Overexpression of Glutathione Peroxidase Prevents Left Ventricular Remodeling and Failure after Myocardial Infarction in Mice

Circulation

Volumn 109, Issue 4, 2004, Pages 544-549

  Shiomi, Tetsuya ^a   Tsutsui, Hiroyuki ^a   Matsusaka, Hidenori ^a   Murakami, Kimiyo ^b   Hayashidani, Shunji ^a   Ikeuchi, Masaki ^a   Wen, Jing ^a   Kubota, Toro ^a   Utsumi, Hideo ^b   Takeshita, Akira ^a  

^a KYUSHU UNIVERSITY   (Japan)

^b KYUSHU UNIVERSITY   (Japan)

Author keywords

Antioxidants; Free radicals; Heart failure; Myocardial infarction; Remodeling

Indexed keywords

GELATINASE B; GLUTATHIONE PEROXIDASE; PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; ATTENUATION; CONCENTRATION RESPONSE; CONTROLLED STUDY; CORONARY ARTERY LIGATION; ENZYME ACTIVITY; ENZYME ANALYSIS; FIBROSING ALVEOLITIS; GENE OVEREXPRESSION; HEART DILATATION; HEART FAILURE; HEART INFARCTION; HEART INFARCTION SIZE; HEART LEFT VENTRICLE; HEART VENTRICLE REMODELING; LEFT CORONARY ARTERY; MALE; MOUSE; MUSCLE CELL; MUSCLE HYPERTROPHY; NONHUMAN; OXIDATIVE STRESS; PATHOPHYSIOLOGY; PRIORITY JOURNAL; STATISTICAL SIGNIFICANCE; TRANSGENIC MOUSE; ZYMOGRAPHY;

ANIMALS; ANTIOXIDANTS; APOPTOSIS; CARDIAC OUTPUT, LOW; GENE EXPRESSION; GLUTATHIONE PEROXIDASE; HEMODYNAMIC PROCESSES; MALE; MATRIX METALLOPROTEINASES; MICE; MICE, TRANSGENIC; MYOCARDIAL INFARCTION; MYOCARDIUM; ORGAN SIZE; SURVIVAL ANALYSIS; THIOBARBITURIC ACID REACTIVE SUBSTANCES; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR REMODELING;

EID: 10744225549     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.0000109701.77059.E9     Document Type: Article

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