Neuron-glia interactions underlie ALS-like axonal cytoskeletal pathology

Neurobiology of Aging

Volumn 32, Issue 3, 2011, Pages 459-469

  King, A E ^a   Dickson, T C ^a   Blizzard, C A ^a   Woodhouse, A ^a   Foster, S S ^a   Chung, R S ^a   Vickers, J C ^a  

^a UNIVERSITY OF TASMANIA   (Australia)

Author keywords

ALS; Axon; Glia; Motor neuron; Neurofilament

Indexed keywords

COPPER ZINC SUPEROXIDE DISMUTASE; NEUROFILAMENT PROTEIN;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL CELL; ARTICLE; CELL INTERACTION; CONTROLLED STUDY; GLIA CELL; MITOCHONDRION; MOUSE; NERVE CELL; NERVE CELL CULTURE; NERVE CELL NECROSIS; NERVE FIBER DEGENERATION; NERVE FIBER TRANSPORT; NEUROPATHOLOGY; NONHUMAN; PRIORITY JOURNAL; SPINAL CORD MOTONEURON; UBIQUITINATION;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; ANIMALS, NEWBORN; AXONS; BACTERIAL PROTEINS; CELL DEATH; CELLS, CULTURED; CYTOCHROMES C; CYTOSKELETON; DISEASE MODELS, ANIMAL; EDEMA; GLIAL FIBRILLARY ACIDIC PROTEIN; HUMANS; LUMINESCENT PROTEINS; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MOTOR NEURONS; NEUROFILAMENT PROTEINS; NEUROGLIA; SPINAL CORD; SUPEROXIDE DISMUTASE; SYNAPTOPHYSIN; TIME FACTORS; TRANSFECTION;

EID: 79952898546     PISSN: 01974580     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2009.04.004     Document Type: Article

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