Jab1 interacts with brain-specific kinase 2 (BRSK2) and promotes its degradation in the ubiquitin-proteasome pathway

Biochemical and Biophysical Research Communications

Volumn 422, Issue 4, 2012, Pages 647-652

  Zhou, Jun ^a   Wan, Bo ^a   Li, Ruwei ^a   Gu, Xiuting ^a   Zhong, Zhaomin ^a   Wang, Yingli ^a   Yu, Long ^a  

^a FUDAN UNIVERSITY   (China)

Author keywords

BRSK2; Cell cycle arrest; Degradation; Jab1; Ubiquitination

Indexed keywords

BRAIN SPECIFIC KINASE 2; C JUN ACTIVATION DOMAIN BINDING PROTEIN 1; CELL PROTEIN; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE KINASE; PROTEASOME; PROTEIN C JUN; UBIQUITIN; UNCLASSIFIED DRUG;

ARTICLE; CELL CYCLE ARREST; CELL CYCLE G2 PHASE; CELL CYCLE M PHASE; CELL CYCLE PROGRESSION; CONTROLLED STUDY; HUMAN; HUMAN CELL; HUMAN TISSUE; IMMUNOPRECIPITATION; IN VITRO STUDY; IN VIVO STUDY; MAMMAL CELL; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN INTERACTION; PROTEIN ISOLATION; PROTEIN LOCALIZATION; SIGNAL TRANSDUCTION; UBIQUITINATION;

ANIMALS; G2 PHASE CELL CYCLE CHECKPOINTS; HEK293 CELLS; HELA CELLS; HEP G2 CELLS; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; M PHASE CELL CYCLE CHECKPOINTS; MICE; PEPTIDE HYDROLASES; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEIN-SERINE-THREONINE KINASES; PROTEOLYSIS; TWO-HYBRID SYSTEM TECHNIQUES; UBIQUITIN;

MAMMALIA; MUS;

EID: 84862298972     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2012.05.045     Document Type: Article

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