A novel pathway of NADPH oxidase/vascular peroxidase 1 in mediating oxidative injury following ischemia-reperfusion

Basic Research in Cardiology

Volumn 107, Issue 3, 2012, Pages

  Zhang, Yi Shuai ^a   He, Lan ^a   Liu, Bin ^a   Li, Nian Sheng ^a   Luo, Xiu Ju ^a   Hu, Chang Ping ^a   Ma, Qi Lin ^a   Zhang, Guo Gang ^a   Li, Yuan Jian ^a   Peng, Jun ^a  

^a CENTRAL SOUTH UNIVERSITY   (China)

Author keywords

Heart; Hypoxia reoxygenation; Ischemia reperfusion; NADPH oxidase (NOX); Vascular peroxidase 1 (VPO1)

Indexed keywords

CASPASE 3; CREATINE KINASE; HYDROGEN PEROXIDE; MITOGEN ACTIVATED PROTEIN KINASE P38; PEROXIDASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE 2; STRESS ACTIVATED PROTEIN KINASE; UNCLASSIFIED DRUG; VASCULAR PEROXIDASE 1; CASP3 PROTEIN, RAT; CYBB PROTEIN, RAT; ENZYME INHIBITOR; HEMOPROTEIN; HYPOCHLOROUS ACID; MEMBRANE PROTEIN; NOX4 PROTEIN, RAT; VASCULAR PEROXIDASE, RAT;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CREATINE KINASE BLOOD LEVEL; ENZYME ACTIVITY; ENZYME INHIBITION; ENZYME LOCALIZATION; HEART INFARCTION SIZE; HEART MUSCLE ISCHEMIA; IN VITRO STUDY; IN VIVO STUDY; MALE; MOLECULAR PATHOLOGY; NONHUMAN; OXIDATIVE STRESS; RAT; REPERFUSION INJURY; SIGNAL TRANSDUCTION; ANIMAL; BLOOD; CELL LINE; DISEASE MODEL; DRUG ANTAGONISM; DRUG EFFECT; ENZYMOLOGY; GENETIC TRANSFECTION; GENETICS; HEART INFARCTION; HEART MUSCLE; METABOLISM; PATHOLOGY; RNA INTERFERENCE; SPRAGUE DAWLEY RAT;

ANIMALS; APOPTOSIS; CASPASE 3; CELL LINE; CREATINE KINASE; DISEASE MODELS, ANIMAL; ENZYME INHIBITORS; HEMEPROTEINS; HYDROGEN PEROXIDE; HYPOCHLOROUS ACID; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MALE; MEMBRANE GLYCOPROTEINS; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; NADPH OXIDASE; OXIDATIVE STRESS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PEROXIDASES; RATS; RATS, SPRAGUE-DAWLEY; RNA INTERFERENCE; SIGNAL TRANSDUCTION; TRANSFECTION;

EID: 84859037766     PISSN: 03008428     EISSN: 14351803     Source Type: Journal    
DOI: 10.1007/s00395-012-0266-4     Document Type: Article

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