Ascorbate protects against vascular leakage in cecal ligation and puncture-induced septic peritonitis

American Journal of Physiology - Regulatory Integrative and Comparative Physiology

Volumn 302, Issue 4, 2012, Pages

  Zhou, Gang ^a   Kamenos, George ^a   Pendem, Suresh ^a   Wilson, John X ^a   Wu, Feng ^a  

^a UNIVERSITY AT BUFFALO   (United States)

Author keywords

Endothelial cells; Occludin; Protein phosphatase 2A

Indexed keywords

3 NITROTYROSINE; ASCORBIC ACID; DEXTRAN; ENDOTHELIAL NITRIC OXIDE SYNTHASE; EVANS BLUE; INDUCIBLE NITRIC OXIDE SYNTHASE; OCCLUDIN; PHOSPHOPROTEIN PHOSPHATASE 2A; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; SERINE; SUPEROXIDE; THREONINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD VESSEL PERMEABILITY; CECAL LIGATION AND PUNCTURE; CONTROLLED STUDY; ENDOTHELIUM CELL; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME PHOSPHORYLATION; IMMUNOPRECIPITATION; INTESTINE SURGERY; MALE; MOUSE; NONHUMAN; PERITONITIS; PRIORITY JOURNAL; SEPSIS; SKELETAL MUSCLE;

ANIMALS; ANTIOXIDANTS; ASCORBIC ACID; CAPILLARY PERMEABILITY; CECUM; MALE; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MUSCLE, SKELETAL; NADPH OXIDASE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE II; NITRIC OXIDE SYNTHASE TYPE III; PERITONITIS; PROTEIN PHOSPHATASE 2; SEPSIS; SUPEROXIDES; TYROSINE;

EID: 84863229258     PISSN: 03636119     EISSN: 15221490     Source Type: Journal    
DOI: 10.1152/ajpregu.00153.2011     Document Type: Article

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