-
1
-
-
0037426726
-
The pathophysiology and treatment of sepsis
-
Hotchkiss, R. S., and I. E. Karl. 2003. The pathophysiology and treatment of sepsis. N. Engl. J. Med. 348: 138-150.
-
(2003)
N. Engl. J. Med.
, vol.348
, pp. 138-150
-
-
Hotchkiss, R.S.1
Karl, I.E.2
-
2
-
-
26244446442
-
Acute lung injury and the acute respiratory distress syndrome: Four decades of inquiry into pathogenesis and rational management
-
Matthay, M. A., and G. A. Zimmerman. 2005. Acute lung injury and the acute respiratory distress syndrome: four decades of inquiry into pathogenesis and rational management. Am. J. Respir. Cell Mol. Biol. 33: 319-327.
-
(2005)
Am. J. Respir. Cell Mol. Biol.
, vol.33
, pp. 319-327
-
-
Matthay, M.A.1
Zimmerman, G.A.2
-
3
-
-
0028331867
-
An anion binding site in human aldose reductase: Mechanistic implications for the binding of citrate, cacodylate, and glucose 6-phosphate
-
Harrison, D. H., K. M. Bohren, D. Ringe, G. A. Petsko, and K. H. Gabbay. 1994. An anion binding site in human aldose reductase: mechanistic implications for the binding of citrate, cacodylate, and glucose 6-phosphate. Biochemistry 33: 2011-2020.
-
(1994)
Biochemistry
, vol.33
, pp. 2011-2020
-
-
Harrison, D.H.1
Bohren, K.M.2
Ringe, D.3
Petsko, G.A.4
Gabbay, K.H.5
-
4
-
-
0028810466
-
Human aldose reductase: PK of tyrosine 48 reveals the preferred ionization state for catalysis and inhibition
-
Grimshaw, C. E., K. M. Bohren, C. J. Lai, and K. H. Gabbay. 1995. Human aldose reductase: pK of tyrosine 48 reveals the preferred ionization state for catalysis and inhibition. Biochemistry 34: 14374-14384.
-
(1995)
Biochemistry
, vol.34
, pp. 14374-14384
-
-
Grimshaw, C.E.1
Bohren, K.M.2
Lai, C.J.3
Gabbay, K.H.4
-
5
-
-
38149099908
-
Current concepts in targeted therapies for the pathophysiology of diabetic microvascular complications
-
Cumbie, B. C., and K. L. Hermayer. 2007. Current concepts in targeted therapies for the pathophysiology of diabetic microvascular complications. Vasc. Health Risk Manag. 3: 823-832.
-
(2007)
Vasc. Health Risk Manag.
, vol.3
, pp. 823-832
-
-
Cumbie, B.C.1
Hermayer, K.L.2
-
6
-
-
61949182565
-
Aldose reductase mediates myocardial ischemia-reperfusion injury in part by opening mitochondrial permeability transition pore
-
Ananthakrishnan, R., M. Kaneko, Y. C. Hwang, T. Gomes, C. Caspersen, and R. Ramasamy. 2009. Aldose reductase mediates myocardial ischemia-reperfusion injury in part by opening mitochondrial permeability transition pore. Am J. Physiol. 296: H333-H341.
-
(2009)
Am J. Physiol.
, vol.296
-
-
Ananthakrishnan, R.1
Kaneko, M.2
Hwang, Y.C.3
Gomes, T.4
Caspersen, C.5
Ramasamy, R.6
-
7
-
-
33747069158
-
Aldose reductase-deficient mice are protected from delayed motor nerve conduction velocity, increased c-Jun NH2-terminal kinase activation, depletion of reduced glutathione, increased superoxide accumulation, and DNA damage
-
Ho, E. C., K. S. Lam, Y. S. Chen, J. C. Yip, M. Arvindakshan, S. Yamagishi, S. Yagihashi, P. J. Oates, C. A. Ellery, S. S. Chung, and S. K. Chung. 2006. Aldose reductase-deficient mice are protected from delayed motor nerve conduction velocity, increased c-Jun NH2-terminal kinase activation, depletion of reduced glutathione, increased superoxide accumulation, and DNA damage. Diabetes 55: 1946-1953.
