Evidence that apoptotic signalling in hypertrophic cardiomyocytes is determined by mitochondrial pathways involving protein kinase Cδ

Clinical and Experimental Pharmacology and Physiology

Volumn 37, Issue 12, 2010, Pages 1120-1128

  Xie, Man Jiang ^a   Chang, Hui ^a   Wang, Yun Ying ^a   Zhang, Lin ^a   Song, Zhen ^a   Guo, Wan Gang ^a   Wang, Tao ^a   Che, Hong Lei ^a   Yu, Zhi Bin ^a  

^a FOURTH MILITARY MEDICAL UNIVERSITY   (China)

Author keywords

Apoptosis; Cardiac myocytes; Hypertrophy; Mitochondria; Protein kinase C

Indexed keywords

1 (4 DIMETHYLAMINO 3 METHYLBENZYL) 5 DIPHENYLACETYL 4,5,6,7 TETRAHYDRO 1H IMIDAZO[4,5 C]PYRIDINE 6 CARBOXYLIC ACID; ANGIOTENSIN II; CASPASE 3; CASPASE 9; CYCLO(DEXTRO TRYPTOPHYL DEXTRO ASPARTYLPROLYL DEXTRO VALYLLEUCYL); ENDOTHELIN 1; LOSARTAN; PROCASPASE 9; PROTEIN KINASE C DELTA; ROTTLERIN; SOMATOMEDIN C; THYROID HORMONE;

ANIMAL CELL; APOPTOSIS; ARTICLE; HEART MUSCLE CELL; HYPERTROPHY; NEWBORN; NONHUMAN; PROTEIN EXPRESSION; RAT;

ACETOPHENONES; ANGIOTENSIN II; ANIMALS; APOPTOSIS; BENZOPYRANS; CARDIOMEGALY; CASPASE 3; CASPASE 9; CELLS, CULTURED; ENDOTHELIN-1; IMIDAZOLES; INSULIN-LIKE GROWTH FACTOR I; MITOCHONDRIA; MYOCYTES, CARDIAC; PEPTIDES, CYCLIC; PROTEIN KINASE C-DELTA; PROTEIN TRANSPORT; PYRIDINES; RATS; RATS, SPRAGUE-DAWLEY; RECEPTOR, ANGIOTENSIN, TYPE 1; RECEPTOR, ANGIOTENSIN, TYPE 2; RECEPTORS, ENDOTHELIN; SIGNAL TRANSDUCTION; TRIIODOTHYRONINE;

EID: 78649732074     PISSN: 03051870     EISSN: 14401681     Source Type: Journal    
DOI: 10.1111/j.1440-1681.2010.05447.x     Document Type: Article

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