Invitedreview: Deterioration of the immune system after trauma: Signals and cellular mechanisms

Innate Immunity

Volumn 14, Issue 6, 2008, Pages 333-344

  Flohé, Stefanie B ^a   Flohé, Sascha ^b   Schade, Ulrich F ^a  

^a UNIVERSITY HOSPITAL ESSEN   (Germany)

^b Heinrich HeineUniversity Düsseldorf ^*  (Germany)

Author keywords

Animal models; Danger signals; Immunosuppression; Injury; Trauma

Indexed keywords

ADVANCED GLYCATION END PRODUCT RECEPTOR; ALARMIN; CELL PROTEIN; CYCLOOXYGENASE 2 INHIBITOR; ENDOTOXIN; GAMMA INTERFERON; HEAT SHOCK PROTEIN; HEAT SHOCK PROTEIN 60; HEAT SHOCK PROTEIN 70; HIGH MOBILITY GROUP B1 PROTEIN; IBUPROFEN; INTERLEUKIN 2; LIPOPOLYSACCHARIDE; MITOGEN ACTIVATED PROTEIN KINASE; MONOCYTE CHEMOTACTIC PROTEIN 1; N (2 CYCLOHEXYLOXY 4 NITROPHENYL)METHANESULFONAMIDE; PATTERN RECOGNITION RECEPTOR; PROSTAGLANDIN E2; TOLL LIKE RECEPTOR; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG;

ANTIGEN PRESENTING CELL; CELLULAR IMMUNITY; CYTOKINE PRODUCTION; HEMORRHAGIC SHOCK; HUMAN; IMMUNE DEFICIENCY; IMMUNE RESPONSE; IMMUNOPATHOGENESIS; INFECTION RISK; INJURY; NONHUMAN; PERIPHERAL BLOOD MONONUCLEAR CELL; PROSTAGLANDIN SYNTHESIS INHIBITION; PROTEIN BINDING; PROTEIN FUNCTION; PROTEIN SECRETION; REGULATORY MECHANISM; REGULATORY T LYMPHOCYTE; REVIEW; SEPSIS; SIGNAL TRANSDUCTION; SOFT TISSUE INJURY; SYSTEMIC INFLAMMATORY RESPONSE SYNDROME; T LYMPHOCYTE ACTIVATION; ANIMAL; ANTIGEN PRESENTATION; DENDRITIC CELL; DISEASE MODEL; IMMUNE SYSTEM; IMMUNOLOGY; INNATE IMMUNITY; MACROPHAGE; MULTIPLE TRAUMA; PATHOLOGY; PATHOPHYSIOLOGY; T LYMPHOCYTE SUBPOPULATION; TH1 CELL;

ANIMALIA;

ANIMALS; ANTIGEN PRESENTATION; DENDRITIC CELLS; DISEASE MODELS, ANIMAL; HEAT-SHOCK PROTEINS; HMGB1 PROTEIN; HUMANS; IMMUNE SYSTEM; IMMUNITY, CELLULAR; IMMUNITY, INNATE; MACROPHAGES; MULTIPLE TRAUMA; SIGNAL TRANSDUCTION; T-LYMPHOCYTE SUBSETS; TH1 CELLS;

EID: 58949095481     PISSN: 17534259     EISSN: 17534267     Source Type: Journal    
DOI: 10.1177/1753425908100016     Document Type: Review

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