-
(2006)
Diabetes
, vol.55
, pp. 1946-1953
-
-
Ho, E.C.1
Lam, K.S.2
Chen, Y.S.3
Yip, J.C.4
Arvindakshan, M.5
Yamagishi, S.6
Yagihashi, S.7
Oates, P.J.8
Ellery, C.A.9
Chung, S.S.10
Chung, S.K.11
-
8
-
-
23744461861
-
Aldose reductase and AGE-RAGE pathways: Key players in myocardial ischemic injury
-
DOI 10.1196/annals.1333.081
-
Kaneko, M., L. Bucciarelli, Y. C. Hwang, L. Lee, S. F. Yan, A. M. Schmidt, and R. Ramasamy. 2005. Aldose reductase and AGE-RAGE pathways: key players in myocardial ischemic injury. Ann. NY Acad. Sci. 1043: 702-709. (Pubitemid 41123963)
-
(2005)
Annals of the New York Academy of Sciences
, vol.1043
, pp. 702-709
-
-
Kaneko, M.1
Bucciarelli, L.2
Hwang, Y.C.3
Lee, L.4
Yan, S.F.5
Schmidt, A.M.6
Ramasamy, R.7
-
9
-
-
24644491335
-
Human aldose reductase expression accelerates diabetic atherosclerosis in transgenic mice
-
Vikramadithyan, R. K., Y. Hu, H. L. Noh, C. P. Liang, K. Hallam, A. R. Tall, R. Ramasamy, and I. J. Goldberg. 2005. Human aldose reductase expression accelerates diabetic atherosclerosis in transgenic mice. J. Clin. Invest. 115: 2434-2443.
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 2434-2443
-
-
Vikramadithyan, R.K.1
Hu, Y.2
Noh, H.L.3
Liang, C.P.4
Hallam, K.5
Tall, A.R.6
Ramasamy, R.7
Goldberg, I.J.8
-
10
-
-
3442890207
-
Central role for aldose reductase pathway in myocardial ischemic injury
-
Hwang, Y. C., M. Kaneko, S. Bakr, H. Liao, Y. Lu, E. R. Lewis, S. Yan, S. Ii, M. Itakura, L. Rui, et al. 2004. Central role for aldose reductase pathway in myocardial ischemic injury. FASEB J. 18: 1192-1199.
-
(2004)
FASEB J.
, vol.18
, pp. 1192-1199
-
-
Hwang, Y.C.1
Kaneko, M.2
Bakr, S.3
Liao, H.4
Lu, Y.5
Lewis, E.R.6
Yan, S.7
Ii, S.8
Itakura, M.9
Rui, L.10
-
11
-
-
33750561283
-
Endotoxin-induced cardiomyopathy and systemic inflammation in mice is prevented by aldose reductase inhibition
-
Ramana, K. V., M. S. Willis, M. D. White, J. W. Horton, J. M. DiMaio, D. Srivastava, A. Bhatnagar, and S. K. Srivastava. 2006. Endotoxin-induced cardiomyopathy and systemic inflammation in mice is prevented by aldose reductase inhibition. Circulation 114: 1838-1846.
-
(2006)
Circulation
, vol.114
, pp. 1838-1846
-
-
Ramana, K.V.1
Willis, M.S.2
White, M.D.3
Horton, J.W.4
DiMaio, J.M.5
Srivastava, D.6
Bhatnagar, A.7
Srivastava, S.K.8
-
12
-
-
40449087466
-
Activation of Rho kinase by TNF-α is required for JNK activation in human pulmonary microvascular endothelial cells
-
Mong, P. Y., C. Petrulio, H. L. Kaufman, and Q. Wang. 2008. Activation of Rho kinase by TNF-α is required for JNK activation in human pulmonary microvascular endothelial cells. J. Immunol. 180: 550-558.
