Similar cation channels mediate protection from cerebellar exitotoxicity by exercise and inheritance

Journal of Cellular and Molecular Medicine

Volumn 16, Issue 3, 2012, Pages 555-568

  Ben Ari, Shani ^a   Ofek, Keren ^a   Barbash, Shahar ^a   Meiri, Hanoch ^a   Kovalev, Eugenia ^b   Greenberg, David Samuel ^a   Soreq, Hermona ^a   Shoham, Shai ^b  

^a HEBREW UNIVERSITY OF JERUSALEM   (Israel)

^b HERZOG HOSPITAL   (Israel)

Author keywords

Exercise; Genetic background; Glutamate; Kir6.1; Potassium channels; Stroke

Indexed keywords

ADENOSINE TRIPHOSPHATE SENSITIVE POTASSIUM CHANNEL; AMINO ACID RECEPTOR STIMULATING AGENT; CYCLIC NUCLEOTIDE GATED CHANNEL; GLIBENCLAMIDE; GLUTAMATE RECEPTOR; GLUTAMIC ACID DERIVATIVE; HYPERPOLARIZATION ACTIVATED CATION CHANNEL; HYPERPOLARIZATION-ACTIVATED CATION CHANNEL; KAINIC ACID; KCNAB2 PROTEIN, MOUSE; POTASSIUM CHANNEL; SHAKER POTASSIUM CHANNEL; UK ATP 1 POTASSIUM CHANNEL; UK-ATP-1 POTASSIUM CHANNEL;

ANIMAL; ANIMAL EXPERIMENT; ARTICLE; C57BL MOUSE; CEREBELLUM; DRUG ANTAGONISM; DRUG EFFECT; GENE EXPRESSION PROFILING; GENETICS; HUMAN; INTRACEREBROVENTRICULAR DRUG ADMINISTRATION; MALE; METABOLISM; MOUSE; PATHOPHYSIOLOGY; STROKE; UPREGULATION;

ANIMALS; CEREBELLUM; CYCLIC NUCLEOTIDE-GATED CATION CHANNELS; EXCITATORY AMINO ACID AGONISTS; GENE EXPRESSION PROFILING; GLUTAMATES; GLYBURIDE; HUMANS; INJECTIONS, INTRAVENTRICULAR; KAINIC ACID; KATP CHANNELS; MALE; MICE; MICE, INBRED C57BL; PHYSICAL CONDITIONING, ANIMAL; POTASSIUM CHANNELS; RECEPTORS, GLUTAMATE; SHAKER SUPERFAMILY OF POTASSIUM CHANNELS; STROKE; UP-REGULATION;

MUS;

EID: 84857540834     PISSN: 15821838     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1582-4934.2011.01331.x     Document Type: Article

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