AFG3L2 supports mitochondrial protein synthesis and Purkinje cell survival

Journal of Clinical Investigation

Volumn 122, Issue 11, 2012, Pages 4048-4058

  Almajan, Eva R ^a   Richter, Ricarda ^a   Paeger, Lars ^a   Martinelli, Paola ^a   Barth, Esther ^a   Decker, Thorsten ^a   Larsson, Nils Göran ^a,b   Kloppenburg, Peter ^a   Langer, Thomas ^a,b   Rugarli, Elena I ^a  

^a UNIVERSITY OF COLOGNE   (Germany)

^b MAX PLANCK INSTITUTE FOR BIOLOGY OF AGEING   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

MITOCHONDRIAL PROTEIN;

AFG3L2 GENE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL STRUCTURE; CELL SURVIVAL; CONTROLLED STUDY; DENDRITE; DISORDERS OF MITOCHONDRIAL FUNCTIONS; EMBRYO; FEMALE; FRAGMENTATION REACTION; GENE; GENE DELETION; GENE EXPRESSION; GENE MUTATION; MALE; MITOCHONDRION; MOUSE; NERVE CELL; NERVE DEGENERATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN SYNTHESIS; PURKINJE CELL; RIBOSOME;

ANIMALS; ATP-DEPENDENT PROTEASES; CELL SURVIVAL; HUMANS; INTELLECTUAL DISABILITY; MEMBRANE PROTEINS; METALLOENDOPEPTIDASES; MICE; MICE, KNOCKOUT; MITOCHONDRIA; MITOCHONDRIAL PROTEINS; MUSCLE SPASTICITY; MUTATION; NERVE TISSUE PROTEINS; OPTIC ATROPHY; PROTEIN BIOSYNTHESIS; PURKINJE CELLS; SPINOCEREBELLAR ATAXIAS; SPINOCEREBELLAR DEGENERATIONS;

EID: 84868628376     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI64604     Document Type: Article

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