-
(2008)
J. Immunol.
, vol.180
, pp. 550-558
-
-
Mong, P.Y.1
Petrulio, C.2
Kaufman, H.L.3
Wang, Q.4
-
13
-
-
14844327760
-
Reactive oxygen species promote TNFα-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases
-
DOI 10.1016/j.cell.2004.12.041
-
Kamata, H., S. Honda, S. Maeda, L. Chang, H. Hirata, and M. Karin. 2005. Reactive oxygen species promote TNFα-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases. Cell 120: 649-661. (Pubitemid 40343077)
-
(2005)
Cell
, vol.120
, Issue.5
, pp. 649-661
-
-
Kamata, H.1
Honda, S.-I.2
Maeda, S.3
Chang, L.4
Hirata, H.5
Karin, M.6
-
14
-
-
8344260568
-
Ferritin heavy chain upregulation by NF-κB inhibits TNFα-induced apoptosis by suppressing reactive oxygen species
-
Pham, C. G., C. Bubici, F. Zazzeroni, S. Papa, J. Jones, K. Alvarez, S. Jayawardena, E. De Smaele, R. Cong, C. Beaumont, et al. 2004. Ferritin heavy chain upregulation by NF-κB inhibits TNFα-induced apoptosis by suppressing reactive oxygen species. Cell 119: 529-542.
-
(2004)
Cell
, vol.119
, pp. 529-542
-
-
Pham, C.G.1
Bubici, C.2
Zazzeroni, F.3
Papa, S.4
Jones, J.5
Alvarez, K.6
Jayawardena, S.7
De Smaele, E.8
Cong, R.9
Beaumont, C.10
-
15
-
-
0042525938
-
NF-κB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell death
-
Sakon, S., X. Xue, M. Takekawa, T. Sasazuki, T. Okazaki, Y. Kojima, J. H. Piao, H. Yagita, K. Okumura, T. Doi, and H. Nakano. 2003. NF-κB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell death. EMBO J. 22: 3898-3909.
-
(2003)
EMBO J.
, vol.22
, pp. 3898-3909
-
-
Sakon, S.1
Xue, X.2
Takekawa, M.3
Sasazuki, T.4
Okazaki, T.5
Kojima, Y.6
Piao, J.H.7
Yagita, H.8
Okumura, K.9
Doi, T.10
Nakano, H.11
-
17
-
-
0020520054
-
Evaluation of factors affecting mortality rate after sepsis in a murine cecal ligation and puncture model
-
Baker, C. I., H. O. Gaines, and A. E. Baue. 1983. Evaluation of factors affecting mortality rate after sepsis in a murine cecal ligation and puncture model. Surgery 94: 331-335.
-
(1983)
Surgery
, vol.94
, pp. 331-335
-
-
Baker, C.I.1
Gaines, H.O.2
Baue, A.E.3
-
18
-
-
61449149438
-
Activation of Rho kinase isoforms in lung endothelial cells during inflammation
-
Mong, P. Y., and Q. Wang. 2009. Activation of Rho kinase isoforms in lung endothelial cells during inflammation. J. Immunol. 182: 2385-2394.
-
(2009)
J. Immunol.
, vol.182
, pp. 2385-2394
-
-
Mong, P.Y.1
Wang, Q.2
-
19
-
-
0029059577
-
C1q triggers neutrophil superoxide production by a unique CD18-dependent mechanism
-
Goodman, E. B., D. C. Anderson, and A. J. Tenner. 1995. C1q triggers neutrophil superoxide production by a unique CD18-dependent mechanism. J. Leukocyte Biol. 58: 168-176.
