MG132, a proteasome inhibitor decreased the growth of Calu-6 lung cancer cells via apoptosis and GSH depletion

Toxicology in Vitro

Volumn 24, Issue 4, 2010, Pages 1237-1242

  Han, Yong Hwan ^a   Park, Woo Hyun ^a  

^a Chonbuk National University   (South Korea)

Author keywords

Apoptosis; Calu 6 lung cancer; Cell cycle; GSH; MG132; ROS

Indexed keywords

BENZYLOXYCARBONYLLEUCYLLEUCYLLEUCINAL; BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE; GLUTATHIONE; REACTIVE OXYGEN METABOLITE;

APOPTOSIS; ARTICLE; CANCER CELL; CANCER INHIBITION; CELL COUNT; CELL CYCLE ARREST; CELL CYCLE S PHASE; CELL DEATH; CONTROLLED STUDY; DRUG MECHANISM; FLOW CYTOMETRY; HUMAN; HUMAN CELL; LUNG CANCER; MITOCHONDRIAL MEMBRANE POTENTIAL; PROTEIN DEPLETION;

ANTINEOPLASTIC AGENTS; APOPTOSIS; CELL CYCLE; CELL LINE, TUMOR; CELL PROLIFERATION; GLUTATHIONE; HUMANS; LEUPEPTINS; LUNG NEOPLASMS; MEMBRANE POTENTIALS; PROTEASOME ENDOPEPTIDASE COMPLEX; REACTIVE OXYGEN SPECIES;

EID: 77952956912     PISSN: 08872333     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.tiv.2010.02.005     Document Type: Article

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