Foxp1 regulates cardiac outflow tract, endocardial cushion morphogenesis and myocyte proliferation and maturation

Development

Volumn 131, Issue 18, 2004, Pages 4477-4487

  Wang, Bin ^a   Weidenfeld, Joel ^b   Lu, Min Min ^b   Maika, Shanna ^a   Kuziel, William A ^a   Morrisey, Edward E ^b   Tucker, Philip W ^a  

^a UNIVERSITY OF TEXAS AT AUSTIN   (United States)

^b UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

Endocardial cushion; Foxp1; Mouse; Myocyte proliferation; Outflow tract

Indexed keywords

MUTANT PROTEIN; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR FOXP1; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL MATURATION; CELL PROLIFERATION; CONGENITAL HEART DISEASE; CONTROLLED STUDY; DEVELOPMENTAL STAGE; EMBRYO; EMBRYO CELL; EMBRYO DEATH; ENDOCARDIAL CUSHION DEFECT; ENDOCARDIUM; GENE INACTIVATION; GENE TARGETING; HEART DEVELOPMENT; HEART MUSCLE; HEART MUSCLE CELL; HEART OUTFLOW TRACT OBSTRUCTION; HEART VENTRICLE; HEART VENTRICLE REMODELING; MESENCHYME; MORPHOGENESIS; MOUSE; NONHUMAN; NULL ALLELE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; REGULATORY MECHANISM; STEM CELL;

ANIMALS; CELL DIFFERENTIATION; CELL DIVISION; CYCLIN-DEPENDENT KINASES; DOWN-REGULATION; EMBRYO; EMBRYO LOSS; ENDOCARDIAL CUSHION DEFECTS; ENDOCARDIUM; ENZYME INHIBITORS; FORKHEAD TRANSCRIPTION FACTORS; GENE EXPRESSION REGULATION, DEVELOPMENTAL; HEART; HEART DISEASES; HIGH MOBILITY GROUP PROTEINS; IN SITU HYBRIDIZATION; MICE; MICE, KNOCKOUT; MORPHOGENESIS; MUSCLE CELLS; MYOCARDIUM; REPRESSOR PROTEINS; RNA, MESSENGER; TRANS-ACTIVATORS;

ANIMALIA; CANIDAE;

EID: 6944232153     PISSN: 09501991     EISSN: None     Source Type: Journal    
DOI: 10.1242/dev.01287     Document Type: Article

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