The deficit in long-term potentiation induced by chronic administration of amyloid-β is attenuated by treatment of rats with a novel phospholipid-based drug formulation, VP025

Experimental Gerontology

Volumn 44, Issue 4, 2009, Pages 300-304

  Miller, Anne Marie ^a   Piazza, Alessia ^a   Martin, Darren S D ^a,b   Walsh, Michelle ^a,b   Mandel, Arkady ^c   Bolton, Anthony E ^d   Lynch, Marina A ^a  

^a TRINITY COLLEGE DUBLIN   (Ireland)

^b Vasogen Ireland Ltd   (Ireland)

^c Vasogen Inc   (Canada)

^d Vasogen Ireland Ltd   (Ireland)

Author keywords

Amyloid ; Caspase 3; Hippocampus; Long term potentiation (LTP); Phospholipid particles

Indexed keywords

AMYLOID BETA PROTEIN; CASPASE 3; GLYCEROL DERIVATIVE; PHOSPHOLIPID; UNCLASSIFIED DRUG; VP 025;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CONTROLLED STUDY; DRUG EFFECT; DRUG FORMULATION; DRUG INHIBITION; HIPPOCAMPUS; LONG TERM POTENTIATION; MALE; NONHUMAN; POSTSYNAPTIC POTENTIAL; PRIORITY JOURNAL; RAT;

AMYLOID BETA-PROTEIN; ANIMALS; CASPASE 3; ELECTRIC STIMULATION; HIPPOCAMPUS; LONG-TERM POTENTIATION; MALE; RATS; RATS, WISTAR;

ANIMALIA; MUS MUSCULUS; RATTUS;

EID: 62349104665     PISSN: 05315565     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.exger.2008.12.001     Document Type: Article

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