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Volumn 120, Issue 3, 2012, Pages 332-339

Do interactions between gut ecology and environmental chemicals contribute to obesity and diabetes?

Author keywords

ADME; Biotransformation enzymes; Diabetes mellitus; Diabetogenic; Environmental chemicals; Gut ecology; Metabolic syndrome; Microbes; Microbiota; Obesity; Obesogen; Obesogenic; Persistent organic pollutants; POPs

Indexed keywords

1,1 DICHLORO 2,2 BIS(4 CHLOROPHENYL)ETHYLENE; 4,4' ISOPROPYLIDENEDIPHENOL; ALACHLOR; CHLORDANE; CHLORPYRIFOS; CYANAZINE; DIETHYLSTILBESTROL; DIMPYLATE; DIOXIN; ENVIRONMENTAL CHEMICAL; ESTROGEN RECEPTOR; GENISTEIN; GLUCAGON LIKE PEPTIDE 1; GLUCOCORTICOID RECEPTOR; HEPTACHLOR; HERBICIDE; HEXACHLOROBENZENE; INSECTICIDE; METRIFONATE; NONYLPHENOL; PEPTIDE YY; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA; POLYBROMINATED DIPHENYL ETHER; POLYCHLORINATED BIPHENYL; RETINOID X RECEPTOR; RETINOID X RECEPTOR ALPHA; RETINOID X RECEPTOR BETA; RETINOID X RECEPTOR GAMMA; TRIBUTYLTIN;

EID: 84857726895     PISSN: 00916765     EISSN: 15529924     Source Type: Journal    
DOI: 10.1289/ehp.1104204     Document Type: Review
Times cited : (142)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.