Sensitization to alloxan-induced diabetes and pancreatic cell apoptosis in acatalasemic mice

Biochimica et Biophysica Acta - Molecular Basis of Disease

Volumn 1802, Issue 2, 2010, Pages 240-246

  Kikumoto, Yoko ^a   Sugiyama, Hitoshi ^a,b   Inoue, Tatsuyuki ^a   Morinaga, Hiroshi ^a   Takiue, Keiichi ^a   Kitagawa, Masashi ^a   Fukuoka, Naomi ^a   Saeki, Mizuho ^a   Maeshima, Yohei ^a   Wang, Da Hong ^c   Ogino, Keiki ^c   Masuoka, Noriyoshi ^d   Makino, Hirofumi ^a  

^a Department of Medicine and Clinical Science ^*  (Japan)

^b Center for Chronic Kidney Disease and Peritoneal Dialysis   (Japan)

^c OKAYAMA UNIVERSITY   (Japan)

^d OKAYAMA UNIVERSITY OF SCIENCE   (Japan)

Author keywords

Acatalasemia; Angiotensin II; Apoptosis; Catalase; Diabetes mellitus; Oxidative stress; Receptor antagonist

Indexed keywords

ALLOXAN; CATALASE; GLUCOSE; GLUTATHIONE PEROXIDASE; SUPEROXIDE DISMUTASE; TELMISARTAN;

ALLOXAN DIABETES MELLITUS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CATALASE DEFICIENCY; CELL DEATH; CONTROLLED STUDY; DIABETES MELLITUS; ENZYME ACTIVITY; HOMOZYGOSITY; HYPERGLYCEMIA; INCIDENCE; MOUSE; MOUSE MUTANT; NONHUMAN; OXIDATIVE STRESS; PANCREAS CELL; PANCREAS ISLET BETA CELL; PANCREAS ISLET DISEASE; PRIORITY JOURNAL; RISK FACTOR; UPREGULATION; WILD TYPE;

ACATALASIA; ALLOXAN; ANGIOTENSIN II TYPE 1 RECEPTOR BLOCKERS; ANIMALS; APOPTOSIS; BENZIMIDAZOLES; BENZOATES; BLOOD GLUCOSE; BODY WEIGHT; CATALASE; CELL DEATH; DIABETES MELLITUS, EXPERIMENTAL; GLUTATHIONE PEROXIDASE; HOMOZYGOTE; HYPERGLYCEMIA; INSULIN-SECRETING CELLS; MALE; MICE; MICE, INBRED C3H; MICE, KNOCKOUT; MICE, MUTANT STRAINS; SUPEROXIDE DISMUTASE;

MUS;

EID: 74149091666     PISSN: 09254439     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.bbadis.2009.10.009     Document Type: Article

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