Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus

Human Molecular Genetics

Volumn 21, Issue 21, 2012, Pages 4703-4717

  Dombret, Carlos ^a   Nguyen, Tuan ^a,g   Schakman, Olivier ^b   Michaud, Jacques L ^c   Hardin Pouzet, Hélène ^d   Bertrand, Mathieu J M ^e,f   De Backer, Olivier ^a  

^a University of Namur   (Belgium)

^b UNIVERSITÉ CATHOLIQUE DE LOUVAIN   (Belgium)

^c UNIVERSITY OF MONTREAL   (Canada)

^d SORBONNE UNIVERSITÉ   (France)

^e DEPARTMENT OF PLANT SYSTEMS BIOLOGY   (Belgium)

^f GHENT UNIVERSITY   (Belgium)

^g Department of Biochemistry and Molecular Biology   (Viet Nam)

Author keywords

[No Author keywords available]

Indexed keywords

OXYTOCIN;

AMNESIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANXIETY DISORDER; ARTICLE; AUTISM; CONTROLLED STUDY; FEMALE; FOOD INTAKE; GENE; GENE LOSS; HYPERPHAGIA; HYPOTHALAMUS; MAGED1 GENE; MALE; MOUSE; NONHUMAN; OBESITY; OXYTOCIN RELEASE; PRIORITY JOURNAL; SEXUAL DEVIATION; SOCIOPATHY;

ANIMALS; ANXIETY; AUTISTIC DISORDER; HUMANS; HYPOTHALAMUS; INTERPERSONAL RELATIONS; MICE; NEOPLASM PROTEINS; OBESITY; OXYTOCIN; PRADER-WILLI SYNDROME; SEXUAL BEHAVIOR, ANIMAL;

EID: 84867824683     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/dds310     Document Type: Article

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