DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis

Cancer Science

Volumn 102, Issue 4, 2011, Pages 677-682

  Oka, Sugako ^a   Nakabeppu, Yusaku ^a  

^a KYUSHU UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

8 HYDROXYGUANINE; APC PROTEIN; CELL NUCLEUS DNA; DNA GLYCOSYLASE MUTY; DNA GLYCOSYLTRANSFERASE; K RAS PROTEIN; MITOCHONDRIAL DNA;

ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; CANCER INHIBITION; CARCINOGENESIS; CELL PROLIFERATION; CONTROLLED STUDY; GASTROINTESTINAL POLYPOSIS; GENE MUTATION; GENE SWITCHING; HUMAN; INTESTINE EPITHELIUM; LOSS OF FUNCTION MUTATION; MISSENSE MUTATION; MOUSE; MUTAGENESIS; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN FUNCTION; REVIEW; SINGLE STRANDED DNA BREAK; STEM CELL;

APOPTOSIS; CELL TRANSFORMATION, NEOPLASTIC; DNA GLYCOSYLASES; GERM-LINE MUTATION; HUMANS; NEOPLASMS; OXIDATIVE STRESS;

EID: 79952765891     PISSN: 13479032     EISSN: 13497006     Source Type: Journal    
DOI: 10.1111/j.1349-7006.2011.01869.x     Document Type: Review

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