-
(1995)
J. Leukocyte Biol.
, vol.58
, pp. 168-176
-
-
Goodman, E.B.1
Anderson, D.C.2
Tenner, A.J.3
-
20
-
-
6344252954
-
Cecal ligation and puncture with total parenteral nutrition: A clinically relevant model of the metabolic, hormonal, and inflammatory dysfunction associated with critical illness
-
Heuer, J. G., D. L. Bailey, G. R. Sharma, T. Zhang, C. Ding, A. Ford, E. J. Stephens, K. C. Holmes, R. L. Grubbs, K. A. Fynboe, et al. 2004. Cecal ligation and puncture with total parenteral nutrition: a clinically relevant model of the metabolic, hormonal, and inflammatory dysfunction associated with critical illness. J. Surg. Res. 121: 178-186.
-
(2004)
J. Surg. Res.
, vol.121
, pp. 178-186
-
-
Heuer, J.G.1
Bailey, D.L.2
Sharma, G.R.3
Zhang, T.4
Ding, C.5
Ford, A.6
Stephens, E.J.7
Holmes, K.C.8
Grubbs, R.L.9
Fynboe, K.A.10
-
22
-
-
22344437262
-
Inflammation and the reciprocal production of granulocytes and lymphocytes in bone marrow
-
DOI 10.1084/jem.20041419
-
Ueda, Y., M. Kondo, and G. Kelsoe. 2005. Inflammation and the reciprocal production of granulocytes and lymphocytes in bone marrow. J. Exp. Med. 201: 1771-1780. (Pubitemid 41002896)
-
(2005)
Journal of Experimental Medicine
, vol.201
, Issue.11
, pp. 1771-1780
-
-
Ueda, Y.1
Kondo, M.2
Kelsoe, G.3
-
23
-
-
0035318540
-
Mechanisms of leukocyte sequestration in inflamed lungs
-
Doerschuk, C. M. 2001. Mechanisms of leukocyte sequestration in inflamed lungs. Microcirculation 8: 71-88.
-
(2001)
Microcirculation
, vol.8
, pp. 71-88
-
-
Doerschuk, C.M.1
-
24
-
-
20044374702
-
Attenuation of endotoxin-induced acute lung injury by the Rho-associated kinase inhibitor, Y-27632
-
Tasaka, S., H. Koh, W. Yamada, M. Shimizu, Y. Ogawa, N. Hasegawa, K. Yamaguchi, Y. Ishii, S. E. Richer, C. M. Doerschuk, and A. Ishizaka. 2005. Attenuation of endotoxin-induced acute lung injury by the Rho-associated kinase inhibitor, Y-27632. Am. J. Respir. Cell Mol. Biol. 32: 504-510.
-
(2005)
Am. J. Respir. Cell Mol. Biol.
, vol.32
, pp. 504-510
-
-
Tasaka, S.1
Koh, H.2
Yamada, W.3
Shimizu, M.4
Ogawa, Y.5
Hasegawa, N.6
Yamaguchi, K.7
Ishii, Y.8
Richer, S.E.9
Doerschuk, C.M.10
Ishizaka, A.11
-
25
-
-
33646763902
-
Rho kinases in cardiovascular physiology and pathophysiology
-
DOI 10.1161/01.RES.0000201960.04223.3c, PII 0000301220060217000008
-
Loirand, G., P. Guerin, and P. Pacaud. 2006. Rho kinases in cardiovascular physiology and pathophysiology. Circ. Res. 98: 322-334. (Pubitemid 43755328)
-
(2006)
Circulation Research
, vol.98
, Issue.3
, pp. 322-334
-
-
Loirand, G.1
Guerin, P.2
Pacaud, P.3
-
27
-
-
0035793913
-
Activation of human neutrophil by cytokine-activated endothelial cells
-
Takahashi, T., F. Hato, T. Yamane, H. Fukumasu, K. Suzuki, S. Ogita, Y. Nishizawa, and S. Kitagawa. 2001. Activation of human neutrophil by cytokine-activated endothelial cells. Circ. Res. 88: 422-429. (Pubitemid 32200116)
-
(2001)
Circulation Research
, vol.88
, Issue.4
, pp. 422-429
-
-
Takahashi, T.1
Hato, F.2
Yamane, T.3
Fukumasu, H.4
Suzuki, K.5
Ogita, S.6
Nishizawa, Y.7
Kitagawa, S.8
-
28
-
-
44049084268
-
Role of nitric oxide in regulating aldose reductase activation in the ischemic heart
-
Kaiserova, K., X. L. Tang, S. Srivastava, and A. Bhatnagar. 2008. Role of nitric oxide in regulating aldose reductase activation in the ischemic heart. J. Biol. Chem. 283: 9101-9112.
