Overexpression of human wild-type amyloid-β protein precursor decreases the iron content and increases the oxidative stress of neuroblastoma SH-SY5Y cells

Journal of Alzheimer's Disease

Volumn 30, Issue 3, 2012, Pages 523-530

  Wan, Li ^a,b   Nie, Guangjun ^c   Zhang, Jie ^d   Zhao, Baolu ^a,b  

^a INSTITUTE OF BIOPHYSICS   (China)

^b UNIVERSITY OF CHINESE ACADEMY OF SCIENCES   (China)

^c NATIONAL CENTER FOR NANOSCIENCE AND TECHNOLOGY   (China)

^d XIAMEN UNIVERSITY   (China)

Author keywords

Alzheimer's disease; amyloid protein precursor; iron deficiency; oxidative stress

Indexed keywords

AMYLOID PRECURSOR PROTEIN; CALCIUM; CATALASE; IRON; REACTIVE OXYGEN METABOLITE;

APOPTOSIS; ARTICLE; CELL VIABILITY; CONTROLLED STUDY; ENZYME ACTIVITY; GENE OVEREXPRESSION; HUMAN; HUMAN CELL; MITOCHONDRIAL MEMBRANE POTENTIAL; NEUROBLASTOMA CELL; OXIDATIVE STRESS; PATHOGENESIS; PRIORITY JOURNAL; STABLE TRANSFECTION;

EID: 84862845774     PISSN: 13872877     EISSN: 18758908     Source Type: Journal    
DOI: 10.3233/JAD-2012-111169     Document Type: Article

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