Pin1 acts as a modulator of cell proliferation through alteration in NF-κB but not β-catenin/TCF4 signalling in a subset of endometrial carcinoma cells

Journal of Pathology

Volumn 222, Issue 4, 2010, Pages 410-420

  Saegusa, Makoto ^a   Hashimura, Miki ^a   Kuwata, Takeshi ^b  

^a KITASATO UNIVERSITY SCHOOL OF MEDICINE   (Japan)

^b NATIONAL CANCER CENTER HOSPITAL EAST   (Japan)

Author keywords

Carcinoma; Catenin; Endometrium; PIN1; TCF4

Indexed keywords

BETA CATENIN; CYCLIN D1; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; PEPTIDYLPROLYL ISOMERASE PIN1; SYNAPTOTAGMIN I; TRANSCRIPTION FACTOR E2F1;

ADENOCARCINOMA; ARTICLE; CELL PROLIFERATION; CELL TRANSDIFFERENTIATION; DOWN REGULATION; ENDOMETRIUM CARCINOMA; GENE OVEREXPRESSION; GENETIC TRANSFECTION; HUMAN; HUMAN CELL; HUMAN TISSUE; MENSTRUAL CYCLE; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN STABILITY; SIGNAL TRANSDUCTION; TRANSACTIVATION;

BASIC HELIX-LOOP-HELIX LEUCINE ZIPPER TRANSCRIPTION FACTORS; BETA CATENIN; CELL CYCLE PROTEINS; CELL PROLIFERATION; ENDOMETRIAL NEOPLASMS; ENDOMETRIUM; FEMALE; HUMANS; NEOPLASM PROTEINS; NF-KAPPA B; PEPTIDYLPROLYL ISOMERASE; SIGNAL TRANSDUCTION; TRANSCRIPTION FACTORS; TUMOR CELLS, CULTURED;

EID: 78349276864     PISSN: 00223417     EISSN: 10969896     Source Type: Journal    
DOI: 10.1002/path.2773     Document Type: Article

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