-
(2008)
J. Biol. Chem.
, vol.283
, pp. 9101-9112
-
-
Kaiserova, K.1
Tang, X.L.2
Srivastava, S.3
Bhatnagar, A.4
-
29
-
-
0037218519
-
Protective effects of IL-6 blockade in sepsis are linked to reduced C5a receptor expression
-
Riedemann, N. C., T. A. Neff, R. F. Guo, K. D. Bernacki, I. J. Laudes, J. V. Sarma, J. D. Lambris, and P. A. Ward. 2003. Protective effects of IL-6 blockade in sepsis are linked to reduced C5a receptor expression. J. Immunol. 170: 503-507.
-
(2003)
J. Immunol.
, vol.170
, pp. 503-507
-
-
Riedemann, N.C.1
Neff, T.A.2
Guo, R.F.3
Bernacki, K.D.4
Laudes, I.J.5
Sarma, J.V.6
Lambris, J.D.7
Ward, P.A.8
-
30
-
-
0025871289
-
High circulating levels of interleukin-6 in patients with septic shock: Evolution during sepsis, prognostic value, and interplay with other cytokines: The Swiss-Dutch J5 Immunoglobulin Study Group
-
Calandra, T., J. Gerain, D. Heumann, J. D. Baumgartner, and M. P. Glauser. 1991. High circulating levels of interleukin-6 in patients with septic shock: evolution during sepsis, prognostic value, and interplay with other cytokines: the Swiss-Dutch J5 Immunoglobulin Study Group. Am. J. Med. 91: 23-33.
-
(1991)
Am. J. Med.
, vol.91
, pp. 23-33
-
-
Calandra, T.1
Gerain, J.2
Heumann, D.3
Baumgartner, J.D.4
Glauser, M.P.5
-
31
-
-
17644401778
-
Role of interleukin-6 in mortality from and physiologic response to sepsis
-
DOI 10.1128/IAI.73.5.2751-2757.2005
-
Remick, D. G., G. Bolgos, S. Copeland, and J. Siddiqui. 2005. Role of interleukin-6 in mortality from and physiologic response to sepsis. Infect. Immun. 73: 2751-2757. (Pubitemid 40570341)
-
(2005)
Infection and Immunity
, vol.73
, Issue.5
, pp. 2751-2757
-
-
Remick, D.G.1
Bolgos, G.2
Copeland, S.3
Siddiqui, J.4
-
32
-
-
0031227757
-
Polymorphonuclear leukocytes released from the bone marrow preferentially sequester in-lung micro vessels
-
van Eeden, S. F., Y. Kitagawa, M. E. Klut, E. Lawrence, and J. C. Hogg. 1997. Polymorphonuclear leukocytes released from the bone marrow preferentially sequester in lung microvessels. Microcirculation 4: 369-380. (Pubitemid 127674019)
-
(1997)
Microcirculation
, vol.4
, Issue.3
, pp. 369-380
-
-
Van Eeden, S.F.1
Kitagawa, Y.2
Klut, M.E.3
Lawrence, E.4
Hogg, J.C.5
-
33
-
-
0000797922
-
Human endothelial cells in culture produce platelet-activating factor (1-alkyl-2-acetyl-sn-glycero-3-phosphocholine) when stimulated with thrombin
-
Prescott, S. M., G. A. Zimmerman, and T. M. Mclntyre. 1984. Human endothelial cells in culture produce platelet-activating factor (1-alkyl-2-acetyl-sn-glycero-3-phosphocholine) when stimulated with thrombin. Proc. Natl. Acad. Sci. USA 81: 3534-3538.
