Lack of periostin leads to suppression of Notch1 signaling and calcific aortic valve disease

Physiological Genomics

Volumn 39, Issue 3, 2009, Pages 160-168

  Tkatchenko, Tatiana V ^a   Moreno Rodriguez, Ricardo A ^b   Conway, Simon J ^c   Molkentin, Jeffery D ^d   Markwald, Roger R ^b   Tkatchenko, Andrei V ^a  

^a WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b MEDICAL UNIVERSITY OF SOUTH CAROLINA   (United States)

^c INDIANA UNIVERSITY SCHOOL OF MEDICINE   (United States)

^d CINCINNATI CHILDREN'S HOSPITAL MEDICAL CENTER   (United States)

Author keywords

Development; Endocardial cushions; Outflow tract

Indexed keywords

CALCIUM; NOTCH1 RECEPTOR; OSTEOCALCIN; OSTEOPONTIN; PLEIOTROPHIN; TRANSCRIPTION FACTOR RUNX2;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA VALVE DISEASE; ARTICLE; BONE DEVELOPMENT; CALCIFICATION; CELL DIFFERENTIATION; CELL FATE; CELL LINEAGE; CONTROLLED STUDY; CORONARY ARTERY BLOOD FLOW; DELTA LIKE 1 HOMOLOG; EMBRYO; EMBRYO DEVELOPMENT; ENDOCARDIAL CUSHION; GENE; GENE EXPRESSION REGULATION; GENE INDUCTION; GENE OVEREXPRESSION; GENE REPRESSION; HEART DEVELOPMENT; HEART OUTPUT; MESENCHYME; MESENCHYME CELL; METASTASIS; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; OSTEOBLAST; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN INDUCTION; SEQUENCE HOMOLOGY; SIGNAL TRANSDUCTION; UPREGULATION;

ANIMALS; AORTIC VALVE; CALCINOSIS; CARRIER PROTEINS; CELL ADHESION MOLECULES; CORE BINDING FACTOR ALPHA 1 SUBUNIT; CYTOKINES; ECHOCARDIOGRAPHY; EMBRYO, MAMMALIAN; GENE EXPRESSION PROFILING; GENE EXPRESSION REGULATION, DEVELOPMENTAL; HEART VALVE DISEASES; IN SITU HYBRIDIZATION, FLUORESCENCE; INTERCELLULAR SIGNALING PEPTIDES AND PROTEINS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCARDIUM; OSTEOPONTIN; RECEPTOR, NOTCH1; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; SIGNAL TRANSDUCTION; TIME FACTORS;

MUS;

EID: 72949089065     PISSN: 10948341     EISSN: 15312267     Source Type: Journal    
DOI: 10.1152/physiolgenomics.00078.2009     Document Type: Article

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