Wnt signaling in caudal dysgenesis and diabetic embryopathy

Birth Defects Research Part A - Clinical and Molecular Teratology

Volumn 82, Issue 10, 2008, Pages 710-719

  Pavlinkova, Gabriela ^a,b   Salbaum, J Michael ^b,c,d   Kappen, Claudia ^b,c,d  

^a UNIVERSITY OF NEBRASKA MEDICAL CENTER   (United States)

^b UNIVERSITY OF NEBRASKA MEDICAL CENTER   (United States)

^c UNIVERSITY OF NEBRASKA MEDICAL CENTER   (United States)

^d PENNINGTON BIOMEDICAL RESEARCH CENTER   (United States)

Author keywords

Caudal dysgenesis; Developmental programs; Expression studies; Maternal diabetes; Midgestation; Mouse model; Neural tube defect; Transcriptional regulation; Transgenic; Wnt pathway

Indexed keywords

WNT3A PROTEIN; WNT5A PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CAUDAL DYSGENESIS; CONTROLLED STUDY; DEVELOPMENTAL DISORDER; EMBRYO DEVELOPMENT; EMBRYOPATHY; GENE EXPRESSION; GENE MUTATION; GENETIC TRANSCRIPTION; GESTATION PERIOD; IN SITU HYBRIDIZATION; INSULIN DEPENDENT DIABETES MELLITUS; MOUSE; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN FOLDING; REAL TIME POLYMERASE CHAIN REACTION; SIGNAL TRANSDUCTION;

ANIMALS; DIABETES MELLITUS, EXPERIMENTAL; DISEASE MODELS, ANIMAL; EMBRYO, MAMMALIAN; FEMALE; FETAL DISEASES; MICE; MICE, TRANSGENIC; NEURAL TUBE DEFECTS; PREGNANCY; PREGNANCY IN DIABETICS; SIGNAL TRANSDUCTION; WNT PROTEINS;

MUS MUSCULUS;

EID: 55449134236     PISSN: 15420752     EISSN: 15420760     Source Type: Journal    
DOI: 10.1002/bdra.20495     Document Type: Article

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