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Volumn 37, Issue 10, 2012, Pages 2065-2075

Role of oxidative stress and Ca2+ signaling on molecular pathways of neuropathic pain in diabetes: Focus on TRP channels

Author keywords

Calcium ion; Diabetes; Mitochondria; Oxidative stress; Pain; Sensory neurons; Transient receptor potential channels

Indexed keywords

ADVANCED GLYCATION END PRODUCT; CALCIUM CHANNEL P TYPE; CALCIUM CHANNEL Q TYPE; CALCIUM ION; CYCLIC AMP DEPENDENT PROTEIN KINASE; CYCLIC GMP; CYCLIC GMP DEPENDENT PROTEIN KINASE; MIBEFRADIL; N (4 TERT BUTYLPHENYL) 4 (3 CHLORO 2 PYRIDINYL) 1 PIPERAZINECARBOXAMIDE; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; NIFEDIPINE; NITRIC OXIDE; OMEGA CONOTOXIN; POLYOL; PROTEIN KINASE C; PROTEIN KINASE C BETA; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); RUBOXISTAURIN; SARCOPLASMIC RETICULUM CALCIUM TRANSPORTING ADENOSINE TRIPHOSPHATASE; TAURINE; TRANSIENT RECEPTOR POTENTIAL CHANNEL; TRANSIENT RECEPTOR POTENTIAL CHANNEL C; TRANSIENT RECEPTOR POTENTIAL CHANNEL M; TRANSIENT RECEPTOR POTENTIAL CHANNEL M2; TRANSIENT RECEPTOR POTENTIAL CHANNEL M6; TRANSIENT RECEPTOR POTENTIAL CHANNEL M7; UBIQUINOL CYTOCHROME C REDUCTASE; UNINDEXED DRUG; VANILLOID RECEPTOR; VANILLOID RECEPTOR 1;

EID: 84866740036     PISSN: 03643190     EISSN: 15736903     Source Type: Journal    
DOI: 10.1007/s11064-012-0850-x     Document Type: Review
Times cited : (133)

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