Relative importance of proteinase-activated receptor-1 versus matrix metalloproteinases in intracerebral hemorrhage-mediated neurotoxicity in mice

Stroke

Volumn 40, Issue 6, 2009, Pages 2199-2204

  Xue, Mengzhou ^a   Hollenberg, Morley D ^b   Demchuk, Andrew ^a   Yong, V Wee ^a,b  

^a UNIVERSITY OF CALGARY   (Canada)

^b UNIVERSITY OF CALGARY   (Canada)

Author keywords

Intracerebral hemorrhage; MMPs; Neurotoxicity; PAR1; Thrombin

Indexed keywords

GELATINASE A; GELATINASE B; HIRUDIN; ILOMASTAT; PROTEINASE ACTIVATED RECEPTOR 1; THROMBIN; ANTITHROMBIN; DIPEPTIDE; GELATIN; HIRUDIN DERIVATIVE; MATRIX METALLOPROTEINASE; PROTEINASE INHIBITOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD; BRAIN DAMAGE; BRAIN HEMORRHAGE; BRAIN INJURY; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME ACTIVITY; FEMALE; MALE; MOUSE; NERVE CELL NECROSIS; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; ANIMAL; BLOOD AUTOTRANSFUSION; BRAIN; C57BL MOUSE; CELL DEATH; CHEMISTRY; DRUG ANTAGONISM; DRUG EFFECT; GENETICS; METABOLISM; MOUSE MUTANT; PATHOLOGY; TOXICITY AND INTOXICATION;

ANIMALS; ANTITHROMBINS; BLOOD TRANSFUSION, AUTOLOGOUS; BRAIN; CELL DEATH; CEREBRAL HEMORRHAGE; DIPEPTIDES; FEMALE; GELATIN; HIRUDINS; MALE; MATRIX METALLOPROTEINASES; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NEUROTOXICITY SYNDROMES; PROTEASE INHIBITORS; RECEPTOR, PAR-1; THROMBIN;

EID: 66849131594     PISSN: 00392499     EISSN: None     Source Type: Journal    
DOI: 10.1161/STROKEAHA.108.540393     Document Type: Article

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