-
(1984)
Proc. Natl. Acad. Sci. USA
, vol.81
, pp. 3534-3538
-
-
Prescott, S.M.1
Zimmerman, G.A.2
Mclntyre, T.M.3
-
34
-
-
0023724630
-
Synthesis and release of platelet-activating factor by human vascular endothelial cells treated with tumor necrosis factor or interleukin 1a
-
Bussolino, F., G. Camussi, and C. Baglioni. 1988. Synthesis and release of platelet-activating factor by human vascular endothelial cells treated with tumor necrosis factor or interleukin 1a. J. Biol. Chem. 263: 11856-11861.
-
(1988)
J. Biol. Chem.
, vol.263
, pp. 11856-11861
-
-
Bussolino, F.1
Camussi, G.2
Baglioni, C.3
-
35
-
-
33746153397
-
Suppression of vascular permeability and inflammation by targeting of the transcription factor c-Jun
-
DOI 10.1038/nbt1225, PII NBT1225
-
Fahmy, R. G., A. Waldman, G. Zhang, A. Mitchell, N. Tedla, H. Cai, C. R. Geczy, C. N. Chesterman, M. Perry, and L. M. Khachigian. 2006. Suppression of vascular permeability and inflammation by targeting of the transcription factor c-Jun. Nat. Biotechnol. 24: 856-863. (Pubitemid 44086629)
-
(2006)
Nature Biotechnology
, vol.24
, Issue.7
, pp. 856-863
-
-
Fahmy, R.G.1
Waldman, A.2
Zhang, G.3
Mitchell, A.4
Tedla, N.5
Cai, H.6
Geczy, C.R.7
Chesterman, C.N.8
Perry, M.9
Khachigian, L.M.10
-
36
-
-
18144362092
-
Inhibition of c-Jun N-terminal kinase limits lipopolysaccharide-induced pulmonary neutrophil influx
-
Arndt, P. G., S. K. Young, J. G. Lieber, M. B. Fessler, J. A. Nick, and G. S. Worthen. 2005. Inhibition of c-Jun N-terminal kinase limits lipopolysaccharide-induced pulmonary neutrophil influx. Am. J. Respir. Crit. Care Med. 171: 978-986.
-
(2005)
Am. J. Respir. Crit. Care Med.
, vol.171
, pp. 978-986
-
-
Arndt, P.G.1
Young, S.K.2
Lieber, J.G.3
Fessler, M.B.4
Nick, J.A.5
Worthen, G.S.6
-
37
-
-
0037562523
-
High glucose-induced tyrosine nitration in endothelial cells: Role of eNOS uncoupling and aldose reductase activation
-
El-Remessy, A. B., G. Abou-Mohamed, R. W. Caldwell, and R. B. Caldwell. 2003. High glucose-induced tyrosine nitration in endothelial cells: role of eNOS uncoupling and aldose reductase activation. Invest. Ophthalmol. Vis. Sci. 44: 3135-3143.
-
(2003)
Invest. Ophthalmol. Vis. Sci.
, vol.44
, pp. 3135-3143
-
-
El-Remessy, A.B.1
Abou-Mohamed, G.2
Caldwell, R.W.3
Caldwell, R.B.4
-
39
-
-
48449094505
-
Polyol pathway mediates iron-induced oxidative injury in ischemic-reperfused rat heart
-
Tang, W. H., S. Wu, T. M. Wong, S. K. Chung, and S. S. Chung. 2008. Polyol pathway mediates iron-induced oxidative injury in ischemic-reperfused rat heart. Free Radical Biol. Med. 45: 602-610.
-
(2008)
Free Radical Biol. Med.
, vol.45
, pp. 602-610
-
-
Tang, W.H.1
Wu, S.2
Wong, T.M.3
Chung, S.K.4
Chung, S.S.5
-
40
-
-
51649127081
-
The phagocytes: Neutrophils and monocytes
-
Dale, D. C., L. Boxer, and W. C. Liles. 2008. The phagocytes: neutrophils and monocytes. Blood 112: 935-945.
