENATAL EXPOSURE;
PRIORITY JOURNAL;
RAT;
VENTRAL TEGMENTUM;
ACTION POTENTIALS;
AMPHETAMINE;
ANIMALS;
CELL COUNT;
CENTRAL NERVOUS SYSTEM DEPRESSANTS;
DOPAMINE;
DOPAMINE UPTAKE INHIBITORS;
ELECTROPHYSIOLOGY;
ETHANOL;
FEMALE;
NEURONS;
PREGNANCY;
PRENATAL EXPOSURE DELAYED EFFECTS;
RATS;
RATS, SPRAGUE-DAWLEY;
VENTRAL TEGMENTAL AREA;
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EID: 0035035093
PISSN: 00223565
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (40)
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References (33)
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