-
(2008)
Blood
, vol.112
, pp. 935-945
-
-
Dale, D.C.1
Boxer, L.2
Liles, W.C.3
-
41
-
-
33749382320
-
Bone marrow-derived cells require a functional glucose 6-phosphate transporter for normal myeloid functions
-
Kim, S. Y., A. D. Nguyen, J. L. Gao, P. M. Murphy, B. C. Mansfield, and J. Y. Chou. 2006. Bone marrow-derived cells require a functional glucose 6-phosphate transporter for normal myeloid functions. J. Biol. Chem. 281: 28794-28801.
-
(2006)
J. Biol. Chem.
, vol.281
, pp. 28794-28801
-
-
Kim, S.Y.1
Nguyen, A.D.2
Gao, J.L.3
Murphy, P.M.4
Mansfield, B.C.5
Chou, J.Y.6
-
42
-
-
47049108414
-
Neutrophil stress and apoptosis underlie myeloid dysfunction in glycogen storage disease type Ib
-
Kim, S. Y., H. S. Jun, P. A. Mead, B. C. Mansfield, and J. Y. Chou. 2008. Neutrophil stress and apoptosis underlie myeloid dysfunction in glycogen storage disease type Ib. Blood 111: 5704-5711.
-
(2008)
Blood
, vol.111
, pp. 5704-5711
-
-
Kim, S.Y.1
Jun, H.S.2
Mead, P.A.3
Mansfield, B.C.4
Chou, J.Y.5
-
43
-
-
33847420515
-
Impaired neutrophil activity and increased susceptibility to bacterial infection in mice lacking glucose-6-phosphatase-β
-
Cheung, Y. Y., S. Y. Kim, W. H. Yiu, C. J. Pan, H. S. Jun, R. A. Ruef, E. J. Lee, H. Westphal, B. C. Mansfield, and J. Y. Chou. 2007. Impaired neutrophil activity and increased susceptibility to bacterial infection in mice lacking glucose-6-phosphatase-β. J. Clin. Invest. 117: 784-893.
-
(2007)
J. Clin. Invest.
, vol.117
, pp. 784-893
-
-
Cheung, Y.Y.1
Kim, S.Y.2
Yiu, W.H.3
Pan, C.J.4
Jun, H.S.5
Ruef, R.A.6
Lee, E.J.7
Westphal, H.8
Mansfield, B.C.9
Chou, J.Y.10
-
44
-
-
34047203065
-
Elevated glucose concentrations promote receptor-independent activation of adherent human neutrophils: An experimental and computational approach
-
DOI 10.1529/biophysj.106.086769
-
Kummer, U., J. Zobeley, J. C. Brasen, R. Fahmy, A. L. Kindzelskii, A. R. Petty, A. J. Clark, and H. R. Petty. 2007. Elevated glucose concentrations promote receptor-independent activation of adherent human neutrophils: an experimental and computational approach. Biophys. J. 92: 2597-2607. (Pubitemid 46536439)
-
(2007)
Biophysical Journal
, vol.92
, Issue.7
, pp. 2597-2607
-
-
Kummer, U.1
Zobeley, J.2
Brasen, J.C.3
Fahmy, R.4
Kindzelskii, A.L.5
Petty, A.R.6
Clark, A.J.7
Petty, H.R.8
-
45
-
-
0037216933
-
A model of the oscillatory metabolism of activated neutrophils
-
Olsen, L. F., U. Kummer, A. L. Kindzelskii, and H. R. Petty. 2003. A model of the oscillatory metabolism of activated neutrophils. Biophys. J. 84: 69-81.
-
(2003)
Biophys. J.
, vol.84
, pp. 69-81
-
-
Olsen, L.F.1
Kummer, U.2
Kindzelskii, A.L.3
Petty, H.R.4
|